Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors
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[1] M. Rogawski,et al. The neuropharmacological basis for the use of memantine in the treatment of Alzheimer's disease. , 2006, CNS drug reviews.
[2] P. Bickford,et al. Sulindac improves memory and increases NMDA receptor subunits in aged Fischer 344 rats , 2004, Neurobiology of Aging.
[3] G. Wenk,et al. The toxicity of tumor necrosis factor-α upon cholinergic neurons within the nucleus basalis and the role of norepinephrine in the regulation of inflammation: implications for alzheimer's disease , 2003, Neuroscience.
[4] C L Galli,et al. Interleukin-1β Enhances NMDA Receptor-Mediated Intracellular Calcium Increase through Activation of the Src Family of Kinases , 2003, The Journal of Neuroscience.
[5] B. Platt,et al. Glutamate receptor function in learning and memory , 2003, Behavioural Brain Research.
[6] Kiyoshi Morimoto,et al. Acute Neuroinflammation Exacerbates Excitotoxicity in Rat Hippocampus in Vivo , 2002, Experimental Neurology.
[7] T. Asada,et al. Alterations of NMDAR1 and NMDAR2A/B immunoreactivity in the hippocampus after perforant pathway lesion , 2002, Neuropathology : official journal of the Japanese Society of Neuropathology.
[8] H. Bi,et al. N-methyl-d-aspartate receptor subunit NR2A and NR2B messenger RNA levels are altered in the hippocampus and entorhinal cortex in Alzheimer's disease , 2002, Journal of the Neurological Sciences.
[9] B. Hauss-Wegrzyniak,et al. Chronic Brain Inflammation Results in Cell Loss in the Entorhinal Cortex and Impaired LTP in Perforant Path-Granule Cell Synapses , 2002, Experimental Neurology.
[10] A. Biegon,et al. Region‐selective effects of neuroinflammation and antioxidant treatment on peripheral benzodiazepine receptors and NMDA receptors in the rat brain , 2002, Journal of neurochemistry.
[11] Guy C. Brown,et al. Inflammatory Neurodegeneration Mediated by Nitric Oxide from Activated Glia-Inhibiting Neuronal Respiration, Causing Glutamate Release and Excitotoxicity , 2001, The Journal of Neuroscience.
[12] Roger N Gunn,et al. In-vivo measurement of activated microglia in dementia , 2001, The Lancet.
[13] A. Baskys,et al. Neuroprotective effects of extracellular glutamate are absent in hippocampal organotypic cultures treated with the amyloid peptide Aβ25–35 , 2001, Brain Research.
[14] G. Kollias,et al. CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity , 2001, Nature Neuroscience.
[15] Carol A. Barnes,et al. Regional changes in the hippocampal density of AMPA and NMDA receptors across the lifespan of the rat , 2000, Brain Research.
[16] Jean-Philippe Galons,et al. Quantitative Volumetric Analyses of Brain Magnetic Resonance Imaging from Rat with Chronic Neuroinflammation , 2000, Experimental Neurology.
[17] G. Wenk,et al. Mechanisms to prevent the toxicity of chronic neuroinflammation on forebrain cholinergic neurons. , 2000, European journal of pharmacology.
[18] G. Wenk,et al. LPS‐induced neuroinflammatory effects do not recover with time , 2000, Neuroreport.
[19] C. Plata-salamán,et al. Inflammation and Alzheimer’s disease , 2000, Neurobiology of Aging.
[20] G. Wenk,et al. Behavioral and ultrastructural changes induced by chronic neuroinflammation in young rats , 2000, Brain Research.
[21] W. Liao,et al. Inflammation in Alzheimer's Disease , 1999, Science.
[22] G. Wenk,et al. The effects of a novel NSAID on chronic neuroinflammation are age dependent☆ , 1999, Neurobiology of Aging.
[23] P. Mcgeer,et al. The importance of inflammatory mechanisms in alzheimer disease , 1998, Experimental Gerontology.
[24] Tullio Pozzan,et al. Prostaglandins stimulate calcium-dependent glutamate release in astrocytes , 1998, Nature.
[25] P. Dobrzanski,et al. Chronic neuroinflammation in rats reproduces components of the neurobiology of Alzheimer's disease , 1998, Brain Research.
[26] F. Morrell,et al. Alzheimer’s Disease: In Vivo Detection of Differential Vulnerability of Brain Regions , 1997, Neurobiology of Aging.
[27] J. Urenjak,et al. Altered glutamatergic transmission in neurological disorders: From high extracellular glutamate to excessive synaptic efficacy , 1997, Progress in Neurobiology.
[28] J. Morris,et al. Profound Loss of Layer II Entorhinal Cortex Neurons Occurs in Very Mild Alzheimer’s Disease , 1996, The Journal of Neuroscience.
[29] G. Levi,et al. Induction of Prostanoid Biosynthesis by Bacterial Lipopolysaccharide and Isoproterenol in Rat Microglial Cultures , 1995, Journal of neurochemistry.
[30] W. Fiers,et al. Direct evidence for tumor necrosis factor-induced mitochondrial reactive oxygen intermediates and their involvement in cytotoxicity. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[31] J. Weiss,et al. Induction of interleukin-1 in various brain regions after peripheral and central injections of lipopolysaccharide , 1994, Journal of Neuroimmunology.
[32] J. Buchhalter,et al. Inhibition of Glutamate Uptake with l‐trans‐Pyrrolidine‐2,4‐Dicarboxylate Potentiates Glutamate Toxicity in Primary Hippocampal Cultures , 1993, Journal of neurochemistry.
[33] J. Rothstein,et al. Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[34] Bert Sakmann,et al. Heteromeric NMDA Receptors: Molecular and Functional Distinction of Subtypes , 1992, Science.
[35] R. Albin,et al. Alternative excitotoxic hypotheses , 1992, Neurology.
[36] A. Arimura,et al. Interleukin-1 beta increases prostaglandin E2 in rat astrocyte cultures: modulatory effect of neuropeptides. , 1989, Endocrinology.
[37] Oliver H. Lowry,et al. Protein measurement with the Folin phenol reagent. , 1951, The Journal of biological chemistry.
[38] H. Braak,et al. Neuropathological stageing of Alzheimer-related changes , 2004, Acta Neuropathologica.
[39] G. Wenk,et al. Chronic Intracerebral LPS as a Model of Neuroinflammation , 2003 .
[40] P. Wood. Neuroinflammation : mechanisms and management , 2003 .
[41] T. Morgan,et al. Inflammation in Alzheimer’s Disease , 2001 .