Association between myeloperoxidase levels and risk of coronary artery disease.
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E J Topol | E. Topol | S. Hazen | Renliang Zhang | M. Brennan | M. Penn | D. Sprecher | G. Pearce | Xiaoming Fu | D L Sprecher | S L Hazen | M S Penn | R Zhang | M L Brennan | X Fu | R J Aviles | G L Pearce | Ronnier J. Aviles | X. Fu | R. Zhang | R. Aviles | M. Brennan | Stanley L Hazen
[1] G. Rouleau,et al. A functional myeloperoxidase polymorphic variant is associated with coronary artery disease in French-Canadians. , 2001, American heart journal.
[2] Jo,et al. Characterization of a human blood monocyte subset with low peroxidase activity. , 1983, The Journal of clinical investigation.
[3] Barry Halliwell,et al. Formation of nitric oxide-derived inflammatory oxidants by myeloperoxidase in neutrophils , 1998, Nature.
[4] A. Daugherty,et al. Myeloperoxidase, a catalyst for lipoprotein oxidation, is expressed in human atherosclerotic lesions. , 1994, The Journal of clinical investigation.
[5] S. L. Hazen,et al. p-Hydroxyphenylacetaldehyde, an Aldehyde Generated by Myeloperoxidase, Modifies Phospholipid Amino Groups of Low Density Lipoprotein in Human Atherosclerotic Intima* , 2000, The Journal of Biological Chemistry.
[6] R Peto,et al. Association of fibrinogen, C-reactive protein, albumin, or leukocyte count with coronary heart disease: meta-analyses of prospective studies. , 1998, JAMA.
[7] P G Anderson,et al. Extensive nitration of protein tyrosines in human atherosclerosis detected by immunohistochemistry. , 1994, Biological chemistry Hoppe-Seyler.
[8] G. Lowe,et al. Lipoprotein-associated phospholipase A2 as an independent predictor of coronary heart disease. West of Scotland Coronary Prevention Study Group. , 2000, The New England journal of medicine.
[9] S. Hazen,et al. The Oxidation of Lipoproteins by Monocytes-Macrophages , 1999, The Journal of Biological Chemistry.
[10] S. Hazen,et al. Nitric oxide modulates the catalytic activity of myeloperoxidase. , 2000, The Journal of biological chemistry.
[11] C. V. Smith,et al. Selective modification of apoB-100 in the oxidation of low density lipoproteins by myeloperoxidase in vitro. , 1999, Journal of lipid research.
[12] S. Hazen,et al. Nitric Oxide Is a Physiological Substrate for Mammalian Peroxidases* , 2000, The Journal of Biological Chemistry.
[13] H. Birkedal‐Hansen,et al. Multiple modes of activation of latent human fibroblast collagenase: evidence for the role of a Cys73 active-site zinc complex in latency and a "cysteine switch" mechanism for activation. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[14] C. Capeillère-Blandin,et al. Oxidation of guaiacol by myeloperoxidase: a two-electron-oxidized guaiacol transient species as a mediator of NADPH oxidation. , 1998, The Biochemical journal.
[15] S. Hazen,et al. Leukocytes utilize myeloperoxidase-generated nitrating intermediates as physiological catalysts for the generation of biologically active oxidized lipids and sterols in serum. , 1999, Biochemistry.
[16] J. Heinecke,et al. Tyrosyl radical generated by myeloperoxidase is a physiological catalyst for the initiation of lipid peroxidation in low density lipoprotein. , 1994, The Journal of biological chemistry.
[17] S. Hazen,et al. Myeloperoxidase-generated oxidants and atherosclerosis. , 2000, Free radical biology & medicine.
[18] S. Hazen,et al. Formation of nitric oxide-derived oxidants by myeloperoxidase in monocytes: pathways for monocyte-mediated protein nitration and lipid peroxidation In vivo. , 1999, Circulation research.
[19] S. Weiss,et al. Activation of the endogenous metalloproteinase, gelatinase, by triggered human neutrophils. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[20] N. Tolbert,et al. A modification of the Lowry procedure to simplify protein determination in membrane and lipoprotein samples. , 1978, Analytical biochemistry.
[21] Leukocytes and the risk of ischemic diseases. , 1987, JAMA.
[22] A. Rebuzzi,et al. Intracellular neutrophil myeloperoxidase is reduced in unstable angina and acute myocardial infarction, but its reduction is not related to ischemia. , 1996, Journal of the American College of Cardiology.
