Analysis and quantitation of ceramide.

[1]  Y. Hannun,et al.  Ceramide in the eukaryotic stress response. , 2000, Trends in cell biology.

[2]  A. Bielawska,et al.  Quantitative determination of ceramide using diglyceride kinase. , 2000, Methods in enzymology.

[3]  A. Bielawska,et al.  Phosphatidic Acid Is a Potent And Selective Inhibitor of Protein Phosphatase 1 and an Inhibitor of Ceramide-mediated Responses* , 1999, The Journal of Biological Chemistry.

[4]  A. Bielawska,et al.  Long Chain Ceramides Activate Protein Phosphatase-1 and Protein Phosphatase-2A , 1999, The Journal of Biological Chemistry.

[5]  M. Tanaka,et al.  Biological activities of scyphostatin, a neutral sphingomyelinase inhibitor from a discomycete, Trichopeziza mollissima. , 1999, The Journal of antibiotics.

[6]  Y. Hannun,et al.  The use of diglyceride kinase for quantifying ceramide. , 1999, Trends in biochemical sciences.

[7]  R. Kolesnick,et al.  Signal transduction of stress via ceramide. , 1998, The Biochemical journal.

[8]  Y. Hirabayashi,et al.  Ceramide and Its Interconvertible Metabolite Sphingosine Function as Indispensable Lipid Factors Involved in Survival and Dendritic Differentiation of Cerebellar Purkinje Cells , 1998, Journal of neurochemistry.

[9]  Weiya Ma,et al.  Direct Evidence for an Important Role of Sphingomyelinase in Ultraviolet-induced Activation of c-Jun N-Terminal Kinase* , 1997, The Journal of Biological Chemistry.

[10]  C. Hoppel,et al.  Direct Inhibition of Mitochondrial Respiratory Chain Complex III by Cell-permeable Ceramide* , 1997, The Journal of Biological Chemistry.

[11]  O. Linderkamp,et al.  Fas- or Ceramide-induced Apoptosis Is Mediated by a Rac1-regulated Activation of Jun N-terminal Kinase/p38 Kinases and GADD153* , 1997, The Journal of Biological Chemistry.

[12]  Obeid,et al.  Sphingomyelinases in cell regulation. , 1997, Seminars in cell & developmental biology.

[13]  John Calvin Reed,et al.  Activation of CPP32 during apoptosis of neurons and astrocytes , 1997, Journal of neuroscience research.

[14]  Y. Hannun,et al.  Expression of Neutral Sphingomyelinase Identifies a Distinct Pool of Sphingomyelin Involved in Apoptosis* , 1997, The Journal of Biological Chemistry.

[15]  R. Kolesnick,et al.  Kinase Suppressor of Ras Is Ceramide-Activated Protein Kinase , 1997, Cell.

[16]  D. Green,et al.  The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of Apoptosis , 1997, Science.

[17]  Y. Hannun,et al.  Cytokine Response Modifier A (CrmA) Inhibits Ceramide Formation in Response to Tumor Necrosis Factor (TNF)-α: CrmA and Bcl-2 Target Distinct Components in the Apoptotic Pathway , 1997, The Journal of experimental medicine.

[18]  S. Spiegel,et al.  Sphingolipids--the enigmatic lipid class: biochemistry, physiology, and pathophysiology. , 1997, Toxicology and applied pharmacology.

[19]  Y. Hannun.  Functions of Ceramide in Coordinating Cellular Responses to Stress , 1996, Science.

[20]  E. Alnemri,et al.  Heteronuclear Ribonucleoproteins C1 and C2, Components of the Spliceosome, Are Specific Targets of Interleukin 1β-converting Enzyme-like Proteases in Apoptosis* , 1996, The Journal of Biological Chemistry.

[21]  S. Rosenzweig,et al.  Activation of C‐jun N‐terminal kinase/stress‐activated protein kinase in primary glial cultures , 1996, Journal of neuroscience research.

[22]  J. Reyes,et al.  c-Jun Is a Downstream Target for Ceramide-activated Protein Phosphatase in A431 Cells* , 1996, The Journal of Biological Chemistry.

