Simvastatin increases neutrophil apoptosis and reduces inflammatory reaction after coronary surgery.
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[1] F. Agrò,et al. Effects of atorvastatin on systemic inflammatory response after coronary bypass surgery , 2006, Critical care medicine.
[2] Shen-Jiang Hu,et al. Effects of simvastatin on cardiohemodynamic responses to ischemia–reperfusion in isolated rat hearts , 2006, Heart and Vessels.
[3] G. Biondi-Zoccai,et al. Myocardial ischemia, stunning, inflammation, and apoptosis during cardiac surgery: a review of evidence. , 2004, European Journal of Cardio-Thoracic Surgery.
[4] Kazuo Ichihara,et al. All hydrophobic HMG-CoA reductase inhibitors induce apoptotic death in rat pulmonary vein endothelial cells. , 2003, Atherosclerosis.
[5] Yu Zhang,et al. Pretreatment with statins enhances myocardial protection during coronary revascularization. , 2003, The Journal of thoracic and cardiovascular surgery.
[6] G. Patti,et al. Simvastatin attenuates leucocyte–endothelial interactions after coronary revascularisation with cardiopulmonary bypass , 2003, Heart.
[7] Á. Pascual,et al. 3-Hydroxy-3-methyl-glutaryl coenzyme A reductase inhibitors, atorvastatin and simvastatin, induce apoptosis of vascular smooth muscle cells by downregulation of Bcl-2 expression and Rho A prenylation. , 2002, Atherosclerosis.
[8] T. Yau,et al. Cardiopulmonary bypass induced inflammation: pathophysiology and treatment. An update. , 2002, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.
[9] G. Cuda,et al. Inhibition of neutrophil apoptosis after coronary bypass operation with cardiopulmonary bypass. , 2002, The Annals of thoracic surgery.
[10] S. Dunzendorfer,et al. Induction of apoptosis and inhibition of migration of inflammatory and vascular wall cells by cerivastatin. , 2001, Atherosclerosis.
[11] D. Moulding,et al. Molecular control of neutrophil apoptosis , 2001, FEBS letters.
[12] K. Fukatsu,et al. Cytokine-modulated inhibition of neutrophil apoptosis at local site augments exudative neutrophil functions and reflects inflammatory response after surgery. , 2001, Surgery.
[13] E. Kobayashi,et al. Enhanced responsiveness of circulatory neutrophils after cardiopulmonary bypass: increased aggregability and superoxide producing capacity. , 2000, Artificial organs.
[14] T. Cotter,et al. Inhibition of neutrophil apoptosis after elective surgery. , 1999, Surgery.
[15] C. Haslett,et al. NF-κB Activation Is a Critical Regulator of Human Granulocyte Apoptosis in Vitro* , 1999, The Journal of Biological Chemistry.
[16] A. Ortiz,et al. 3-Hydroxy-3-methylglutaryl coenzyme a reductase and isoprenylation inhibitors induce apoptosis of vascular smooth muscle cells in culture. , 1998, Circulation research.
[17] J. Marshall,et al. Dysregulated expression of neutrophil apoptosis in the systemic inflammatory response syndrome. , 1997, Archives of surgery.
[18] B. Davis,et al. The effect of pravastatin on coronary events after myocardial infarction in patients with average cholesterol levels. Cholesterol and Recurrent Events Trial investigators. , 1996, The New England journal of medicine.
[19] P. Kiener,et al. Differential expression of Fas (CD95) and Fas ligand on normal human phagocytes: implications for the regulation of apoptosis in neutrophils , 1996, The Journal of experimental medicine.
[20] A. R. Marchese,et al. Reduction by indobufen of neutrophil activation in peripheral arterial occlusive disease. , 1996, Journal of cardiovascular pharmacology.
[21] P. Henkart. ICE family proteases: mediators of all apoptotic cell death? , 1996, Immunity.
[22] B. Kasiske,et al. Inhibition of the mevalonate pathway: benefits beyond cholesterol reduction? , 1996, The Lancet.
[23] W. Liles,et al. Regulation of apoptosis in neutrophils--Fas track to death? , 1995, Journal of immunology.
[24] C. Haslett,et al. Impairment of function in aging neutrophils is associated with apoptosis. , 1993, Journal of immunology.
[25] L. Gerschenson,et al. Apoptosis: a different type of cell death , 1992, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.