Neuroprotection against Aβ and glutamate toxicity by melatonin: Are GABA receptors involved?

[1]  George Perry,et al.  The neuroprotective activities of melatonin against the Alzheimer β‐protein are not mediated by melatonin membrane receptors , 2002, Journal of pineal research.

[2]  A. Westlind-Danielsson,et al.  Spontaneous in vitro formation of supramolecular beta-amyloid structures, "betaamy balls", by beta-amyloid 1-40 peptide. , 2001, Biochemistry.

[3]  M G Fried,et al.  Host-guest study of left-handed polyproline II helix formation. , 2001, Biochemistry.

[4]  S. Ferreira,et al.  Dual role of glutamatergic neurotransmission on amyloid β1–42 aggregation and neurotoxicity in embryonic avian retina , 2001, Neuroscience Letters.

[5]  Josep M. Guerrero,et al.  Neuroprotection by melatonin from glutamate‐induced excitotoxicity during development of the cerebellum in the chick embryo , 2000, Journal of pineal research.

[6]  R. Reiter,et al.  Melatonin protects hippocampal neurons in vivo against kainic acid‐induced damage in mice , 1998, Journal of neuroscience research.

[7]  C. Soto,et al.  Inhibition of Alzheimer β-Fibrillogenesis by Melatonin* , 1998, The Journal of Biological Chemistry.

[8]  G. Konopka,et al.  Mechanism and prevention of neurotoxicity caused by β-amyloid peptides: relation to Alzheimer's disease , 1997, Brain Research.

[9]  L. Niles,et al.  Mechanisms underlying the antidopaminergic effect of clonazepam and melatonin in striatum , 1997, Neuropharmacology.

[10]  R. Reiter,et al.  Melatonin Prevents Death of Neuroblastoma Cells Exposed to the Alzheimer Amyloid Peptide , 1997, The Journal of Neuroscience.

[11]  L. Niles,et al.  Central-type benzodiazepine receptors mediate the antidopaminergic effect of clonazepam and melatonin in 6-hydroxydopamine lesioned rats: involvement of a GABAergic mechanism. , 1995, The Journal of pharmacology and experimental therapeutics.

[12]  S. Lipton,et al.  Excitatory amino acids as a final common pathway for neurologic disorders. , 1994, The New England journal of medicine.

[13]  M. Mattson,et al.  beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity , 1992, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[14]  G. Konopka,et al.  Mechanism and prevention of neurotoxicity caused by beta-amyloid peptides: relation to Alzheimer's disease. , 1997, Brain research.

[15]  L. Niles Melatonin interaction with the benzodiazepine-GABA receptor complex in the CNS. , 1991, Advances in experimental medicine and biology.