Cutaneous lesions in psoriatic arthritis are enriched in chemokine transcriptomic pathways

[1]  John J. Cole,et al.  Searchlight: automated bulk RNA-seq exploration and visualisation using dynamically generated R scripts , 2021, BMC Bioinform..

[2]  A. Stewart,et al.  Analysis of combinatorial chemokine receptor expression dynamics using multi-receptor reporter mice , 2021, bioRxiv.

[3]  S. Teichmann,et al.  Developmental cell programs are co-opted in inflammatory skin disease , 2021, Science.

[4]  D. Chaussabel,et al.  A Neutrophil-Driven Inflammatory Signature Characterizes the Blood Transcriptome Fingerprint of Psoriasis , 2020, Frontiers in Immunology.

[5]  M. Simard,et al.  Transcriptome Profiling Analyses in Psoriasis: A Dynamic Contribution of Keratinocytes to the Pathogenesis , 2020, Genes.

[6]  Shih-Ying Wu,et al.  Polyfunctional, Proinflammatory, Tissue‐Resident Memory Phenotype and Function of Synovial Interleukin‐17A+CD8+ T Cells in Psoriatic Arthritis , 2020, Arthritis & Rheumatology (Hoboken, N.j.).

[7]  T. Hwang,et al.  Neutrophils in Psoriasis , 2019, Front. Immunol..

[8]  C. Pitzalis,et al.  Interleukin-36 family dysregulation drives joint inflammation and therapy response in psoriatic arthritis , 2019, Rheumatology.

[9]  Ling-juan Zhang,et al.  Keratin 6, 16 and 17—Critical Barrier Alarmin Molecules in Skin Wounds and Psoriasis , 2019, Cells.

[10]  R. Qi,et al.  Integrated bioinformatic analysis of differentially expressed genes and signaling pathways in plaque psoriasis , 2019, Molecular medicine reports.

[11]  G. Graham,et al.  Chemokine Receptor Redundancy and Specificity Are Context Dependent , 2019, Immunity.

[12]  A. Ogdie,et al.  Preventing psoriatic arthritis: focusing on patients with psoriasis at increased risk of transition , 2019, Nature Reviews Rheumatology.

[13]  A. Gottlieb,et al.  Prevalence of psoriatic arthritis in patients with psoriasis: A systematic review and meta‐analysis of observational and clinical studies , 2019, Journal of the American Academy of Dermatology.

[14]  K. Ladell,et al.  CCR8 Expression Defines Tissue-Resident Memory T Cells in Human Skin , 2018, The Journal of Immunology.

[15]  Barbara B. Shih,et al.  Derivation of marker gene signatures from human skin and their use in the interpretation of the transcriptional changes associated with dermatological disorders , 2017, The Journal of pathology.

[16]  S. Holmes,et al.  Spread of Psoriasiform Inflammation to Remote Tissues Is Restricted by the Atypical Chemokine Receptor ACKR2 , 2017, The Journal of investigative dermatology.

[17]  A. Baranova,et al.  Integrated computational approach to the analysis of RNA-seq data reveals new transcriptional regulators of psoriasis , 2016, Experimental & Molecular Medicine.

[18]  James T. Elder,et al.  Sebaceous Gland Atrophy in Psoriasis: An Explanation for Psoriatic Alopecia? , 2016, The Journal of investigative dermatology.

[19]  S. Tian,et al.  Increased expression of interleukin‐17 pathway genes in nonlesional skin of moderate‐to‐severe psoriasis vulgaris , 2016, The British journal of dermatology.

[20]  E. Tinazzi,et al.  Gene Expression Profiling in Peripheral Blood Cells and Synovial Membranes of Patients with Psoriatic Arthritis , 2015, PloS one.

[21]  B. Moser CXCR5, the Defining Marker for Follicular B Helper T (TFH) Cells , 2015, Front. Immunol..

[22]  Kristi Abram,et al.  Transcriptional landscape of psoriasis identifies the involvement of IL36 and IL36RN , 2015, BMC Genomics.

[23]  Vinod Chandran,et al.  Quantitative tandem mass-spectrometry of skin tissue reveals putative psoriatic arthritis biomarkers , 2015, Clinical Proteomics.

[24]  W. Huber,et al.  Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2 , 2014, Genome Biology.

