Dysregulated Type I Interferon and Inflammatory Monocyte-Macrophage Responses Cause Lethal Pneumonia in SARS-CoV-Infected Mice
暂无分享,去创建一个
David K. Meyerholz | Rahul Vijay | Matthias Mack | Jincun Zhao | S. Perlman | R. Vijay | Anthony R. Fehr | D. Meyerholz | M. Mack | R. Channappanavar | Jincun Zhao | Stanley Perlman | Rudragouda Channappanavar | Rudragouda Channappanavar
[1] L. Hennighausen,et al. Induction of Alternatively Activated Macrophages Enhances Pathogenesis during Severe Acute Respiratory Syndrome Coronavirus Infection , 2012, Journal of Virology.
[2] H. Feldmann,et al. Lymphopenia Associated with Highly Virulent H5N1 Virus Infection Due to Plasmacytoid Dendritic Cell–Mediated Apoptosis of T Cells , 2014, The Journal of Immunology.
[3] S. Akira,et al. Alveolar macrophage–derived type I interferons orchestrate innate immunity to RSV through recruitment of antiviral monocytes , 2015, The Journal of experimental medicine.
[4] L. Enjuanes,et al. Combined action of type I and type III interferon restricts initial replication of severe acute respiratory syndrome coronavirus in the lung but fails to inhibit systemic virus spread. , 2012, The Journal of general virology.
[5] K. Legge,et al. Lymph node dendritic cells control CD8+ T cell responses through regulated FasL expression. , 2005, Immunity.
[6] S. Akira,et al. Control of coronavirus infection through plasmacytoid dendritic-cell–derived type I interferon , 2007, Blood.
[7] R. Schreiber,et al. Timing and magnitude of type I interferon responses by distinct sensors impact CD8 T cell exhaustion and chronic viral infection. , 2012, Cell host & microbe.
[8] K. Subbarao,et al. The Immunobiology of SARS*. , 2007, Annual review of immunology.
[9] E. Ramsburg,et al. CCR2+ Monocyte-Derived Dendritic Cells and Exudate Macrophages Produce Influenza-Induced Pulmonary Immune Pathology and Mortality1 , 2008, The Journal of Immunology.
[10] A. Sher,et al. Type I interferons in infectious disease , 2015, Nature Reviews Immunology.
[11] R. Randall,et al. Influenza virus activation of the interferon system , 2015, Virus research.
[12] Wei Cui,et al. Expression of Lymphocytes and Lymphocyte Subsets in Patients with Severe Acute Respiratory Syndrome , 2003, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.
[13] B. Aronow,et al. Blockade of Chronic Type I Interferon Signaling to Control Persistent LCMV Infection , 2013, Science.
[14] K. Okumura,et al. Type I Interferons (IFNs) Regulate Tumor Necrosis Factor–related Apoptosis-inducing Ligand (TRAIL) Expression on Human T Cells: A Novel Mechanism for the Antitumor Effects of Type I IFNs , 1999, The Journal of experimental medicine.
[15] Malik Peiris,et al. Middle East respiratory syndrome , 2015, The Lancet.
[16] Scott A. Brown,et al. TNF/iNOS-producing dendritic cells are the necessary evil of lethal influenza virus infection , 2009, Proceedings of the National Academy of Sciences.
[17] Ali Danesh,et al. Interferon-Mediated Immunopathological Events Are Associated with Atypical Innate and Adaptive Immune Responses in Patients with Severe Acute Respiratory Syndrome , 2007, Journal of Virology.
[18] E. Boritz,et al. Type I interferon responses in rhesus macaques prevent SIV infection and slow disease progression , 2014, Nature.
[19] K. Kuchler,et al. Type I Interferons Promote Fatal Immunopathology by Regulating Inflammatory Monocytes and Neutrophils during Candida Infections , 2012, PLoS pathogens.
[20] Jason R. Madsen,et al. Single-dose intranasal administration with mDEF201 (adenovirus vectored mouse interferon-alpha) confers protection from mortality in a lethal SARS-CoV BALB/c mouse model , 2010, Antiviral Research.
