XRCC1 Arg194Trp and Arg399Gln polymorphisms and arsenic methylation capacity are associated with urothelial carcinoma.
暂无分享,去创建一个
Y. Hsueh | Chao-yuan Huang | Y. Pu | Wei-Jen Chen | Ying-Chin Lin | Ya-Li Huang | C. Chiang | Horng‐Sheng Shiue
[1] Jing-yi Shi,et al. Association of XRCC1 Arg399Gln polymorphism with bladder cancer susceptibility: a meta-analysis. , 2014, Gene.
[2] Shan Li,et al. DNA Repair Gene XRCC1 Polymorphisms, Smoking, and Bladder Cancer Risk: A Meta-Analysis , 2013, PloS one.
[3] Wei Chen,et al. XRCC1 Arg194Trp and Arg280His Polymorphisms Increase Bladder Cancer Risk in Asian Population: Evidence from a Meta-Analysis , 2013, PloS one.
[4] Y. Hsueh,et al. Urinary arsenic profiles and the risks of cancer mortality: a population-based 20-year follow-up study in arseniasis-endemic areas in Taiwan. , 2013, Environmental research.
[5] E. Tokar,et al. Chronic exposure of renal stem cells to inorganic arsenic induces a cancer phenotype. , 2013, Chemical research in toxicology.
[6] Arthur P. Grollman,et al. Aristolochic acid-associated urothelial cancer in Taiwan , 2012, Proceedings of the National Academy of Sciences.
[7] Z. Hseu,et al. Partitioning of arsenic in soil-crop systems irrigated using groundwater: a case study of rice paddy soils in southwestern Taiwan. , 2012, Chemosphere.
[8] A. J. Gandolfi,et al. Interdependent genotoxic mechanisms of monomethylarsonous acid: role of ROS-induced DNA damage and poly(ADP-ribose) polymerase-1 inhibition in the malignant transformation of urothelial cells. , 2011, Toxicology and applied pharmacology.
[9] A. Hartwig,et al. Arsenicals affect base excision repair by several mechanisms. , 2011, Mutation research.
[10] Shiuh-Jen Jiang,et al. Speciation analysis of arsenic compounds in edible oil by ion chromatography-inductively coupled plasma mass spectrometry. , 2011, Journal of chromatography. A.
[11] Y. Hsueh,et al. Low ratio of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanol-glucuronides (NNAL-Gluc)/free NNAL increases urothelial carcinoma risk. , 2011, The Science of the total environment.
[12] Shiuh-Jen Jiang,et al. Microwave-assisted Extraction and Ion Chromatography Dynamic Reaction Cell Inductively Coupled Plasma Mass Spectrometry for the Speciation Analysis of Arsenic and Selenium in Cereals , 2011, Analytical sciences : the international journal of the Japan Society for Analytical Chemistry.
[13] Kamaleshwar P Singh,et al. Chronic exposure to arsenic causes increased cell survival, DNA damage, and increased expression of mitochondrial transcription factor A (mtTFA) in human prostate epithelial cells. , 2011, Chemical research in toxicology.
[14] K. Chaudhuri,et al. Arsenic-induced cell proliferation is associated with enhanced ROS generation, Erk signaling and CyclinA expression. , 2010, Toxicology letters.
[15] C. Liao,et al. Assessing the cancer risk associated with arsenic-contaminated seafood. , 2010, Journal of hazardous materials.
[16] Rory Conolly,et al. Arsenic-induced carcinogenesis--oxidative stress as a possible mode of action and future research needs for more biologically based risk assessment. , 2010, Chemical research in toxicology.
[17] B. Diwan,et al. Arsenic Exposure Transforms Human Epithelial Stem/Progenitor Cells into a Cancer Stem-like Phenotype , 2009, Environmental health perspectives.
[18] D. Wolf,et al. Dimethylarsinic Acid in Drinking Water Changed the Morphology of Urinary Bladder but Not the Expression of DNA Repair Genes of Bladder Transitional Epithelium in F344 Rats , 2009, Toxicologic pathology.
[19] Chien-Jen Chen,et al. SNPs of GSTM1, T1, P1, epoxide hydrolase and DNA repair enzyme XRCC1 and risk of urinary transitional cell carcinoma in southwestern Taiwan. , 2008, Toxicology and applied pharmacology.
[20] Karen H. Almeida,et al. A unified view of base excision repair: lesion-dependent protein complexes regulated by post-translational modification. , 2007, DNA repair.
[21] Y. Hsueh,et al. Urinary arsenic profile affects the risk of urothelial carcinoma even at low arsenic exposure. , 2007, Toxicology and applied pharmacology.
[22] P. Brennan,et al. Genetic polymorphisms in the base excision repair pathway and cancer risk: a HuGE review. , 2005, American journal of epidemiology.
[23] Mark Daly,et al. Haploview: analysis and visualization of LD and haplotype maps , 2005, Bioinform..
[24] G. Fròsina. CommentaryDNA Base Excision Repair Defects in Human Pathologies , 2004, Free radical research.
[25] M. Spitz,et al. From genotype to phenotype: correlating XRCC1 polymorphisms with mutagen sensitivity. , 2003, DNA repair.
[26] D. Melton,et al. Lack of involvement of nucleotide excision repair gene polymorphisms in colorectal cancer , 2003, British Journal of Cancer.
