Down-regulation of MHC class II receptors of DH82 cells, following infection with Ehrlichia canis.
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[1] M. Bendinelli,et al. Rickettsial Infection and Immunity , 2013, Infectious Agents and Pathogenesis.
[2] G. Winslow,et al. Antibodies Highly Effective in SCID Mice During Infection by the Intracellular Bacterium Ehrlichia chaffeensis Are of Picomolar Affinity and Exhibit Preferential Epitope and Isotype Utilization1 , 2002, The Journal of Immunology.
[3] D. Charron,et al. MHC class II isotype‐specific signaling complex on human B cells , 2002, European journal of immunology.
[4] R. Ganta,et al. Persistent Ehrlichia chaffeensis Infection Occurs in the Absence of Functional Major Histocompatibility Complex Class II Genes , 2002, Infection and Immunity.
[5] S. Ray,et al. Reorganization of genera in the families Rickettsiaceae and Anaplasmataceae in the order Rickettsiales: unification of some species of Ehrlichia with Anaplasma, Cowdria with Ehrlichia and Ehrlichia with Neorickettsia, descriptions of six new species combinations and designation of Ehrlichia equi and , 2001, International journal of systematic and evolutionary microbiology.
[6] G. Reddy,et al. Outer Membrane Protein-Specific Monoclonal Antibodies Protect SCID Mice from Fatal Infection by the Obligate Intracellular Bacterial Pathogen Ehrlichia chaffeensis1 , 2001, The Journal of Immunology.
[7] S. Carter,et al. Immunological and inflammatory characterisation of three canine cell lines: K1, K6 and DH82. , 2000, Veterinary immunology and immunopathology.
[8] M. Day. Clinical Immunology of the Dog and Cat , 1999 .
[9] F. Jongejan,et al. Recent Advances in Determining the Pathogenesis of Canine Monocytic Ehrlichiosis , 1999, Journal of Clinical Microbiology.
[10] G. Reddy,et al. Variability in the 28-kDa surface antigen protein multigene locus of isolates of the emerging disease agent Ehrlichia chaffeensis suggests that it plays a role in immune evasion. , 1999, Molecular cell biology research communications : MCBRC.
[11] N. Vachiéry,et al. Inhibition of MHC class I and class II cell surface expression on bovine endothelial cells upon infection with Cowdria ruminantium. , 1998, Veterinary immunology and immunopathology.
[12] J. Foley,et al. Amplification of Ehrlichial DNA from Dogs 34 Months after Infection with Ehrlichia canis , 1998, Journal of Clinical Microbiology.
[13] J. Dawson,et al. Persistent infection of C3H/HeJ mice by Ehrlichia chaffeensis. , 1996, Veterinary microbiology.
[14] J. Dawson,et al. The first isolation, in vitro propagation, and genetic characterization of Ehrlichia canis in Israel. , 1996, Veterinary parasitology.
[15] Y. Rikihisa,et al. Abrogation of gamma interferon-induced inhibition of Ehrlichia chaffeensis infection in human monocytes with iron-transferrin , 1994, Infection and immunity.
[16] W. Bessler,et al. Engagement of major histocompatibility complex class II molecules leads to nitrite production in bone marrow‐derived macrophages , 1993, European journal of immunology.
[17] D. Walker,et al. Interferon-γ and tumor necrosis factor-α exert their antirickettsial effect via induction of synthesis of nitric oxide , 1993 .
[18] H. Winkler,et al. Cytokines Influencing Infections by Rickettsia Species , 2002 .
[19] M. Fellous,et al. Interferon and major histocompatibility complex genes: a model to analyse eukaryotic gene regulation? , 1986, Interferon.