Tenascin C+ papillary fibroblasts facilitate neuro-immune interaction in a mouse model of psoriasis
暂无分享,去创建一个
Hong Wang | Hong Wang | Honglin Wang | Q. Yin | Zhikai Wang | Zhenyao Xu | Qun Li | Yang Sun | Hong Zhou | Xiangxiao Li | Maoying Han | Xiaojie Cai | Siyu Deng | Xichen Zheng | Libo Sun | Bin Zhou | T. Zhang | F. Lou | Taiyu Zhang | Xichen Zheng
[1] Jonathan L. Linehan,et al. Immunity to the microbiota promotes sensory neuron regeneration , 2023, Cell.
[2] Yuehua Wu,et al. Lidocaine Ameliorates Psoriasis by Obstructing Pathogenic CGRP Signaling-Mediated Sensory Neuron-Dendritic Cell Communication. , 2022, The Journal of investigative dermatology.
[3] Yucheng Hu,et al. Anatomically distinct fibroblast subsets determine skin autoimmune patterns , 2021, Nature.
[4] M. Vesely,et al. A pruritic psoriatic plaque develops at the donor site of an autologous skin graft: Koebner phenomenon , 2021, The Lancet.
[5] Yongfei Hu,et al. Single-cell RNA-seq reveals fibroblast heterogeneity and increased mesenchymal fibroblasts in human fibrotic skin diseases , 2021, Nature Communications.
[6] Deshka S. Foster,et al. Preventing Engrailed-1 activation in fibroblasts yields wound regeneration without scarring , 2021, Science.
[7] Preet G S Makker,et al. Cutaneous Neuroimmune Interactions in Peripheral Neuropathic Pain States , 2021, Frontiers in Immunology.
[8] L. Tang,et al. Koebner phenomenon induced by cupping therapy in the unstable stage of psoriasis: A case report , 2021, Dermatologic therapy.
[9] J. I. Izpisúa Belmonte,et al. A Single-Cell Transcriptomic Atlas of Human Skin Aging. , 2020, Developmental cell.
[10] J. C. Love,et al. Second-Strand Synthesis-Based Massively Parallel scRNA-Seq Reveals Cellular States and Molecular Features of Human Inflammatory Skin Pathologies , 2020, Immunity.
[11] Yanling He,et al. The Role of Nociceptive Neurons in the Pathogenesis of Psoriasis , 2020, Frontiers in Immunology.
[12] Jingwei Jiang,et al. Single-cell RNA-Seq reveals the transcriptional landscape and heterogeneity of skin macrophages in Vsir-/- murine psoriasis , 2020, Theranostics.
[13] Sang Hoon Lee,et al. Sensory neuron-expressed transient receptor potential channel 4 is a target for the relief of psoriasiform itch and skin inflammation in mice. , 2020, The Journal of investigative dermatology.
[14] Hyun Je Kim,et al. Single-cell transcriptome analysis of human skin identifies novel fibroblast subpopulation and enrichment of immune subsets in atopic dermatitis. , 2020, The Journal of allergy and clinical immunology.
[15] Bin Zhou,et al. Generation of a self‐cleaved inducible Cre recombinase for efficient temporal genetic manipulation , 2020, The EMBO journal.
[16] H. Siiskonen,et al. Mast Cells and Sensory Nerves Contribute to Neurogenic Inflammation and Pruritus in Chronic Skin Inflammation , 2019, Front. Cell. Neurosci..
[17] B. Davis,et al. Cutaneous TRPV1+ Neurons Trigger Protective Innate Type 17 Anticipatory Immunity , 2019, Cell.
[18] D. Ginty,et al. Deep Sequencing of Somatosensory Neurons Reveals Molecular Determinants of Intrinsic Physiological Properties , 2019, Neuron.
[19] I. Chiu,et al. Neuronal Regulation of Immunity in the Skin and Lungs , 2019, Trends in Neurosciences.
[20] Alireza Hadj Khodabakhshi,et al. Metascape provides a biologist-oriented resource for the analysis of systems-level datasets , 2019, Nature Communications.
[21] P. Petzelbauer,et al. Lineage Identity and Location within the Dermis Determine the Function of Papillary and Reticular Fibroblasts in Human Skin. , 2019, The Journal of investigative dermatology.
[22] Zev J. Gartner,et al. DoubletFinder: Doublet detection in single-cell RNA sequencing data using artificial nearest neighbors , 2018, bioRxiv.
[23] Paul Hoffman,et al. Integrating single-cell transcriptomic data across different conditions, technologies, and species , 2018, Nature Biotechnology.
[24] F. Watt,et al. Fibroblast heterogeneity: implications for human disease , 2018, The Journal of clinical investigation.
[25] B. Marsden,et al. Mapping tenascin-C interaction with toll-like receptor 4 reveals a new subset of endogenous inflammatory triggers , 2017, Nature Communications.
[26] G. Yosipovitch,et al. Role of neurturin in spontaneous itch and increased nonpeptidergic intraepidermal fiber density in a mouse model of psoriasis , 2017, Pain.
[27] Shaohua Zhang,et al. Preexisting endothelial cells mediate cardiac neovascularization after injury , 2017, The Journal of clinical investigation.
[28] M. Chiquet,et al. Tenascin-C at a glance , 2016, Journal of Cell Science.
[29] Swati Bhattacharyya,et al. Tenascin-C drives persistence of organ fibrosis , 2016, Nature Communications.