[21] Ton de Jong,et al. Pegylated interferon-α protects type 1 pneumocytes against SARS coronavirus infection in macaques , 2004, Nature Medicine.
[22] T. Kuiken,et al. The Pathology and Pathogenesis of Experimental Severe Acute Respiratory Syndrome and Influenza in Animal Models , 2014, Journal of Comparative Pathology.
[23] Jincun Zhao,et al. T Cell Responses Are Required for Protection from Clinical Disease and for Virus Clearance in Severe Acute Respiratory Syndrome Coronavirus-Infected Mice , 2010, Journal of Virology.
[24] Ralph Baric,et al. A Mouse-Adapted SARS-Coronavirus Causes Disease and Mortality in BALB/c Mice , 2007, PLoS pathogens.
[25] E. Unanue,et al. Type I Interferon Sensitizes Lymphocytes to Apoptosis and Reduces Resistance to Listeria Infection , 2004, The Journal of experimental medicine.
[26] R. Baric,et al. SARS coronavirus pathogenesis: host innate immune responses and viral antagonism of interferon , 2012, Current Opinion in Virology.
[27] Ralph S. Baric,et al. SARS-CoV Pathogenesis Is Regulated by a STAT1 Dependent but a Type I, II and III Interferon Receptor Independent Mechanism , 2010, PLoS pathogens.
[28] Lisa E. Gralinski,et al. Molecular pathology of emerging coronavirus infections , 2014, The Journal of pathology.
[29] E. Pamer,et al. Monocyte recruitment during infection and inflammation , 2011, Nature Reviews Immunology.
[30] I. Campbell,et al. Inflammatory monocytes and the pathogenesis of viral encephalitis , 2012, Journal of Neuroinflammation.
[31] R. Schreiber,et al. Persistent LCMV Infection Is Controlled by Blockade of Type I Interferon Signaling , 2013, Science.
[32] Y. Lau,et al. Chemokine up-regulation in SARS-coronavirus–infected, monocyte-derived human dendritic cells , 2005, Blood.
[33] A. Danchin,et al. The Severe Acute Respiratory Syndrome , 2003 .
[34] Y. Guan,et al. Cytokine Responses in Severe Acute Respiratory Syndrome Coronavirus-Infected Macrophages In Vitro: Possible Relevance to Pathogenesis , 2005, Journal of Virology.
[35] Lisa E. Gralinski,et al. A SARS-like cluster of circulating bat coronaviruses shows potential for human emergence , 2015, Nature Medicine.
[36] G. Trinchieri,et al. Type I interferon: friend or foe? , 2010, The Journal of experimental medicine.
[37] Frederick Klauschen,et al. A Systems Analysis Identifies a Feedforward Inflammatory Circuit Leading to Lethal Influenza Infection , 2013, Cell.
[38] Jincun Zhao,et al. Evasion by Stealth: Inefficient Immune Activation Underlies Poor T Cell Response and Severe Disease in SARS-CoV-Infected Mice , 2009, PLoS pathogens.
[39] Jincun Zhao,et al. T cell-mediated immune response to respiratory coronaviruses , 2014, Immunologic Research.
[40] A. Osterhaus,et al. Isolation of a novel coronavirus from a man with pneumonia in Saudi Arabia. , 2012, The New England journal of medicine.
[41] Hong Tang,et al. Adaptive immune cells temper initial innate responses , 2007, Nature Medicine.
[42] E. Pamer,et al. TNF/iNOS-producing dendritic cells mediate innate immune defense against bacterial infection. , 2003, Immunity.
[43] M. Mack,et al. Unique and redundant functions of NKp46+ ILC3s in models of intestinal inflammation , 2015, The Journal of experimental medicine.
[44] Junya Fukuoka,et al. Lung pathology of severe acute respiratory syndrome (SARS): a study of 8 autopsy cases from Singapore , 2003, Human Pathology.
