Relationship between baseline white blood cell count and degree of coronary artery disease and mortality in patients with acute coronary syndromes ☆: A TACTICS-TIMI 18 substudy

[1]  R. Gibbons,et al.  Double-Blind, Randomized Trial of an Anti-CD18 Antibody in Conjunction With Recombinant Tissue Plasminogen Activator for Acute Myocardial Infarction: Limitation of Myocardial Infarction Following Thrombolysis in Acute Myocardial Infarction (LIMIT AMI) Study , 2001, Circulation.

[2]  E. Braunwald,et al.  Ability of minor elevations of troponins I and T to predict benefit from an early invasive strategy in patients with unstable angina and non-ST elevation myocardial infarction: results from a randomized trial. , 2001, JAMA.

[3]  A. Siegbahn,et al.  Relationship between interleukin 6 and mortality in patients with unstable coronary artery disease: effects of an early invasive or noninvasive strategy. , 2001, JAMA.

[4]  E. Braunwald,et al.  Comparison of early invasive and conservative strategies in patients with unstable coronary syndromes treated with the glycoprotein IIb/IIIa inhibitor tirofiban. , 2001, The New England journal of medicine.

[5]  P. Libby,et al.  Inflammation and thrombosis: the clot thickens. , 2001, Circulation.

[6]  E. Braunwald,et al.  Association of white blood cell count with increased mortality in acute myocardial infarction and unstable angina pectoris , 2001 .

[7]  E. Braunwald,et al.  Association Between White Blood Cell Count, Epicardial Blood Flow, Myocardial Perfusion, and Clinical Outcomes in the Setting of Acute Myocardial Infarction: A Thrombolysis In Myocardial Infarction 10 Substudy , 2000, Circulation.

[8]  A. Siegbahn,et al.  Markers of myocardial damage and inflammation in relation to long-term mortality in unstable coronary artery disease. FRISC Study Group. Fragmin during Instability in Coronary Artery Disease. , 2000, The New England journal of medicine.

[9]  C. Heeschen,et al.  Predictive value of C-reactive protein and troponin T in patients with unstable angina: a comparative analysis. CAPTURE Investigators. Chimeric c7E3 AntiPlatelet Therapy in Unstable angina REfractory to standard treatment trial. , 2000, Journal of the American College of Cardiology.

[10]  C M Gibson,et al.  Relationship of TIMI myocardial perfusion grade to mortality after administration of thrombolytic drugs. , 2000, Circulation.

[11]  F. Neumann,et al.  Effect of glycoprotein IIb/IIIa receptor blockade on platelet-leukocyte interaction and surface expression of the leukocyte integrin Mac-1 in acute myocardial infarction. , 1999, Journal of the American College of Cardiology.

[12]  R. Schaub,et al.  Recombinant soluble form of PSGL-1 accelerates thrombolysis and prevents reocclusion in a porcine model. , 1999, Circulation.

[13]  R. Ross,et al.  Atherosclerosis is an inflammatory disease. , 1998, American heart journal.

[14]  F. Neumann,et al.  Procoagulant inflammatory responses of monocytes after direct balloon angioplasty in acute myocardial infarction. , 1998, The American journal of cardiology.

[15]  E. Antman,et al.  C-Reactive Protein Is a Potent Predictor of Mortality Independently of and in Combination With Troponin T in Acute Coronary Syndromes: A TIMI 11A Substudy , 1998 .

[16]  A. Siegbahn,et al.  Prognostic influence of increased fibrinogen and C-reactive protein levels in unstable coronary artery disease. FRISC Study Group. Fragmin during Instability in Coronary Artery Disease. , 1997, Circulation.

[17]  F. Neumann,et al.  Induction of cytokine expression in leukocytes in acute myocardial infarction. , 1997, Journal of the American College of Cardiology.

[18]  P. Ridker,et al.  Inflammation, aspirin, and the risk of cardiovascular disease in apparently healthy men. , 1997, The New England journal of medicine.

[19]  E. Braunwald,et al.  TIMI frame count: a quantitative method of assessing coronary artery flow. , 1996, Circulation.

[20]  P. Libby.  Molecular bases of the acute coronary syndromes. , 1995, Circulation.

[21]  A. Becker,et al.  Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology. , 1994, Circulation.

[22]  E. Braunwald,et al.  Myocardial reperfusion: a double-edged sword? , 1985, The Journal of clinical investigation.

[23]  H. S. Mueller,et al.  The Thrombolysis in Myocardial Infarction (TIMI) trial. Phase I findings. , 1985, The New England journal of medicine.

[24]  G. Schmid-Schönbein,et al.  Leukocyte capillary plugging in myocardial ischemia and reperfusion in the dog. , 1983, The American journal of pathology.

[25]  R. Schlant,et al.  The Natural History of Coronary Heart Disease: Prognostic Factors After Recovery from Myocardial Infarction in 2789 Men The 5‐year Findings of the Coronary Drug Project prepared for the Coronary Drug Project Research Group bv , 1982, Circulation.

[26]  A. Siegelaub,et al.  The leukocyte count as a predictor of myocardial infarction. , 1974, The New England journal of medicine.