Increased PAI activity and PAI-1 antigen occurring with an oral fat load: associations with PAI-1 genotype and plasma active TGF-beta levels.
暂无分享,去创建一个
[1] C. Byrne,et al. Transforming growth factor beta is sequestered into an inactive pool by lipoproteins. , 1997, Journal of lipid research.
[2] P. Ridker,et al. Arterial and venous thrombosis is not associated with the 4G/5G polymorphism in the promoter of the plasminogen activator inhibitor gene in a large cohort of US men. , 1997, Circulation.
[3] D. Grainger,et al. Tamoxifen: Teaching an old drug new tricks? , 1996, Nature Medicine.
[4] D. Grainger,et al. A PIVOTAL ROLE FOR TGF‐β* IN ATHEROGENESIS? , 1995 .
[5] D. Grainger,et al. Tamoxifen elevates transforming growth factor–β and suppresses diet–induced formation of lipid lesions in mouse aorta , 1995, Nature Medicine.
[6] P. Grant,et al. Environmental and Genetic Factors in Relation to Elevated Circulating Levels of Plasminogen Activator Inhibitor-1 in Caucasian Patients with Non-Insulin-Dependent Diabetes Mellitus , 1995, Thrombosis and Haemostasis.
[7] L. Wakefield,et al. Release and activation of platelet latent TGF–β in blood clots during dissolution with plasmin , 1995, Nature Medicine.
[8] K. Huber,et al. Tissue-type plasminogen activator and Type-1 plasminogen activator inhibitor in patients with coronary artery disease — Relations to clinical variables and cardiovascular risk factors , 1995 .
[9] A. Hamsten,et al. Allele-specific increase in basal transcription of the plasminogen-activator inhibitor 1 gene is associated with myocardial infarction. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[10] P. Weissberg,et al. Active and acid-activatable TGF-β in human sera, platelets and plasma , 1995 .
[11] B. Sobel,et al. Induction of plasminogen activator inhibitor type-1 (PAI-1) by proinsulin and insulin in vivo. , 1995, Circulation.
[12] J. Yudkin,et al. Determinants of Plasminogen Activator Inhibitor 1 Activity in Treated NIDDM and Its Relation to a Polymorphism in the Plasminogen Activator Inhibitor 1 Gene , 1995, Diabetes.
[13] A. Grace,et al. The serum concentration of active transforming growth factor-β is severely depressed in advanced atherosclerosis , 1995, Nature Medicine.
[14] A. Kulkarni,et al. Transforming growth factor-beta 1 null mice. An animal model for inflammatory disorders. , 1995, The American journal of pathology.
[15] R. Lawn,et al. Activation of transforming growth factor-β is inhibited in transgenic apolipoprotein(a) mice , 1994, Nature.
[16] D. D. Bono,et al. Significance of raised plasma concentrations of tissue-type plasminogen activator and plasminogen activator inhibitor in patients at risk from ischaemic heart disease. , 1994 .
[17] P. Weissberg,et al. Heparin decreases the rate of proliferation of rat vascular smooth muscle cells by releasing transforming growth factor beta-like activity from serum. , 1993, Cardiovascular research.
[18] S. Thompson,et al. Involvement of the hemostatic system in the insulin resistance syndrome. A study of 1500 patients with angina pectoris. The ECAT Angina Pectoris Study Group. , 1993, Arteriosclerosis and thrombosis : a journal of vascular biology.
[19] P. Weissberg,et al. Proliferation of human smooth muscle cells promoted by lipoprotein(a). , 1993, Science.
[20] A. Henney,et al. The two allele sequences of a common polymorphism in the promoter of the plasminogen activator inhibitor-1 (PAI-1) gene respond differently to interleukin-1 in HepG2 cells. , 1993, The Journal of biological chemistry.
[21] J. Gamble,et al. Transforming growth factor-beta inhibits E-selectin expression on human endothelial cells. , 1993, Journal of immunology.