[45] T. Mitchell,et al. Monocytes Regulate the Mechanism of T-cell Death by Inducing Fas-Mediated Apoptosis during Bacterial Infection , 2012, PLoS pathogens.
[46] A. Sher,et al. Host-directed therapy of tuberculosis based on interleukin-1 and type I interferon crosstalk , 2014, Nature.
[47] H. Brühl,et al. Expression and Characterization of the Chemokine Receptors CCR2 and CCR5 in Mice1 , 2001, The Journal of Immunology.
[48] S. Akira,et al. Type I Interferon Signaling Regulates Ly6Chi Monocytes and Neutrophils during Acute Viral Pneumonia in Mice , 2011, PLoS pathogens.
[49] Yi Guan,et al. Lung pathology of fatal severe acute respiratory syndrome , 2003, The Lancet.
[50] Bo Zhang,et al. Multiple organ infection and the pathogenesis of SARS , 2005, The Journal of experimental medicine.
[51] A. Dzutsev,et al. Cell Depletion in Mice That Express Diphtheria Toxin Receptor under the Control of SiglecH Encompasses More Than Plasmacytoid Dendritic Cells , 2014, The Journal of Immunology.
[52] Thomas O. Metz,et al. Pathogenic Influenza Viruses and Coronaviruses Utilize Similar and Contrasting Approaches To Control Interferon-Stimulated Gene Responses , 2014, mBio.
[53] Javier A. Carrero. Confounding roles for type I interferons during bacterial and viral pathogenesis , 2013, International immunology.
[54] R. Schreiber,et al. Bone Marrow Stromal Cell Antigen 2 Is a Specific Marker of Type I IFN-Producing Cells in the Naive Mouse, but a Promiscuous Cell Surface Antigen following IFN Stimulation1 , 2006, The Journal of Immunology.
[55] Kapil Bahl,et al. Type 1 Interferons and Antiviral CD8 T-Cell Responses , 2012, PLoS pathogens.
[56] F. Weber,et al. Interaction of severe acute respiratory syndrome-associated coronavirus with dendritic cells. , 2006, The Journal of general virology.
[57] Ali Danesh,et al. Human immunopathogenesis of severe acute respiratory syndrome (SARS) , 2007, Virus Research.
[58] L. Poon,et al. Initial viral load and the outcomes of SARS , 2004, Canadian Medical Association Journal.
[59] Sophia Davidson,et al. Pathogenic potential of interferon αβ in acute influenza infection , 2014, Nature Communications.
[60] J. Epstein,et al. Isolation and characterization of a bat SARS-like coronavirus that uses the ACE2 receptor , 2013, Nature.
[61] J. Sung,et al. Plasma inflammatory cytokines and chemokines in severe acute respiratory syndrome , 2004, Clinical and experimental immunology.
[62] D. Portnoy,et al. Mice Lacking the Type I Interferon Receptor Are Resistant to Listeria monocytogenes , 2004, The Journal of experimental medicine.
[63] S. Akira,et al. Type-I Interferon is Critical for FasL Expression on Lung Cells to Determine the Severity of Influenza , 2013, PloS one.
[64] C. Hsiao,et al. Modeling the Early Events of Severe Acute Respiratory Syndrome Coronavirus Infection In Vitro , 2006, Journal of Virology.
[65] Jincun Zhao,et al. Age-related increases in PGD(2) expression impair respiratory DC migration, resulting in diminished T cell responses upon respiratory virus infection in mice. , 2011, The Journal of clinical investigation.
[66] B. Ludewig,et al. Type I IFN-Mediated Protection of Macrophages and Dendritic Cells Secures Control of Murine Coronavirus Infection1 , 2009, The Journal of Immunology.
[67] Geethamma Jolly,et al. Middle East Respiratory Syndrome Coronavirus (MERS-CoV) , 2016 .
[68] M. Katze,et al. Transcriptomic Analysis Reveals a Mechanism for a Prefibrotic Phenotype in STAT1 Knockout Mice during Severe Acute Respiratory Syndrome Coronavirus Infection , 2010, Journal of Virology.