Co-expression of SOX9 and SOX10 during melanocytic differentiation in vitro.

[1]  P. Nelson,et al.  Suppression of growth and tumorigenicity in the prostate tumor cell line M12 by overexpression of the transcription factor SOX9 , 2004, Oncogene.

[2]  H. Lee,et al.  Spatiotemporal regulation of endothelin receptor-B by SOX10 in neural crest–derived enteric neuron precursors , 2004, Nature Genetics.

[3]  M. Wegner,et al.  Transcription factors Sox8 and Sox10 perform non-equivalent roles during oligodendrocyte development despite functional redundancy , 2004, Development.

[4]  R. Marais,et al.  The Brn-2 Transcription Factor Links Activated BRAF to Melanoma Proliferation , 2004, Molecular and Cellular Biology.

[5]  L. Larue,et al.  Brn-2 Expression Controls Melanoma Proliferation and Is Directly Regulated by β-Catenin , 2004, Molecular and Cellular Biology.

[6]  C. Berking,et al.  Induction of Melanoma Phenotypes in Human Skin by Growth Factors and Ultraviolet B , 2004, Cancer Research.

[7]  M. Wegner,et al.  Melanocyte‐specific expression of dopachrome tautomerase is dependent on synergistic gene activation by the Sox10 and Mitf transcription factors , 2004, FEBS letters.

[8]  J. Briscoe,et al.  Neural crest development is regulated by the transcription factor Sox9 , 2003, Development.

[9]  M. Herlyn,et al.  Human melanoblasts in culture: expression of BRN2 and synergistic regulation by fibroblast growth factor-2, stem cell factor, and endothelin-3. , 2003, The Journal of investigative dermatology.

[10]  Yasuhito Kobayashi,et al.  Mouse models for four types of Waardenburg syndrome. , 2003, Pigment cell research.

[11]  D. Bennett,et al.  The color loci of mice--a genetic century. , 2003, Pigment cell research.

[12]  W. Mandemakers,et al.  The POU proteins Brn-2 and Oct-6 share important functions in Schwann cell development. , 2003, Genes & development.

[13]  David J. Anderson,et al.  SOX10 Maintains Multipotency and Inhibits Neuronal Differentiation of Neural Crest Stem Cells , 2003, Neuron.

[14]  J. Naeyaert,et al.  Evidence for an autoimmune pathogenesis of vitiligo. , 2003, Pigment cell research.

[15]  A. Read,et al.  SLUG (SNAI2) deletions in patients with Waardenburg disease. , 2002, Human molecular genetics.

[16]  M. Wegner,et al.  Sox10 Is an Active Nucleocytoplasmic Shuttle Protein, and Shuttling Is Crucial for Sox10-Mediated Transactivation , 2002, Molecular and Cellular Biology.

[17]  A. Nicholson,et al.  Mutations of the BRAF gene in human cancer , 2002, Nature.

[18]  S. Easteal,et al.  The human melanocortin-1 receptor locus: analysis of transcription unit, locus polymorphism and haplotype evolution. , 2001, Gene.

[19]  J. Perheentupa,et al.  The Transcription Factors SOX9 and SOX10 Are Vitiligo Autoantigens in Autoimmune Polyendocrine Syndrome Type I* , 2001, The Journal of Biological Chemistry.

[20]  W. Pavan,et al.  Analysis of SOX10 function in neural crest-derived melanocyte development: SOX10-dependent transcriptional control of dopachrome tautomerase. , 2001, Developmental biology.

[21]  C. Basilico,et al.  Coevolution of HMG domains and homeodomains and the generation of transcriptional regulation by Sox/POU complexes , 2001, Journal of cellular physiology.

[22]  R. Ballotti,et al.  Direct Regulation of the Microphthalmia Promoter by Sox10 Links Waardenburg-Shah Syndrome (WS4)-associated Hypopigmentation and Deafness to WS2* , 2000, The Journal of Biological Chemistry.

[23]  P. Parsons,et al.  Domains of Brn-2 that mediate homodimerization and interaction with general and melanocytic transcription factors. , 2000, European journal of biochemistry.

[24]  K. Bille,et al.  Regulation of the Microphthalmia-associated Transcription Factor Gene by the Waardenburg Syndrome Type 4 Gene,SOX10 * , 2000, The Journal of Biological Chemistry.

[25]  M. Wegner,et al.  The glial transcription factor Sox10 binds to DNA both as monomer and dimer with different functional consequences. , 2000, Nucleic acids research.

