MK-801 ameliorates delayed amnesia, but potentiates acute amnesia induced by CO

[1]  V. Beral,et al.  ORAL CONTRACEPTION AND GENITAL TRACT MALIGNANCY , 1989, The Lancet.

[2]  R. Čapek,et al.  Phencyclidine suppresses hippocampal long-term potentiation through stereospecific activation of phencyclidine receptors , 1989, Neuropharmacology.

[3]  J. Penney,et al.  Anatomic correlation of NMDA and 3H-TCP-labeled receptors in rat brain , 1988, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[4]  R. Gill,et al.  Systemic administration of MK-801 protects against ischemia-induced hippocampal neurodegeneration in the gerbil , 1987, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[5]  S. Ando,et al.  Delayed memory dysfunction by transient hypoxia, and its prevention with forskolin , 1987, Brain Research.

[6]  A. L. Horowitz,et al.  Carbon monoxide toxicity: MR imaging in the brain. , 1987, Radiology.

[7]  L. Iversen,et al.  The anticonvulsant MK-801 is a potent N-methyl-D-aspartate antagonist. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[8]  Y. Takahashi,et al.  COMPUTERISED TOMOGRAPHY AS AN INDICATION OF LONG-TERM OUTCOME AFTER ACUTE CARBON MONOXIDE POISONING , 1980, The Lancet.

[9]  G. Lynch,et al.  Selective impairment of learning and blockade of long-term potentiation by an N-methyl-D-aspartate receptor antagonist, AP5 , 1986, Nature.

[10]  M. Ginsberg Delayed neurological deterioration following hypoxia. , 1979, Advances in neurology.

[11]  M. Fardeau,et al.  The central nervous system and carbon monoxide poisoning. II. Anatomical study of brain lesions following intoxication with carbon monixide (22 cases). , 1967, Progress in brain research.