[23] P. Libby,et al. Macrophage myeloperoxidase regulation by granulocyte macrophage colony-stimulating factor in human atherosclerosis and implications in acute coronary syndromes. , 2001, The American journal of pathology.
[24] H. Rosen,et al. Antimicrobial activity of myeloperoxidase. , 1984, Methods in enzymology.
[25] R. Virmani,et al. A comparison of the Framingham risk index, coronary artery calcification, and culprit plaque morphology in sudden cardiac death. , 2000, Circulation.
[26] F. Shabani,et al. The oxidative inactivation of tissue inhibitor of metalloproteinase-1 (TIMP-1) by hypochlorous acid (HOCI) is suppressed by anti-rheumatic drugs. , 1998, Free radical research.
[27] G. Francis,et al. Tyrosyl radical generated by myeloperoxidase catalyzes the oxidative cross-linking of proteins. , 1993, The Journal of clinical investigation.
[28] H. Rosen,et al. [52] Antimicrobial activity of myeloperoxidase , 1984 .
[29] J. Heinecke. Mass spectrometric quantification of amino acid oxidation products in proteins: insights into pathways that promote LDL oxidation in the human artery wall , 1999, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[30] D. Steinberg,et al. The oxidative modification hypothesis of atherogenesis: an overview. , 2000, Free radical biology & medicine.
[31] S. L. Hazen,et al. Reactive Nitrogen Intermediates Promote Low Density Lipoprotein Oxidation in Human Atherosclerotic Intima* , 1997, The Journal of Biological Chemistry.
[32] R. Stocker,et al. Oxidation of low-density lipoprotein with hypochlorite causes transformation of the lipoprotein into a high-uptake form for macrophages. , 1993, The Biochemical journal.
[33] J. Crowley,et al. Increased atherosclerosis in myeloperoxidase-deficient mice. , 2001, The Journal of clinical investigation.
[34] F. Hsu,et al. Mass Spectrometric Quantification of Markers for Protein Oxidation by Tyrosyl Radical, Copper, and Hydroxyl Radical in Low Density Lipoprotein Isolated from Human Atherosclerotic Plaques* , 1997, The Journal of Biological Chemistry.
[35] S. L. Hazen,et al. Elevated levels of protein-bound p-hydroxyphenylacetaldehyde, an amino-acid-derived aldehyde generated by myeloperoxidase, are present in human fatty streaks, intermediate lesions and advanced atherosclerotic lesions. , 2000, The Biochemical journal.
[36] S. L. Hazen,et al. 3-Chlorotyrosine, a specific marker of myeloperoxidase-catalyzed oxidation, is markedly elevated in low density lipoprotein isolated from human atherosclerotic intima. , 1997, The Journal of clinical investigation.
[37] S. Hazen,et al. p-Hydroxyphenylacetaldehyde Is the Major Product of L-Tyrosine Oxidation by Activated Human Phagocytes , 1996, The Journal of Biological Chemistry.
[38] J. Schultz,et al. Studies on the chlorinating activity of myeloperoxidase. , 1976, The Journal of biological chemistry.
[39] S. Hazen,et al. Peroxidases inhibit nitric oxide (NO) dependent bronchodilation: development of a model describing NO-peroxidase interactions. , 2001, Biochemistry.
[40] R. Stockley,et al. A discrete subpopulation of human monocytes expresses a neutrophil-like proinflammatory (P) phenotype. , 1994, The American journal of physiology.
[41] D. Kutter,et al. Consequences of Total and Subtotal Myeloperoxidase Deficiency: Risk or Benefit ? , 2000, Acta Haematologica.
[42] P. Ridker,et al. Inflammation, aspirin, and the risk of cardiovascular disease in apparently healthy men. , 1997, The New England journal of medicine.
[43] S. Hazen,et al. Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species. , 2000, The Journal of clinical investigation.
[44] R. Stocker,et al. Immunologic detection and measurement of hypochlorite-modified LDL with specific monoclonal antibodies. , 1995, Arteriosclerosis, Thrombosis and Vascular Biology.
[45] A. Carr,et al. Myeloperoxidase binds to low‐density lipoprotein: potential implications for atherosclerosis , 2000, FEBS letters.
[46] S. Hazen,et al. Myeloperoxidase-generated reactive nitrogen species convert LDL into an atherogenic form in vitro. , 1999, The Journal of clinical investigation.
[47] S. Hazen,et al. Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice. , 2000, The Journal of clinical investigation.
[48] H. Malech,et al. Primary inherited defects in neutrophil function: etiology and treatment. , 1997, Seminars in hematology.