[23]  S. Spiegel,et al.  Suppression of ceramide-mediated programmed cell death by sphingosine-1-phosphate , 1996, Nature.

[24]  Y. Hannun,et al.  Ceramide Inactivates Cellular Protein Kinase Cα* , 1996, The Journal of Biological Chemistry.

[25]  Y. Hannun,et al.  prICE: a downstream target for ceramide-induced apoptosis and for the inhibitory action of Bcl-2. , 1996, The Biochemical journal.

[26]  L. Zon,et al.  Requirement for ceramide-initiated SAPK/JNK signalling in stress-induced apoptosis , 1996, Nature.

[27]  J. Broach,et al.  A ceramide-activated protein phosphatase mediates ceramide-induced G1 arrest of Saccharomyces cerevisiae. , 1996, Genes & development.

[28]  G. Koretzky,et al.  Fas ligation induces apoptosis and Jun kinase activation independently of CD45 and Lck in human T cells. , 1996, Blood.

[29]  D. Brenner,et al.  Ceramide Activates the Stress-activated Protein Kinases (*) , 1995, The Journal of Biological Chemistry.

[30]  D. Perl,et al.  Acid sphingomyelinase deficient mice: a model of types A and B Niemann–Pick disease , 1995, Nature Genetics.

[31]  W. Stoffel,et al.  Acid sphingomyelinase-deficient mice mimic the neurovisceral form of human lysosomal storage disease (Niemann-Pick disease) , 1995, Cell.

[32]  Y. Miyake,et al.  Serine palmitoyltransferase is the primary target of a sphingosine-like immunosuppressant, ISP-1/myriocin. , 1995, Biochemical and biophysical research communications.

[33]  W. R. Bishop,et al.  Ceramide quantitation: evaluation of a mixed micellar assay using E. coli diacylglycerol kinase. , 1995, Biochemistry and molecular biology international.

[34]  Y. Hannun,et al.  Retinoblastoma gene product as a downstream target for a ceramide-dependent pathway of growth arrest. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[35]  A. Bielawska,et al.  Role for Ceramide in Cell Cycle Arrest (*) , 1995, The Journal of Biological Chemistry.

[36]  T. Okazaki,et al.  A redox factor protein, ref1, is involved in negative gene regulation by extracellular calcium. , 1994, The Journal of biological chemistry.

[37]  G. Blobe,et al.  Identification of a defect in the phospholipase D/diacylglycerol pathway in cellular senescence. , 1994, The Journal of biological chemistry.

[38]  R. Dobrowsky,et al.  Role of ceramide-activated protein phosphatase in ceramide-mediated signal transduction. , 1994, The Journal of biological chemistry.

[39]  Y. Hannun,et al.  Programmed cell death induced by ceramide. , 1993, Science.

[40]  M. Liscovitch.  Crosstalk among multiple signal-activated phospholipases. , 1992, Trends in biochemical sciences.

[41]  W. R. Bishop,et al.  Quantitative measurement of sn-1,2-diacylglycerols present in platelets, hepatocytes, and ras- and sis-transformed normal rat kidney cells. , 1986, The Journal of biological chemistry.

[42]  J. Hauw,et al.  Effect of tricyclic antidepressants on sphingomyelinase and other sphingolipid hydrolases in C6 cultured glioma cells , 1983, Neuroscience Letters.

[43]  J. Bernert,et al.  Biosynthesis of sphingomyelin from erythro-ceramides and phosphatidylcholine by a microsomal cholinephosphotransferase. , 1981, Biochimica et biophysica acta.

[44]  E. P. Kennedy,et al.  Sphingomyelinase in normal human spleens and in spleens from subjects with Niemann-Pick disease. , 1967, Journal of lipid research.

[45]  L. Hokin,et al.  Diglyceride phosphokinase: an enzyme which catalyzes the synthesis of phosphatidic acid. , 1959, Biochimica et biophysica acta.

[46]  J. Folch,et al.  A simple method for the isolation and purification of total lipides from animal tissues. , 1957, The Journal of biological chemistry.