A genetic screen to identify genes that rescue the slow growth phenotype of c-myc null fibroblasts
暂无分享,去创建一个
G. Daley | R. Bernards | K. Berns | E. M. Hijmans | E. Koh
[1] J F Barrett,et al. Identification of CDK4 as a target of c-MYC. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[2] W. Ansorge,et al. Direct induction of cyclin D2 by Myc contributes to cell cycle progression and sequestration of p27 , 1999, The EMBO journal.
[3] K. Helin,et al. CDC25A Phosphatase Is a Target of E2F and Is Required for Efficient E2F-Induced S Phase , 1999, Molecular and Cellular Biology.
[4] John M. Sedivy,et al. c-Myc Regulates Cyclin D-Cdk4 and -Cdk6 Activity but Affects Cell Cycle Progression at Multiple Independent Points , 1999, Molecular and Cellular Biology.
[5] J. Sedivy,et al. Transactivation-defective c-MycS retains the ability to regulate proliferation and apoptosis. , 1998, Genes & development.
[6] M. Cole,et al. c-myc null cells misregulate cad and gadd45 but not other proposed c-Myc targets. , 1998, Genes & development.
[7] J. Sedivy,et al. Phenotypes of c-Myc-deficient rat fibroblasts isolated by targeted homologous recombination. , 1997, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[8] C. Dang,et al. Identification of putative c-Myc-responsive genes: characterization of rcl, a novel growth-related gene , 1997, Molecular and cellular biology.
[9] R. Bernards,et al. Repression of c-Myc responsive genes in cycling cells causes G1 arrest through reduction of cyclin E/CDK2 kinase activity , 1997, Oncogene.
[10] A. Fornace,et al. Myc represses the growth arrest gene gadd45 , 1997, Oncogene.
[11] Andreas Sewing,et al. Myc activation of cyclin E/Cdk2 kinase involves induction of cyclin E gene transcription and inhibition of p27Kip1 binding to newly formed complexes , 1997, Oncogene.
[12] D. Beach,et al. Cdc25 cell-cycle phosphatase as a target of c-myc , 1996, Nature.
[13] R. Eisenman,et al. Myc‐Max heterodimers activate a DEAD box gene and interact with multiple E box‐related sites in vivo. , 1996, The EMBO journal.
[14] I. Rosenwald,et al. Upregulated expression of the genes encoding translation initiation factors eIF-4E and eIF-2alpha in transformed cells. , 1996, Cancer letters.
[15] M. Serrano,et al. A p16INK4a-insensitive CDK4 mutant targeted by cytolytic T lymphocytes in a human melanoma , 1995, Science.
[16] P. Farnham,et al. An E-box-mediated increase in cad transcription at the G1/S-phase boundary is suppressed by inhibitory c-Myc mutants , 1995, Molecular and cellular biology.
[17] R. DePinho,et al. Inhibition of ras-induced proliferation and cellular transformation by p16INK4 , 1995, Science.
[18] G. Prendergast,et al. c‐Myc represses transcription in vivo by a novel mechanism dependent on the initiator element and Myc box II. , 1994, The EMBO journal.
[19] J L Cleveland,et al. The ornithine decarboxylase gene is a transcriptional target of c-Myc. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[20] V. Rotter,et al. c-Myc trans-activates the p53 promoter through a required downstream CACGTG motif. , 1993, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[21] S. Epstein,et al. Inhibitory effects of antisense oligodeoxynucleotides targeting c-myc mRNA on smooth muscle cell proliferation and migration. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[22] R. Eisenman,et al. Myc and Max proteins possess distinct transcriptional activities , 1992, Nature.
[23] R. Ralston. Complementation of transforming domains in E1a/myc chimaeras , 1991, Nature.
[24] J. Bishop,et al. The MYC protein activates transcription of the alpha‐prothymosin gene. , 1991, The EMBO journal.
[25] J. Barrett,et al. An amino-terminal c-myc domain required for neoplastic transformation activates transcription , 1990, Molecular and cellular biology.
[26] H. Land,et al. Advanced mammalian gene transfer: high titre retroviral vectors with multiple drug selection markers and a complementary helper-free packaging cell line. , 1990, Nucleic acids research.
[27] P. Yaciuk,et al. TGF-β1 inhibition of c-myc transcription and growth in keratinocytes is abrogated by viral transforming proteins with pRB binding domains , 1990, Cell.
[28] L. Penn,et al. Negative autoregulation of c‐myc transcription. , 1990, The EMBO journal.
[29] E. Prochownik,et al. c-myc antisense transcripts accelerate differentiation and inhibit G1 progression in murine erythroleukemia cells , 1988, Molecular and cellular biology.
[30] H. Varmus,et al. Definition of regions in human c-myc that are involved in transformation and nuclear localization , 1987, Molecular and cellular biology.
[31] R. Weinberg,et al. N-myc amplification causes down-modulation of MHC class I antigen expression in neuroblastoma , 1986, Cell.
[32] A. Kimchi,et al. Close link between reduction of c-myc expression by interferon and G0/G1 arrest , 1985, Nature.
[33] P. Leder,et al. Cell-specific regulation of the c-myc gene by lymphocyte mitogens and platelet-derived growth factor , 1983, Cell.
[34] M. Henriksson,et al. Proteins of the Myc network: essential regulators of cell growth and differentiation. , 1996, Advances in cancer research.
[35] Eric Wickstrom,et al. A c-myc antisense oligodeoxynucleotide inhibits entry into S phase but not progress from G0 to G1 , 1987, Nature.