Rituximab- and ocrelizumab-induced early- and late-onset neutropenia in a multiple sclerosis patient
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J. Correale | M. Marrodan | Julia Laviano | Florencia Fornillo | R. Delorme | J. Fernández | Sofía Oneto | Fabricio M Reino | J. Laviano
[1] W. Stevenson,et al. Severe Delayed-Onset Neutropenia Induced by Ocrelizumab , 2021, The Neurohospitalist.
[2] T. Berger,et al. Late-onset neutropenia in a multiple sclerosis patient after first dose ocrelizumab switched from rituximab. , 2020, Multiple sclerosis and related disorders.
[3] D. Biotti,et al. Late-onset neutropenia after RITUXIMAB therapy for multiple sclerosis, neuromyelitis optica spectrum disorders and MOG-antibody-associated diseases. , 2020, Multiple sclerosis and related disorders.
[4] M. Filippi,et al. Late onset absolute neutropenia associated with ocrelizumab treatment in multiple sclerosis: A case report and review of the literature , 2019, Journal of the Neurological Sciences.
[5] B. Cohen. Late-onset neutropenia following ocrelizumab therapy for multiple sclerosis , 2019, Neurology.
[6] W. Rigby,et al. Rituximab associated late-onset neutropenia—a rheumatology case series and review of the literature , 2016, Clinical Rheumatology.
[7] J. Palmblad,et al. Late-onset neutropenia following rituximab therapy: incidence, clinical features and possible mechanisms , 2011, Expert review of hematology.
[8] P. Lavori,et al. Immunoglobulin G Fc receptor FcgammaRIIIa 158 V/F polymorphism correlates with rituximab-induced neutropenia after autologous transplantation in patients with non-Hodgkin's lymphoma. , 2010, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[9] W. Wilson,et al. B-cell recovery following rituximab-based therapy is associated with perturbations in stromal derived factor-1 and granulocyte homeostasis. , 2005, Blood.
[10] A. Grigg,et al. Delayed‐onset neutropenia associated with rituximab therapy , 2003, British journal of haematology.
[11] N. Fujii,et al. The earliest stages of B cell development require a chemokine stromal cell-derived factor/pre-B cell growth-stimulating factor. , 2001, Immunity.
[12] L. Weiner,et al. Multiple sclerosis flares associated with recombinant granulocyte colony-stimulating factor , 2000, Neurology.
[13] D. Brooks,et al. CD8+, CD57+ T cells from healthy elderly subjects suppress neutrophil development in vitro: implications for the neutropenia of Felty's and large granular lymphocyte syndromes. , 2000, Arthritis and rheumatism.