Essential role of mda-5 in type I IFN responses to polyriboinosinic:polyribocytidylic acid and encephalomyocarditis picornavirus.
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Richard A Flavell | M. Diamond | R. Flavell | B. Beutler | M. Colonna | M. Cella | W. Barchet | S. Gilfillan | Bruce Beutler | Michael S Diamond | Marco Colonna | Leonid Gitlin | Marina Cella | Susan Gilfillan | Winfried Barchet | Leonid Gitlin
[1] A. Aguzzi,et al. Deficient signaling in mice devoid of double‐stranded RNA‐dependent protein kinase. , 1995, The EMBO journal.
[2] K. Rajewsky,et al. A cre-transgenic mouse strain for the ubiquitous deletion of loxP-flanked gene segments including deletion in germ cells. , 1995, Nucleic acids research.
[3] A. C. Palmenberg,et al. The immunogenic and pathogenic potential of short poly(C) tract Mengo viruses. , 1996, Virology.
[4] G. Montelione,et al. RNA binding by the novel helical domain of the influenza virus NS1 protein requires its dimer structure and a small number of specific basic amino acids. , 1999, RNA.
[5] T. Taniguchi,et al. Distinct and Essential Roles of Transcription Factors IRF-3 and IRF-7 in Response to Viruses for IFN-α/β Gene Induction , 2000 .
[6] R. Flavell,et al. Recognition of double-stranded RNA and activation of NF-κB by Toll-like receptor 3 , 2001, Nature.
[7] C. Janeway,et al. Innate immune recognition. , 2002, Annual review of immunology.
[8] Paul B. Fisher,et al. mda-5: An interferon-inducible putative RNA helicase with double-stranded RNA-dependent ATPase activity and melanoma growth-suppressive properties , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[9] F. Martinon,et al. Overexpression of Helicard, a CARD-Containing Helicase Cleaved during Apoptosis, Accelerates DNA Degradation , 2002, Current Biology.
[10] G. Barber,et al. The dsRNA binding protein family: critical roles, diverse cellular functions , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[11] Richard A Flavell,et al. Upregulation of costimulatory molecules induced by lipopolysaccharide and double-stranded RNA occurs by Trif-dependent and Trif-independent pathways , 2003, Nature Immunology.
[12] A. Yamamoto,et al. Subcellular Localization of Toll-Like Receptor 3 in Human Dendritic Cells 1 , 2003, The Journal of Immunology.
[13] Guo-Ping Zhou,et al. Triggering the Interferon Antiviral Response Through an IKK-Related Pathway , 2003, Science.
[14] S. Akira,et al. Toll‐like receptor expression in murine DC subsets: lack of TLR7 expression by CD8α+ DC correlates with unresponsiveness to imidazoquinolines , 2003, European journal of immunology.
[15] S. Akira,et al. Role of Adaptor TRIF in the MyD88-Independent Toll-Like Receptor Signaling Pathway , 2003, Science.
[16] A. García-Sastre,et al. A Recombinant Influenza A Virus Expressing anRNA-Binding-Defective NS1 Protein Induces High Levels of BetaInterferon and Is Attenuated inMice , 2003, Journal of Virology.
[17] Jiahuai Han,et al. Identification of Lps2 as a key transducer of MyD88-independent TIR signalling , 2003, Nature.
[18] T. Maniatis,et al. IKKε and TBK1 are essential components of the IRF3 signaling pathway , 2003, Nature Immunology.
[19] Shizuo Akira,et al. The RNA helicase RIG-I has an essential function in double-stranded RNA-induced innate antiviral responses , 2004, Nature Immunology.
[20] S. Goodbourn,et al. The V proteins of paramyxoviruses bind the IFN-inducible RNA helicase, mda-5, and inhibit its activation of the IFN-beta promoter. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[21] Osamu Takeuchi,et al. The Roles of Two IκB Kinase-related Kinases in Lipopolysaccharide and Double Stranded RNA Signaling and Viral Infection , 2004, The Journal of experimental medicine.
[22] E. Fikrig,et al. Toll-like receptor 3 mediates West Nile virus entry into the brain causing lethal encephalitis , 2004, Nature Medicine.