[22] T. Urano,et al. Impaired fibrinolytic activity induced by ingestion of butter: effect of increased plasma lipids on the fibrinolytic activity. , 1993, Thrombosis research.
[23] A. Hamsten,et al. HDLs and alimentary lipemia. Studies in men with previous myocardial infarction at a young age. , 1993, Arteriosclerosis and thrombosis : a journal of vascular biology.
[24] J. K. Dunn,et al. Relation of triglyceride metabolism and coronary artery disease. Studies in the postprandial state. , 1992, Arteriosclerosis and thrombosis : a journal of vascular biology.
[25] G. Proetzel,et al. Targeted disruption of the mouse transforming growth factor-β1 gene results in multifocal inflammatory disease , 1992, Nature.
[26] A. Hamsten,et al. Lipoprotein lipase in plasma after an oral fat load: relation to free fatty acids. , 1992, Journal of lipid research.
[27] N. Crowther,et al. Sensitive Amplified Immunoenzymometric Assays (IEMA) for Human Insulin and Intact Proinsulin , 1992, European journal of clinical chemistry and clinical biochemistry : journal of the Forum of European Clinical Chemistry Societies.
[28] L. Mannucci,et al. Hypertriglyceridemia and regulation of fibrinolytic activity. , 1992, Arteriosclerosis and thrombosis : a journal of vascular biology.
[29] A. Gotto,et al. Influence of short term dietary cholesterol and fat on human plasma Lp[a] and LDL levels. , 1991, Journal of lipid research.
[30] D. Rifkin,et al. Lipoprotein (a) inhibits the generation of transforming growth factor beta: an endogenous inhibitor of smooth muscle cell migration , 1991, The Journal of cell biology.
[31] A. Hofman,et al. Postprandial lipoprotein metabolism in normolipidemic men with and without coronary artery disease. , 1991, Arteriosclerosis and thrombosis : a journal of vascular biology.
[32] A. Henney,et al. Genetic variation at the plasminogen activator inhibitor-1 locus is associated with altered levels of plasma plasminogen activator inhibitor-1 activity. , 1991, Arteriosclerosis and thrombosis : a journal of vascular biology.
[33] C. Packard,et al. Postprandial lipemia, fenofibrate and coronary artery disease. , 1990, Atherosclerosis.
[34] D. Rifkin,et al. Characterization of the activation of latent TGF-beta by co-cultures of endothelial cells and pericytes or smooth muscle cells: a self- regulating system , 1990, The Journal of cell biology.
[35] A. Hamsten,et al. The Fibrinolytic Enzyme System and Its Role in the Etiology of Thromboembolic Disease , 1990, Seminars in thrombosis and hemostasis.
[36] F. Welt,et al. Transforming growth factor-beta activity is potentiated by heparin via dissociation of the transforming growth factor-beta/alpha 2- macroglobulin inactive complex , 1989, The Journal of cell biology.
[37] D. Owens,et al. Sensitive and specific two-site immunoradiometric assays for human insulin, proinsulin, 65-66 split and 32-33 split proinsulins. , 1989, The Biochemical journal.
[38] L. Bernstein,et al. Chylomicrons and chylomicron remnants in coronary artery disease: a case-control study. , 1987, Atherosclerosis.
[39] A. Gotto,et al. Inverse relationship between blood levels of high density lipoprotein subfraction 2 and magnitude of postprandial lipemia. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[40] D. B. Zilversmit,et al. Comparison of Cholesteryl Ester Transfer from Chylomicrons and Other Plasma Lipoproteins to Aortic Intima Media of Cholesterol‐Fed Rabbits , 1982, Arteriosclerosis.
[41] D. B. Zilversmit. Atherogenesis: a postprandial phenomenon. , 1979, Circulation.
[42] R. Havel,et al. The distribution and chemical composition of ultracentrifugally separated lipoproteins in human serum. , 1955, The Journal of clinical investigation.