Elevated F2-isoprostanes in thalassemic patients.
暂无分享,去创建一个
B. Halliwell | S. Fucharoen | P. Wilairat | C. Matayatsuk | C. J. Lee | P. Sirankapracha | R. Kalpravidh
[1] E. Vichinsky,et al. Oxidative stress and inflammation in iron‐overloaded patients with β‐thalassaemia or sickle cell disease , 2006 .
[2] B. Halliwell,et al. Cautions in the use of biomarkers of oxidative damage; the vascular and antioxidant effects of dark soy sauce in humans. , 2006, Biochemical and biophysical research communications.
[3] R. Naithani,et al. Peroxidative stress and antioxidant enzymes in children with β‐thalassemia major , 2006 .
[4] S. Gangemi,et al. Increased protein carbonyl groups in the serum of patients affected by thalassemia major , 2006, Annals of Hematology.
[5] J. Morrow. Quantification of Isoprostanes as Indices of Oxidant Stress and the Risk of Atherosclerosis in Humans , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[6] E. Rachmilewitz,et al. MEDICAL PROGRESS : β-thalassemia , 2005 .
[7] P. Mecocci,et al. Increased F2 isoprostane plasma levels in patients with congestive heart failure are correlated with antioxidant status and disease severity. , 2004, Journal of cardiac failure.
[8] B. Chung,et al. Increased urinary F2-isoprostanes levels in the patients with Alzheimer’s disease , 2004, Brain Research Bulletin.
[9] B. Halliwell,et al. Rapid preparation of human urine and plasma samples for analysis of F2-isoprostanes by gas chromatography-mass spectrometry. , 2004, Biochemical and biophysical research communications.
[10] B. Halliwell,et al. Measuring reactive species and oxidative damage in vivo and in cell culture: how should you do it and what do the results mean? , 2004, British journal of pharmacology.
[11] I. Papassotiriou,et al. A rare example that coinheritance of a severe form of β-thalassemia and α-thalassemia interact in a “synergistic” manner to balance the phenotype of classic thalassemic syndromes , 2004 .
[12] G. FitzGerald,et al. F2-isoprostanes as indices of lipid peroxidation in inflammatory diseases. , 2004, Chemistry and physics of lipids.
[13] D. Tsikas,et al. Urinary 8-iso-Prostaglandin F2&agr; as a Risk Marker in Patients With Coronary Heart Disease: A Matched Case-Control Study , 2004 .
[14] S. Basu. ReviewIsoprostanes: Novel Bioactive Products of Lipid Peroxidation , 2004, Free radical research.
[15] T. Montine,et al. Quantification of F-ring isoprostane-like compounds (F4-neuroprostanes) derived from docosahexaenoic acid in vivo in humans by a stable isotope dilution mass spectrometric assay. , 2004, Journal of chromatography. B, Analytical technologies in the biomedical and life sciences.
[16] S. Laradi,et al. Oxidant, antioxidant status and metabolic data in patients with beta-thalassemia. , 2003, Clinica chimica acta; international journal of clinical chemistry.
[17] M. Stacey,et al. Iron and 8-isoprostane levels in acute and chronic wounds. , 2003, The Journal of investigative dermatology.
[18] J. Morrow,et al. The isoprostanes: unique products of arachidonic acid oxidation-a review. , 2003, Current medicinal chemistry.
[19] A. Dash,et al. A simple LC method with UV detection for the analysis of creatine and creatinine and its application to several creatine formulations. , 2002, Journal of pharmaceutical and biomedical analysis.
[20] M. Cappellini,et al. Oxidative status and malondialdehyde in β‐thalassaemia patients , 2002 .
[21] B. Bonaz,et al. Increased urinary F2-isoprostanes in patients with Crohn's disease. , 2002 .
[22] K.,et al. Increase in plasma esterified F2-isoprostanes following intravenous iron infusion in patients on hemodialysis. , 2001, Kidney international.
[23] F. Cuccurullo,et al. Reaction conditions affecting the relationship between thiobarbituric acid reactivity and lipid peroxides in human plasma. , 2001, Free radical biology & medicine.
[24] M. Bhattacharyya,et al. Antioxidant defense status of red blood cells of patients with β-thalassemia and Eβ-thalassemia , 2001 .
[25] B. Halliwell. Lipid peroxidation, antioxidants and cardiovascular disease: how should we move forward? , 2000, Cardiovascular research.
[26] B. Halliwell. Establishing the significance and optimal intake of dietary antioxidants: the biomarker concept. , 2009, Nutrition reviews.
[27] S. Basu,et al. Metabolism of 8‐iso‐prostaglandin F2α , 1998 .
[28] R. Hillman,et al. Hematology in clinical practice : a guide to diagnosis and management , 1995 .
[29] A. Dabbagh,et al. The effect of iron overload on rat plasma and liver oxidant status in vivo. , 1994, The Biochemical journal.
[30] A. Hoffbrand,et al. Serum non‐transferrin‐bound iron in beta‐thalassaemia major patients treated with desferrioxamine and L1 , 1992, British journal of haematology.
[31] J. Morrow,et al. A series of prostaglandin F2-like compounds are produced in vivo in humans by a non-cyclooxygenase, free radical-catalyzed mechanism. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[32] R. Mccomb,et al. Candidate reference method for determination of total bilirubin in serum: development and validation. , 1985, Clinical chemistry.
[33] R. Carrell,et al. The estimation of red cell superoxide dismutase activity. , 1975, The Journal of laboratory and clinical medicine.
[34] J. Stocks,et al. The Autoxidation of Human Red Cell Lipids Induced by Hydrogen Peroxide , 1971, British journal of haematology.
[35] E. Beutler,et al. Improved method for the determination of blood glutathione. , 1963, The Journal of laboratory and clinical medicine.