IL-1β Promotes Antimicrobial Immunity in Macrophages by Regulating TNFR Signaling and Caspase-3 Activation
暂无分享,去创建一个
Ana C Anderson | V. Kuchroo | S. Behar | Vijay K Kuchroo | A. Anderson | H. Remold | Pushpa Jayaraman | Tomoyasu Nishimura | Isabel Sada-Ovalle | Heinz G Remold | Samuel M Behar | Pushpa Jayaraman | T. Nishimura | I. Sada-Ovalle
[1] J. Sedgwick,et al. Structural deficiencies in granuloma formation in TNF gene-targeted mice underlie the heightened susceptibility to aerosol Mycobacterium tuberculosis infection, which is not compensated for by lymphotoxin. , 1999, Journal of immunology.
[2] Y. Iwakura,et al. Protective role of interleukin-1 in mycobacterial infection in IL-1 alpha/beta double-knockout mice. , 2000, Laboratory investigation; a journal of technical methods and pathology.
[3] Minjian Chen,et al. Lipid mediators in innate immunity against tuberculosis: opposing roles of PGE2 and LXA4 in the induction of macrophage death , 2008, The Journal of experimental medicine.
[4] 加塩 裕美子. Galectin-9 induces apoptosis through calcium-calpain-caspase-1 pathway , 2003 .
[5] M. Karin,et al. Caspase 1-independent activation of interleukin-1beta in neutrophil-predominant inflammation. , 2009, Arthritis and rheumatism.
[6] J. Mudgett,et al. Identification of nitric oxide synthase as a protective locus against tuberculosis. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[7] C. Dinarello,et al. Immunological and inflammatory functions of the interleukin-1 family. , 2009, Annual review of immunology.
[8] S. Yagihashi,et al. Interferon‐γ stimulates the expression of galectin‐9 in cultured human endothelial cells , 2002, Journal of Leukocyte Biology.
[9] B. Ryffel,et al. IL-1 Receptor-Mediated Signal Is an Essential Component of MyD88-Dependent Innate Response to Mycobacterium tuberculosis Infection , 2007, The Journal of Immunology.
[10] Tatyana Chernova,et al. Th1-specific cell surface protein Tim-3 regulates macrophage activation and severity of an autoimmune disease , 2002, Nature.
[11] Gregory A. Taylor,et al. Immune Control of Tuberculosis by IFN-γ-Inducible LRG-47 , 2003, Science.
[12] D. Kirschner,et al. Tumor Necrosis Factor Blockade in Chronic Murine Tuberculosis Enhances Granulomatous Inflammation and Disorganizes Granulomas in the Lungs , 2008, Infection and Immunity.
[13] S. Mizuno,et al. Role of Interleukin (IL)‐1 Type 1 Receptor in Mycobacterial Infection , 2001, Microbiology and immunology.
[14] S. Nagata,et al. Identification of Tim4 as a phosphatidylserine receptor , 2007, Nature.
[15] A. Sher,et al. Cutting Edge: Caspase-1 Independent IL-1β Production Is Critical for Host Resistance to Mycobacterium tuberculosis and Does Not Require TLR Signaling In Vivo , 2010, The Journal of Immunology.
[16] J. Keane,et al. Pathogenic Mycobacterium tuberculosis evades apoptosis of host macrophages by release of TNF-R2, resulting in inactivation of TNF-alpha. , 1998, Journal of immunology.
[17] J. Finkelstein,et al. The Effects of Interleukin-1β in Tumor Necrosis Factor-α-Induced Acute Pulmonary Inflammation in Mice , 2009, Mediators of inflammation.
[18] S. Yagihashi,et al. Interferon-gamma stimulates the expression of galectin-9 in cultured human endothelial cells. , 2002, Journal of leukocyte biology.
[19] Yaping Zhang,et al. Interleukin-1beta up-regulates tumor necrosis factor receptors in the mouse airways. , 2008, Pulmonary pharmacology & therapeutics.
[20] V. Kuchroo,et al. Tim3 binding to galectin-9 stimulates antimicrobial immunity , 2010, The Journal of experimental medicine.
[21] I. Tabas,et al. The role of macrophages and dendritic cells in the clearance of apoptotic cells in advanced atherosclerosis , 2011, European journal of immunology.