[25]  Andrew Johnston,et al.  Transcriptome analysis of psoriasis in a large case-control sample: RNA-seq provides insights into disease mechanisms , 2014, The Journal of investigative dermatology.

[26]  A. Mantovani,et al.  International Union of Basic and Clinical Pharmacology. LXXXIX. Update on the Extended Family of Chemokine Receptors and Introducing a New Nomenclature for Atypical Chemokine Receptors , 2014, Pharmacological Reviews.

[27]  I. Melero,et al.  Initial Afferent Lymphatic Vessels Controlling Outbound Leukocyte Traffic from Skin to Lymph Nodes , 2013, Front. Immunol..

[28]  Gianfranco Altomare,et al.  Inhibition of CCR7/CCL19 axis in lesional skin is a critical event for clinical remission induced by TNF blockade in patients with psoriasis. , 2013, The American journal of pathology.

[29]  Andrew Johnston,et al.  Dissecting the psoriasis transcriptome: inflammatory- and cytokine-driven gene expression in lesions from 163 patients , 2013, BMC Genomics.

[30]  I. McInnes,et al.  Elevated expression of the chemokine-scavenging receptor D6 is associated with impaired lesion development in psoriasis. , 2012, The American journal of pathology.

[31]  Hideki Fujita,et al.  Combined Use of Laser Capture Microdissection and cDNA Microarray Analysis Identifies Locally Expressed Disease-Related Genes in Focal Regions of Psoriasis Vulgaris Skin Lesions , 2012, The Journal of investigative dermatology.

[32]  S. Kaesler,et al.  CXCL16 and CXCR6 are upregulated in psoriasis and mediate cutaneous recruitment of human CD8+ T cells. , 2012, The Journal of investigative dermatology.

[33]  Xing Li,et al.  Global gene expression analysis reveals evidence for decreased lipid biosynthesis and increased innate immunity in uninvolved psoriatic skin. , 2009, The Journal of investigative dermatology.

[34]  James T. Elder,et al.  Induction of IL-17+ T Cell Trafficking and Development by IFN-γ: Mechanism and Pathological Relevance in Psoriasis1 , 2008, The Journal of Immunology.

[35]  Howard Y. Chang,et al.  A systems biology approach to anatomic diversity of skin. , 2008, The Journal of investigative dermatology.

[36]  J. Naeyaert,et al.  Expression of the chemokine receptor CCR5 in psoriasis and results of a randomized placebo controlled trial with a CCR5 inhibitor , 2007, Archives of Dermatological Research.

[37]  Andreas Radbruch,et al.  Regulation of CXCR3 and CXCR4 expression during terminal differentiation of memory B cells into plasma cells. , 2005, Blood.

[38]  T. Blankenstein,et al.  CCR7 governs skin dendritic cell migration under inflammatory and steady-state conditions. , 2004, Immunity.

[39]  K. Matsushima,et al.  Differential Expression of the Chemokine Receptors by the Th1- and Th2-type Effector Populations within Circulating Cd4 T Cells , 2022 .

[40]  M. Ashburner,et al.  Gene Ontology: tool for the unification of biology , 2000, Nature Genetics.

[41]  H. Wigzell,et al.  The Expression of the Gene Coding for the Antibacterial Peptide LL-37 Is Induced in Human Keratinocytes during Inflammatory Disorders* , 1997, The Journal of Biological Chemistry.

[42]  E. Wolf,et al.  A Putative Chemokine Receptor, BLR1, Directs B Cell Migration to Defined Lymphoid Organs and Specific Anatomic Compartments of the Spleen , 1996, Cell.

[43]  Shih-Ying Wu,et al.  Polyfunctional, Proinflammatory, Tissue‐Resident Memory Phenotype and Function of Synovial Interleukin‐17A+CD8+ T Cells in Psoriatic Arthritis , 2020, Arthritis & rheumatology.

[44]  K. Matsushima,et al.  CCR4 and its ligands: from bench to bedside. , 2015, International immunology.

[45]  Thomas R. Gingeras,et al.  STAR: ultrafast universal RNA-seq aligner , 2013, Bioinform..

[46]  B. Kofler,et al.  Galanin family of peptides in skin function. , 2010, Experientia supplementum.

[47]  Hilde van der Togt,et al.  Publisher's Note , 2003, J. Netw. Comput. Appl..