Role of Bcl-2 family of proteins in mediating apoptotic death of PC12 cells exposed to oxygen and glucose deprivation
暂无分享,去创建一个
Hao Jiang | R. Levine | Lijie Zhang | D. Koubi | Wenxue Tang
[1] P. Lazarovici,et al. Neuroprotective effects of carnosine and homocarnosine on pheochromocytoma PC12 cells exposed to ischemia , 2002, Journal of neuroscience research.
[2] John Calvin Reed,et al. Upregulation of bax protein levels in neurons following cerebral ischemia , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[3] P. Lipton,et al. Ischemic cell death in brain neurons. , 1999, Physiological reviews.
[4] D. Stephenson,et al. Bcl-Xshort is elevated following severe global ischemia in rat brains , 1997, Brain Research.
[5] P. Lazarovici,et al. Expression of human p140trk receptors in p140trk‐deficient, PC12/endothelial cells results in nerve growth factor‐induced signal transduction and DNA synthesis , 1997, Journal of cellular biochemistry.
[6] D. Green,et al. The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of Apoptosis , 1997, Science.
[7] P. Lazarovici,et al. Rasagiline, a monoamine oxidase‐B inhibitor, protects NGF‐differentiated PC12 cells against oxygen‐glucose deprivation , 1999, Journal of neuroscience research.
[8] Seth Love,et al. Apoptosis and brain ischaemia , 2003, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[9] K. Murphy,et al. Bcl-2 inhibits Bax translocation from cytosol to mitochondria during drug-induced apoptosis of human tumor cells , 2000, Cell Death and Differentiation.
[10] P. Lazarovici,et al. A tissue culture ischemic device to study eicosanoid release by pheochromocytoma PC12 cultures , 1993, Journal of Neuroscience Methods.
[11] H. Nakanishi,et al. Differential effects of Bcl‐2 overexpression on hippocampal CA1 neurons and dentate granule cells following hypoxic ischemia in adult mice , 1999, Journal of neuroscience research.
[12] A. Passaniti,et al. Bcl-2 overexpression attenuates resveratrol-induced apoptosis in U937 cells by inhibition of caspase-3 activity. , 2001, Carcinogenesis.
[13] J C Reed,et al. Bcl-x1 Bax Interaction after Transient Global Ischemia , 1998, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[14] S. Cory,et al. The Bcl-2 protein family: arbiters of cell survival. , 1998, Science.
[15] John Calvin Reed,et al. Heterodimerization-independent Functions of Cell Death Regulatory Proteins Bax and Bcl-2 in Yeast and Mammalian Cells* , 1997, The Journal of Biological Chemistry.
[16] D A Turner,et al. Mitochondrial and intrinsic optical signals imaged during hypoxia and spreading depression in rat hippocampal slices. , 2000, Journal of neurophysiology.
[17] M. Hori,et al. Amelioration of hippocampal neuronal damage after global ischemia by neuronal overexpression of BCL-2 in transgenic mice. , 1998, Stroke.
[18] G. Kroemer,et al. Caspases disrupt mitochondrial membrane barrier function , 1998, FEBS letters.
[19] Hao Jiang,et al. Tetrahydrobiopterin Enhances Apoptotic PC12 Cell Death following Withdrawal of Trophic Support* , 2001, The Journal of Biological Chemistry.
[20] M. Minami,et al. Intracellular Bax Translocation after Transient Cerebral Ischemia: Implications for a Role of the Mitochondrial Apoptotic Signaling Pathway in Ischemic Neuronal Death , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[21] P. Lazarovici,et al. Neuroprotective and neurotoxic effects of monoamine oxidase-B inhibitors and derived metabolites under ischemia in PC12 cells. , 2002, European journal of pharmacology.
[22] I. Ferrer,et al. Bcl-2, Bax, and Bcl-x expression in the CA1 area of the hippocampus following transient forebrain ischemia in the adult gerbil , 1998, Experimental Brain Research.
[23] John Calvin Reed,et al. Differential Regulation of Bax, Bcl‐2, and Bcl‐X Proteins in Focal Cortical Ischemia in the Rat , 1998, Brain pathology.
[24] G. Robertson,et al. Attenuation of Ischemia-Induced Cellular and Behavioral Deficits by X Chromosome-Linked Inhibitor of Apoptosis Protein Overexpression in the Rat Hippocampus , 1999, The Journal of Neuroscience.
[25] G. Kroemer,et al. Inhibitors of permeability transition interfere with the disruption of the mitochondrial transmembrane potential during apoptosis , 1996, FEBS letters.
[26] G. Kroemer,et al. Disruption of the outer mitochondrial membrane as a result of large amplitude swelling: the impact of irreversible permeability transition , 1998, FEBS letters.
[27] Y. Tsujimoto,et al. Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[28] P. Lazarovici,et al. Both p140trk and p75NGFR Nerve Growth Factor Receptors Mediate Nerve Growth Factor-stimulated Calcium Uptake* , 1997, The Journal of Biological Chemistry.
[29] M. Benito,et al. Inhibition of caspases rescues brown adipocytes from apoptosis downregulating BCL-XS and upregulating BCL-2 gene expression. , 1999, Experimental cell research.
[30] R. Simon,et al. Induction of Caspase-3-Like Protease May Mediate Delayed Neuronal Death in the Hippocampus after Transient Cerebral Ischemia , 1998, The Journal of Neuroscience.
[31] Y. Tsujimoto,et al. Bcl-2 blocks loss of mitochondrial membrane potential while ICE inhibitors act at a different step during inhibition of death induced by respiratory chain inhibitors. , 1996, Oncogene.
[32] E. White,et al. Interaction of E1B 19K with Bax is required to block Bax-induced loss of mitochondrial membrane potential and apoptosis , 1998, Oncogene.
[33] C. Borner,et al. Bcl-xS can form homodimers and heterodimers and its apoptotic activity requires localization of Bcl-xS to the mitochondria and its BH3 and loop domains , 2001, Cell Death and Differentiation.
[34] G. Kroemer,et al. The Central Executioner of Apoptosis: Multiple Connections between Protease Activation and Mitochondria in Fas/APO-1/CD95- and Ceramide-induced Apoptosis , 1997, The Journal of experimental medicine.
[35] B. Herman,et al. Mitochondrial permeability transition in pH-dependent reperfusion injury to rat hepatocytes. , 1997, American journal of physiology. Cell physiology.
[36] R. Maroto,et al. BCL‐2‐Related Protein Expression in Apoptosis: Oxidative Stress Versus Serum Deprivation in PC12 Cells , 1997, Journal of neurochemistry.