[22] J. Mckinney,et al. Immune control of tuberculosis by IFN-gamma-inducible LRG-47. , 2003, Science.
[23] B. Nandi,et al. Regulation of neutrophils by interferon-γ limits lung inflammation during tuberculosis infection , 2011, The Journal of experimental medicine.
[24] S. Fortune,et al. Efferocytosis is an innate antibacterial mechanism. , 2012, Cell host & microbe.
[25] D. Green,et al. T and B cell hyperactivity and autoimmunity associated with niche-specific defects in apoptotic body clearance in TIM-4-deficient mice , 2010, Proceedings of the National Academy of Sciences.
[26] Ingo Schmitz,et al. Differential Modulation of Apoptosis Sensitivity in CD95 Type I and Type II Cells* , 1999, The Journal of Biological Chemistry.
[27] C. Lowenstein,et al. Tumor necrosis factor-alpha is required in the protective immune response against Mycobacterium tuberculosis in mice. , 1995, Immunity.
[28] M. Hirashima,et al. Interleukin-1β stimulates galectin-9 expression in human astrocytes , 2001, Neuroreport.
[29] R. Das,et al. A Family of IFN-γ–Inducible 65-kD GTPases Protects Against Bacterial Infection , 2011, Science.
[30] J. Flynn,et al. Tumor necrosis factor and chemokine interactions in the formation and maintenance of granulomas in tuberculosis. , 2005, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.
[31] S. Akira,et al. Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function. , 1998, Immunity.
[32] V. Kuchroo,et al. The Tim-3 ligand galectin-9 negatively regulates T helper type 1 immunity , 2005, Nature Immunology.
[33] A. Sher,et al. Innate and adaptive interferons suppress IL-1α and IL-1β production by distinct pulmonary myeloid subsets during Mycobacterium tuberculosis infection. , 2011, Immunity.
[34] J. Flynn,et al. An essential role for interferon gamma in resistance to Mycobacterium tuberculosis infection , 1993, The Journal of experimental medicine.
[35] Valeria Panebianco,et al. Supplementary Figure 2 , 2012 .
[36] A. Chiba,et al. Innate Invariant NKT Cells Recognize Mycobacterium tuberculosis–Infected Macrophages, Produce Interferon-γ, and Kill Intracellular Bacteria , 2008, PLoS pathogens.
[37] J. Finkelstein,et al. IL-1beta augments TNF-alpha-mediated inflammatory responses from lung epithelial cells. , 2009, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[38] V. Kuchroo,et al. Tim-3 inhibits T helper type 1–mediated auto- and alloimmune responses and promotes immunological tolerance , 2003, Nature Immunology.
[39] Hardy Kornfeld,et al. Virulent Mycobacterium tuberculosis Strains Evade Apoptosis of Infected Alveolar Macrophages1 , 2000, The Journal of Immunology.
[40] R. Hunter,et al. Pathology of postprimary tuberculosis in humans and mice: contradiction of long-held beliefs. , 2007, Tuberculosis.
[41] I. Orme,et al. Disseminated tuberculosis in interferon gamma gene-disrupted mice , 1993, The Journal of experimental medicine.
[42] Y. Iwakura,et al. Protective Role of Interleukin-1 in Mycobacterial Infection in IL-1 α/β Double-Knockout Mice , 2000, Laboratory Investigation.
[43] D. Underhill,et al. Faculty Opinions recommendation of Caspase-1 independent IL-1beta production is critical for host resistance to mycobacterium tuberculosis and does not require TLR signaling in vivo. , 2010 .
[44] D. MacEwan. TNF ligands and receptors – a matter of life and death , 2002, British journal of pharmacology.
[45] Hannah E. Volkman,et al. Tumor necrosis factor signaling mediates resistance to mycobacteria by inhibiting bacterial growth and macrophage death. , 2008, Immunity.
[46] G. Freeman,et al. Interaction of Tim-3 and Tim-3 ligand regulates T helper type 1 responses and induction of peripheral tolerance , 2003, Nature Immunology.
[47] T. van der Poll,et al. Interleukin-1 signaling is essential for host defense during murine pulmonary tuberculosis. , 2000, The Journal of infectious diseases.
[48] H. Haas,et al. Guanidinoethyl sulphonate is a glycine receptor antagonist in striatum , 2002, British journal of pharmacology.