[23] K. Tyler,et al. Does Toll-like receptor 3 play a biological role in virus infections? , 2004, Virology.
[24] R. Flavell,et al. TLR-Independent Induction of Dendritic Cell Maturation and Adaptive Immunity by Negative-Strand RNA Viruses1 , 2004, The Journal of Immunology.
[25] S. Pestka,et al. Expression analysis and genomic characterization of human melanoma differentiation associated gene-5, mda-5: a novel type I interferon-responsive apoptosis-inducing gene , 2004, Oncogene.
[26] S. Lemon,et al. Regulating Intracellular Antiviral Defense and Permissiveness to Hepatitis C Virus RNA Replication through a Cellular RNA Helicase, RIG-I , 2005, Journal of Virology.
[27] K. Honda,et al. Regulation of the type I IFN induction: a current view. , 2005, International immunology.
[28] A. Bowie,et al. TLR3 in antiviral immunity: key player or bystander? , 2005, Trends in immunology.
[29] Shizuo Akira,et al. Shared and Unique Functions of the DExD/H-Box Helicases RIG-I, MDA5, and LGP2 in Antiviral Innate Immunity1 , 2005, The Journal of Immunology.
[30] S. Akira,et al. Inhibition of RIG-I-Dependent Signaling to the Interferon Pathway during Hepatitis C Virus Expression and Restoration of Signaling by IKKε , 2005, Journal of Virology.
[31] Osamu Takeuchi,et al. Cell type-specific involvement of RIG-I in antiviral response. , 2005, Immunity.
[32] C. Duckett,et al. Differential Role for TLR3 in Respiratory Syncytial Virus-Induced Chemokine Expression , 2005, Journal of Virology.
[33] Z. Zhai,et al. VISA Is an Adapter Protein Required for Virus-Triggered IFN-β Signaling , 2005 .
[34] Osamu Takeuchi,et al. IPS-1, an adaptor triggering RIG-I- and Mda5-mediated type I interferon induction , 2005, Nature Immunology.
[35] Ralf Bartenschlager,et al. Cardif is an adaptor protein in the RIG-I antiviral pathway and is targeted by hepatitis C virus , 2005, Nature.
[36] S. Lemon,et al. Control of antiviral defenses through hepatitis C virus disruption of retinoic acid-inducible gene-I signaling. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[37] Zhijian J. Chen,et al. Hepatitis C virus protease NS3/4A cleaves mitochondrial antiviral signaling protein off the mitochondria to evade innate immunity. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[38] S. Akira,et al. The RNA Helicase Lgp2 Inhibits TLR-Independent Sensing of Viral Replication by Retinoic Acid-Inducible Gene-I1 , 2005, The Journal of Immunology.
[39] R. Flavell,et al. Toll-like receptor 3 promotes cross-priming to virus-infected cells , 2005, Nature.
[40] S. Akira,et al. Involvement of Toll-like Receptor 3 in the Immune Response of Lung Epithelial Cells to Double-stranded RNA and Influenza A Virus* , 2005, Journal of Biological Chemistry.
[41] M. Colonna,et al. Dendritic cells respond to influenza virus through TLR7‐ and PKR‐independent pathways , 2005, European journal of immunology.
[42] Zhijian J. Chen,et al. Identification and Characterization of MAVS, a Mitochondrial Antiviral Signaling Protein that Activates NF-κB and IRF3 , 2005, Cell.
[43] S. Akira,et al. Pathogen Recognition and Innate Immunity , 2006, Cell.
[44] M. Mann,et al. Specificity in Toll-like receptor signalling through distinct effector functions of TRAF3 and TRAF6 , 2006, Nature.
[45] M. Gale,et al. West Nile Virus Evades Activation of Interferon Regulatory Factor 3 through RIG-I-Dependent and -Independent Pathways without Antagonizing Host Defense Signaling , 2006, Journal of Virology.
[46] A. Shahangian,et al. Critical role of TRAF3 in the Toll-like receptor-dependent and -independent antiviral response , 2006, Nature.