[27] Thomas J. Smith,et al. Arsenic methylation and bladder cancer risk in Taiwan , 2003, Cancer Causes & Control.
[28] M. Vahter. Mechanisms of arsenic biotransformation. , 2002, Toxicology.
[29] Y. Hsueh,et al. Stability of arsenic species and insoluble arsenic in human urine. , 2002, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[30] I. Jaspers,et al. The role of biomethylation in toxicity and carcinogenicity of arsenic: a research update. , 2002, Environmental health perspectives.
[31] J. Luo,et al. Effects on sister chromatid exchange frequency of polymorphisms in DNA repair gene XRCC1 in smokers. , 2002, Mutation research.
[32] Y. Hsueh,et al. Urinary arsenic speciation in subjects with or without restriction from seafood dietary intake. , 2002, Toxicology letters.
[33] H. Mohrenweiser,et al. Amino acid substitution variants of APE1 and XRCC1 genes associated with ionizing radiation sensitivity. , 2001, Carcinogenesis.
[34] C I Amos,et al. Modulation of nucleotide excision repair capacity by XPD polymorphisms in lung cancer patients. , 2001, Cancer research.
[35] Jack A. Taylor,et al. DNA repair gene XRCC1 polymorphisms, smoking, and bladder cancer risk. , 2001, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[36] S. Z. Abdel‐Rahman,et al. The 399Gln polymorphism in the DNA repair gene XRCC1 modulates the genotoxic response induced in human lymphocytes by the tobacco-specific nitrosamine NNK. , 2000, Cancer letters.
[37] C. Wilson,et al. Mammalian recombination-repair genes XRCC2 and XRCC3 promote correct chromosome segregation , 2000, Nature Cell Biology.
[38] W. Cullen,et al. Comparative toxicity of trivalent and pentavalent inorganic and methylated arsenicals in rat and human cells , 2000, Archives of Toxicology.
[39] L. Thompson,et al. XRCC3 promotes homology-directed repair of DNA damage in mammalian cells. , 1999, Genes & development.
[40] B. Pan,et al. Solution structure of the single-strand break repair protein XRCC1 N-terminal domain , 1999, Nature Structural Biology.
[41] W. de Laat,et al. Molecular mechanism of nucleotide excision repair. , 1999, Genes & development.
[42] Y. Hsueh,et al. Urinary levels of inorganic and organic arsenic metabolites among residents in an arseniasis-hyperendemic area in Taiwan. , 1998, Journal of toxicology and environmental health. Part A.
[43] J. Lamerdin,et al. XRCC2 and XRCC3, new human Rad51-family members, promote chromosome stability and protect against DNA cross-links and other damages. , 1998, Molecular cell.
[44] I. M. Jones,et al. Nonconservative amino acid substitution variants exist at polymorphic frequency in DNA repair genes in healthy humans. , 1998, Cancer research.
[45] A. Abbondandolo,et al. Involvement of XRCC1 and DNA Ligase III Gene Products in DNA Base Excision Repair* , 1997, The Journal of Biological Chemistry.
[46] Y. Hsueh,et al. Serum beta-carotene level, arsenic methylation capability, and incidence of skin cancer. , 1997, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[47] A. Zuckerman,et al. IARC Monographs on the Evaluation of Carcinogenic Risks to Humans , 1995, IARC monographs on the evaluation of carcinogenic risks to humans.
[48] A. Smith,et al. Human studies do not support the methylation threshold hypothesis for the toxicity of inorganic arsenic. , 1993, Environmental research.
[49] S Lemeshow,et al. Confidence interval estimation of interaction. , 1992, Epidemiology.
[50] L. Thompson,et al. Molecular cloning and biological characterization of a human gene, ERCC2, that corrects the nucleotide excision repair defect in CHO UV5 cells , 1988, Molecular and cellular biology.
[51] T. Kuo,et al. ARSENIC AND CANCERS , 1988, The Lancet.
[52] S. You,et al. A retrospective study on malignant neoplasms of bladder, lung and liver in blackfoot disease endemic area in Taiwan. , 1986, British Journal of Cancer.
[53] E. White,et al. Persistence of urothelial carcinoma of the bladder risk among former smokers: results from a contemporary, prospective cohort study. , 2014, Urologic oncology.
[54] D. Saleh,et al. Genetic polymorphisms in NQO1 and SOD2: interactions with smoking, schistosoma infection, and bladder cancer risk in Egypt. , 2014, Urologic oncology.
[55] J. Persson,et al. Protein kinase A (PKA) pathway is functionally linked to androgen receptor (AR) in the progression of prostate cancer. , 2014, Urologic oncology.
[56] Y. Hsueh,et al. Urinary 8-hydroxydeoxyguanosine and urothelial carcinoma risk in low arsenic exposure area. , 2008, Toxicology and applied pharmacology.
[57] D. Bleyl,et al. IARC Monographs on the Evaluation of Carcinogenic Risks to Humans. Overall Evaluations of Carcinogenicity: An Updating of IARC Monographs vol. 1 to 42. Supplement 7. 440 Seiten. International Agency for Research on Cancer, Lyon 1987. Preis: 65, – s.Fr , 1989 .
[58] J. Harman. Letter: Realistic goals for smoking and health. , 1974, Lancet.