[30] Eric J Berns,et al. A tenascin-C mimetic peptide amphiphile nanofiber gel promotes neurite outgrowth and cell migration of neurosphere-derived cells. , 2016, Acta biomaterialia.
[31] Kathy O. Lui,et al. Endocardium Contributes to Cardiac Fat. , 2016, Circulation research.
[32] S. McMahon,et al. Crosstalk between the nociceptive and immune systems in host defence and disease , 2015, Nature Reviews Neuroscience.
[33] Aaron M. Newman,et al. Identification and isolation of a dermal lineage with intrinsic fibrogenic potential , 2015, Science.
[34] F. Watt,et al. Understanding fibroblast heterogeneity in the skin. , 2015, Trends in cell biology.
[35] Hoon-Soo Kim,et al. Clinical Characteristics of Pruritus in Patients with Scalp Psoriasis and Their Relation with Intraepidermal Nerve Fiber Density , 2014, Annals of Dermatology.
[36] U. V. von Andrian,et al. Nociceptive Sensory Neurons Drive Interleukin-23 Mediated Psoriasiform Skin Inflammation , 2014, Nature.
[37] Cole Trapnell,et al. Pseudo-temporal ordering of individual cells reveals dynamics and regulators of cell fate decisions , 2014, Nature Biotechnology.
[38] Fiona M. Watt,et al. Distinct fibroblast lineages determine dermal architecture in skin development and repair , 2013, Nature.
[39] S. Dib-Hajj,et al. Gain-of-function Nav1.8 mutations in painful neuropathy , 2012, Proceedings of the National Academy of Sciences.
[40] R. Geha,et al. Leukotriene B4-driven neutrophil recruitment to the skin is essential for allergic skin inflammation. , 2012, Immunity.
[41] Zhen-Ge Luo,et al. Laminin/β1 integrin signal triggers axon formation by promoting microtubule assembly and stabilization , 2012, Cell Research.
[42] V. Jala,et al. Pivotal role of dermal IL-17-producing γδ T cells in skin inflammation. , 2011, Immunity.
[43] Edi Brogi,et al. Breast cancer cells produce tenascin C as a metastatic niche component to colonize the lungs , 2011, Nature Medicine.
[44] D. Diaconu,et al. Cutaneous denervation of psoriasiform mouse skin improves acanthosis and inflammation in a sensory neuropeptide dependent manner , 2011, The Journal of investigative dermatology.
[45] L. Sagi,et al. The Koebner phenomenon. , 2011, Clinics in dermatology.
[46] R. Coffman,et al. Autoimmune skin inflammation is dependent on plasmacytoid dendritic cell activation by nucleic acids via TLR7 and TLR9 , 2010, The Journal of experimental medicine.
[47] O. Griesbeck,et al. In vivo imaging of partially reversible th17 cell-induced neuronal dysfunction in the course of encephalomyelitis. , 2010, Immunity.
[48] Allan R. Jones,et al. A robust and high-throughput Cre reporting and characterization system for the whole mouse brain , 2009, Nature Neuroscience.
[49] L. Boon,et al. Imiquimod-Induced Psoriasis-Like Skin Inflammation in Mice Is Mediated via the IL-23/IL-17 Axis1 , 2009, The Journal of Immunology.
[50] J. Fawcett,et al. α9 Integrin Promotes Neurite Outgrowth on Tenascin-C and Enhances Sensory Axon Regeneration , 2009, The Journal of Neuroscience.
[51] W. Born,et al. gammadelta T cells: an important source of IL-17. , 2008, Current opinion in immunology.
[52] Ellen A. Lumpkin,et al. Mechanisms of sensory transduction in the skin , 2007, Nature.
[53] J. Clifford,et al. Stat3 links activated keratinocytes and immunocytes required for development of psoriasis in a novel transgenic mouse model , 2005, Nature Medicine.
[54] S. Meiners,et al. Neurite Outgrowth by the Alternatively Spliced Region of Human Tenascin-C Is Mediated by Neuronal α7β1 Integrin , 2004, The Journal of Neuroscience.
[55] Jacques J. Peschon,et al. Semaphorin 7A promotes axon outgrowth through integrins and MAPKs , 2003, Nature.
[56] Mark Lebwohl,et al. Psoriasis , 1906, The Lancet.
[57] Alex E. Lash,et al. Gene Expression Omnibus: NCBI gene expression and hybridization array data repository , 2002, Nucleic Acids Res..
[58] S. Meiners,et al. Identification of a Neurite Outgrowth-Promoting Motif within the Alternatively Spliced Region of Human Tenascin-C , 2001, The Journal of Neuroscience.
[59] A. Faissner,et al. The structure and function of tenascins in the nervous system. , 2001, Matrix biology : journal of the International Society for Matrix Biology.
[60] P. V. D. van de Kerkhof,et al. Tenascin-C is not a useful marker for disease activity in psoriasis. , 1998, Acta dermato-venereologica.
[61] E. Farber,et al. Double‐labeled immunofluorescence study of cutaneous nerves in psoriasis , 1998, International journal of dermatology.
[62] M. Chiquet,et al. The integrin receptor α 8 β 1 mediates interactions of embryonic chick motor and sensory neurons with tenascin-C , 1995, Neuron.
[63] D. C. Henckel,et al. Case report. , 1995, Journal.
[64] J. Schalkwijk,et al. Tenascin expression in hyperproliferative skin diseases , 1991, The British journal of dermatology.
[65] R. Chiquet‐Ehrismann,et al. Tenascin: an extracellular matrix protein involved in tissue interactions during fetal development and oncogenesis , 1986, Cell.