The metabolism of the anoxic heart after surgical denervation or reserpine pretreatment.

[1]  E. Krause,et al.  Metabolic control characteristics of the acutely ischemic myocardium. , 1968, The American journal of cardiology.

[2]  C. Honig,et al.  Depression of myosin B by catecholamine analogs: mechanism and in vivo significance. , 1968, The American journal of physiology.

[3]  R. Berne,et al.  Effect of sympathectomy on cardiac UDPG-glycogen transferase activity in the cat. , 1967, The American journal of physiology.

[4]  I. Øye.  The Role of Phosphorylase a and b for the Control of Glycogenolysis in the Isolated Working Rat Heart , 1967 .

[5]  N. Dhalla,et al.  Studies on the relationship between phosphorylase activation and increase in cardiac function. , 1967, The Journal of pharmacology and experimental therapeutics.

[6]  A. Stam,et al.  THE INFLUENCE OF ADRENERGIC MEDIATORS AND THEIR STRUCTURAL ANALOGS ON CARDIAC ACTOMYOSIN SYSTEMS * , 1967, Annals of the New York Academy of Sciences.

[7]  S. Mayer,et al.  ADRENERGIC MECHANISMS IN CARDIAC GLYCOGEN METABOLISM * , 1967, Annals of the New York Academy of Sciences.

[8]  B. Williams,et al.  Hormonal effects on glycogen metabolism in the rat heart in situ. , 1966, Molecular pharmacology.

[9]  J. Williamson.  Metabolic effects of epinephrine in the perfused rat heart. II. Control steps of glucose and glycogen metabolism. , 1966, Molecular pharmacology.

[10]  I. Øye.  The action of adrenaline in cardiac muscle. Dissociation between phosphorylase activation and inotropic response. , 1965 .

[11]  R. Butcher,et al.  The effect of epinephrine on adenosine 3', 5'-phosphate levels in the isolated perfused rat heart. , 1965, Molecular pharmacology.

[12]  N. Dhalla,et al.  Cardiac Phosphorylase in Different Species and after Reserpine Administration.∗ , 1965, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[13]  E. Krause,et al.  [On the activation of phosphorylase and the glycolyserates in acutely anoxic hearts in dogs]. , 1965, Biochemische Zeitschrift.

[14]  A. Wollenberger,et al.  Anoxia-induced Release of Noradrenaline from the Isolated Perfused Heart , 1965, Nature.

[15]  J. Williamson,et al.  Dissociation of the Inotropic from the Glycogenolytic Effect of Epinephrine in the Isolated Rat Heart , 1965, Nature.

[16]  G. Drummond,et al.  Effect of Epinephrine on Contractile Tension and Phosphorylase Activation in Rat and Dog Hearts.∗ , 1964, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[17]  A. Parmeggiani,et al.  REGULATION OF GLYCOGENOLYSIS IN MUSCLE. II. CONTROL OF GLYCOGEN PHOSPHORYLASE REACTION IN ISOLATED PERFUSED HEART. , 1964, The Journal of biological chemistry.

[18]  T. Cooper,et al.  METABOLISM OF THE TRANSPLANTED HEART: EFFECT OF EXCISION AND REIMPLANTATION ON MYOCARDIAL GLYCOGEN, HEXOKINASE, AND ACETYLCHOLINE ESTERASE. , 1964, Surgery.

[19]  T. Cooper,et al.  ALTERATIONS IN CHEMICAL COMPOSITION OF CANINE HEART AFTER SYMPATHETIC DENERVATION. , 1964, The American journal of physiology.

[20]  D. Young,et al.  ADJUSTMENT OF GLYCOLYSIS TO ENERGY UTILIZATION IN THE PERFUSED RAT HEART. THE EFFECT OF CHANGES IN THE IONIC COMPOSITION OF THE MEDIUM ON PHOSPHOFRUCTOKINASE ACTIVITY. , 1964, The Journal of biological chemistry.

[21]  P. J. Randle,et al.  Regulation of glycogenolysis in muscle. Effects of glucagon and anoxia on lactate production, glycogen content, and phosphorylase activity in the perfused isolated rat heart. , 1963, The Journal of biological chemistry.

[22]  S. Mayer,et al.  Dissociation of the augmentation of cardiac contractile force from the activation of myocardial phosphorylase by catecholamines. , 1961, The Journal of pharmacology and experimental therapeutics.

[23]  T. Cooper,et al.  Chronic Extrinsic Cardiac Denervation by Regional Neural Ablation: Description of the Operation, Verification of the Denervation, and Its Effects on Myocardial Catecholamines , 1961, Circulation research.

[24]  M. Feinleib,et al.  Phosphorylase activity of heart muscle under various conditions affecting force of contraction. , 1959, The Journal of pharmacology and experimental therapeutics.

[25]  D. Waud,et al.  Threshold dose and time course of norepinephrine depletion of the mammalian heart by reserpine. , 1958, The Journal of pharmacology and experimental therapeutics.

[26]  M. Paasonen,et al.  The release of norepinephrine from the mammalian heart by reserpine. , 1958, The Journal of pharmacology and experimental therapeutics.

[27]  A. Wollenberger,et al.  [Release of noradrenaline from the isolated perfused heart in acute anoxia and following administration of metabolic poisons]. , 1967, Acta biologica et medica Germanica.

[28]  G. Gercken,et al.  [Static concentration of metabolites in decompensated mammal hearts following monoiodidacetate and sodium fluoride poisoning]. , 1966, Pflugers Archiv fur die gesamte Physiologie des Menschen und der Tiere.

[29]  Bretschneider Hj.  SURVIVAL TIME AND RECUPERATIVE TIME OF THE HEART IN NORMOTHERMIA AND HYPOTHERMIA , 1964 .

[30]  C. Park,et al.  THE REGULATION OF HEXOKINASE AND PHOSPHOFRUCTOKINASE ACTIVITY IN HEART MUSCLE. EFFECTS OF ALLOXAN DIABETES, GROWTH HORMONE, CORTISOL, AND ANOXIA. , 1964, The Journal of biological chemistry.