[26]  M. Wegner,et al.  Interaction among SOX10, PAX3 and MITF, three genes altered in Waardenburg syndrome. , 2000, Human molecular genetics.

[27]  C. Goding,et al.  Mitf from neural crest to melanoma: signal transduction and transcription in the melanocyte lineage. , 2000, Genes & development.

[28]  W. Pavan,et al.  Transcription factor hierarchy in Waardenburg syndrome: regulation of MITF expression by SOX10 and PAX3 , 2000, Human Genetics.

[29]  H. Kondoh,et al.  Pairing SOX off: with partners in the regulation of embryonic development. , 2000, Trends in genetics : TIG.

[30]  T. Dexter,et al.  Pax3 and Regulation of the Melanocyte-specific Tyrosinase-related Protein-1 Promoter* , 1999, The Journal of Biological Chemistry.

[31]  M. Wegner,et al.  Functional Analysis of Sox10 Mutations Found in Human Waardenburg-Hirschsprung Patients* , 1998, The Journal of Biological Chemistry.

[32]  D. Fisher,et al.  Involvement of Microphthalmia in the Inhibition of Melanocyte Lineage Differentiation and of Melanogenesis by Agouti Signal Protein* , 1998, The Journal of Biological Chemistry.

[33]  M. Wegner,et al.  Cooperative Function of POU Proteins and SOX Proteins in Glial Cells* , 1998, The Journal of Biological Chemistry.

[34]  K. Takeda,et al.  Epistatic relationship between Waardenburg Syndrome genes MITF and PAX3 , 1998, Nature Genetics.

[35]  Giovanni Romeo,et al.  SOX10 mutations in patients with Waardenburg-Hirschsprung disease , 1998, Nature Genetics.

[36]  P. Parsons,et al.  Redox regulation of Brn-2/N-Oct-3 POU domain DNA binding activity and proteolytic formation of N-Oct-5 during melanoma cell nuclear extraction , 1998, Melanoma research.

[37]  E. Price,et al.  MAP kinase links the transcription factor Microphthalmia to c-Kit signalling in melanocytes , 1998, Nature.

[38]  J. Schreiber,et al.  Redundancy of Class III POU Proteins in the Oligodendrocyte Lineage* , 1997, The Journal of Biological Chemistry.

[39]  A. Sinclair,et al.  A male-specific role for SOX9 in vertebrate sex determination. , 1996, Development.

[40]  G. Sutherland,et al.  The brn-2 gene regulates the melanocytic phenotype and tumorigenic potential of human melanoma cells. , 1995, Oncogene.

[41]  G. Muscat,et al.  Trans-activation and DNA-binding properties of the transcription factor, Sox-18. , 1995, Nucleic acids research.

[42]  Sahar Mansour,et al.  Campomelic dysplasia and autosomal sex reversal caused by mutations in an SRY-related gene , 1994, Nature.

[43]  R. Spritz.  The molecular basis of human piebaldism. , 1994, Pigment cell research.

[44]  H. Clevers,et al.  Sox‐4, an Sry‐like HMG box protein, is a transcriptional activator in lymphocytes. , 1993, The EMBO journal.

[45]  P. Parsons,et al.  In vivo and in vitro expression of octamer binding proteins in human melanoma metastases, brain tissue, and fibroblasts. , 1993, Pigment cell research.

[46]  N. Heintz,et al.  Histone gene transcription factor binding in extracts of normal human cells , 1991, Molecular and cellular biology.

[47]  P. Parsons,et al.  A melanoma octamer binding protein is responsive to differentiating agents. , 1991, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

[48]  W. Herr,et al.  OBP100 binds remarkably degenerate octamer motifs through specific interactions with flanking sequences. , 1988, Genes & development.

[49]  M. Raff,et al.  All classes of intermediate filaments share a common antigenic determinant defined by a monoclonal antibody , 1981, Cell.

[50]  M. Wegner,et al.  Identification of Sox8 as a modifier gene in a mouse model of Hirschsprung disease reveals underlying molecular defect. , 2005, Developmental biology.

[51]  P. Meltzer,et al.  High frequency of BRAF mutations in nevi , 2003, Nature Genetics.

[52]  W. Pavan,et al.  SOX10 mutation disrupts neural crest development in Dom Hirschsprung mouse model , 1998, Nature Genetics.

[53]  R. Merchant,et al.  Primary brain tumors differ in their expression of octamer deoxyribonucleic acid-binding transcription factors from long-term cultured glioma cell lines. , 1994, Neurosurgery.