Toll-like receptor 4 signaling has a critical role in Porphyromonas gingivalis-accelerated neointimal formation after arterial injury in mice

[1]  Jingsheng Guo,et al.  Interleukin-1 Receptor-Associated Kinase 1/4 as a Novel Target for Inhibiting Neointimal Formation After Carotid Balloon Injury. , 2015, Journal of atherosclerosis and thrombosis.

[2]  M. Matsuzaki,et al.  TLR4 is a critical regulator of angiotensin II-induced vascular remodeling: the roles of extracellular SOD and NADPH oxidase , 2015, Hypertension Research.

[3]  L. Kesavalu,et al.  Myxomavirus Anti-Inflammatory Chemokine Binding Protein Reduces the Increased Plaque Growth Induced by Chronic Porphyromonas gingivalis Oral Infection after Balloon Angioplasty Aortic Injury in Mice , 2014, PloS one.

[4]  H. Itoh,et al.  Significant roles of the (pro)renin receptor in integrity of vascular smooth muscle cells , 2014, Hypertension Research.

[5]  Y. Izumi,et al.  Porphyromonas gingivalis promotes neointimal formation after arterial injury through toll-like receptor 2 signaling , 2014, Heart and Vessels.

[6]  Y. Pi,et al.  Inhibition of reactive oxygen species generation attenuates TLR4-mediated proinflammatory and proliferative phenotype of vascular smooth muscle cells , 2013, Laboratory Investigation.

[7]  Cun-Yu Wang,et al.  Tetra- and Penta-Acylated Lipid A Structures of Porphyromonas gingivalis LPS Differentially Activate TLR4-Mediated NF-κB Signal Transduction Cascade and Immuno-Inflammatory Response in Human Gingival Fibroblasts , 2013, PloS one.

[8]  Y. Izumi,et al.  Porphyromonas gingivalis Accelerates Neointimal Formation after Arterial Injury , 2012, Journal of Vascular Research.

[9]  A. Progulske-Fox,et al.  Both the unique and repeat regions of the Porphyromonas gingivalis hemagglutin A are involved in adhesion and invasion of host cells. , 2012, Anaerobe.

[10]  Jing Xiang,et al.  PPARγ attenuates intimal hyperplasia by inhibiting TLR4-mediated inflammation in vascular smooth muscle cells. , 2011, Cardiovascular research.

[11]  S. Akira,et al.  Toll-like receptors and their crosstalk with other innate receptors in infection and immunity. , 2011, Immunity.

[12]  Zhi-yong Chen,et al.  The periodontal anaerobe Porphyromonas gingivalis induced platelet activation and increased aggregation in whole blood by rat model. , 2011, Thrombosis research.

[13]  D. Grenier,et al.  Proteases of Porphyromonas gingivalis as important virulence factors in periodontal disease and potential targets for plant-derived compounds: a review article. , 2011, Current drug targets.

[14]  X. Liu,et al.  Pathogen-Mediated Inflammatory Atherosclerosis Is Mediated in Part via Toll-Like Receptor 2-Induced Inflammatory Responses , 2010, Journal of Innate Immunity.

[15]  Y. Taniyama,et al.  Ultrasound-microbubble-mediated intercellular adhesion molecule-1 small interfering ribonucleic acid transfection attenuates neointimal formation after arterial injury in mice. , 2010, Journal of the American College of Cardiology.

[16]  増田 隆宏 Mouse macrophages primed with alendronate down-regulate monocyte chemoattractant protein-1 (MCP-1) and macrophage inflammatory protein-1alpha (MIP-1alpha) production in response to Toll-like receptor (TLR) 2 and TLR4 agonist , 2009 .

[17]  R. Nagai,et al.  Fluvastatin accelerates re-endothelialization impaired by local sirolimus treatment. , 2009, European journal of pharmacology.

[18]  AndreasSchober Chemokines in Vascular Dysfunction and Remodeling , 2008 .

[19]  A. Itai,et al.  The effects of pharmacological PAI-1 inhibition on thrombus formation and neointima formation after arterial injury , 2008, Expert opinion on therapeutic targets.

[20]  K. Chayama,et al.  Role of the Angiotensin II Type 2 Receptor in Arterial Remodeling after Wire Injury in Mice , 2008, Hypertension Research.

[21]  F. C. Gibson,et al.  Sensitization of Human Aortic Endothelial Cells to Lipopolysaccharide via Regulation of Toll-Like Receptor 4 by Bacterial Fimbria-Dependent Invasion , 2005, Infection and Immunity.

[22]  Y. Taniyama,et al.  Ultrasound-Microbubble-Mediated NF-κB Decoy Transfection Attenuates Neointimal Formation after Arterial Injury in Mice , 2005, Journal of Vascular Research.

[23]  F. C. Gibson,et al.  Innate Immune Recognition of Invasive Bacteria Accelerates Atherosclerosis in Apolipoprotein E–Deficient Mice , 2004, Circulation.

[24]  H. Matsubara,et al.  Bone Marrow Monocyte Lineage Cells Adhere on Injured Endothelium in a Monocyte Chemoattractant Protein-1–Dependent Manner and Accelerate Reendothelialization as Endothelial Progenitor Cells , 2003, Circulation research.

[25]  M. M. Cowan,et al.  Virulence factors of Porphyromonas gingivalis are modified by polyphenol oxidase and asparaginase. , 2003, Oral microbiology and immunology.

[26]  U. Kalnins,et al.  Heparin-Coated Stent Placement for the Treatment of Stenoses in Small Coronary Arteries of Symptomatic Patients , 2003, Circulation.

[27]  F. Van de Werf,et al.  Chlamydia pneumoniae induces neointima formation in coronary arteries of normal pigs. , 2003, Cardiovascular research.

[28]  S. Socransky,et al.  Subgingival microbial profiles in refractory periodontal disease. , 2002, Journal of clinical periodontology.

[29]  K. Ohura,et al.  Porphyromonas gingivalis lipopolysaccharide signaling in gingival fibroblasts-CD14 and Toll-like receptors. , 2002, Critical reviews in oral biology and medicine : an official publication of the American Association of Oral Biologists.

[30]  E. L. Batista,et al.  Porphyromonas gingivalis Infection Accelerates the Progression of Atherosclerosis in a Heterozygous Apolipoprotein E–Deficient Murine Model , 2002, Circulation.

[31]  S. Hokimoto,et al.  Plasma monocyte chemoattractant protein-1 antigen levels and the risk of restenosis after coronary stent implantation. , 2001, Japanese circulation journal.

[32]  M. Makuuchi,et al.  A mouse model of vascular injury that induces rapid onset of medial cell apoptosis followed by reproducible neointimal hyperplasia. , 2000, Journal of molecular and cellular cardiology.

[33]  M. Trevisan,et al.  Identification of periodontal pathogens in atheromatous plaques. , 2000, Journal of periodontology.

[34]  H. Pahl Activators and target genes of Rel/NF-κB transcription factors , 1999, Oncogene.

[35]  KohOno,et al.  Anti–Monocyte Chemoattractant Protein-1/Monocyte Chemotactic and Activating Factor Antibody Inhibits Neointimal Hyperplasia in Injured Rat Carotid Arteries , 1999 .

[36]  P. Vokonas,et al.  An association between periodontal disease and peripheral vascular disease. , 1998, American journal of surgery.

[37]  T. Warner,et al.  Monocyte chemotactic protein-1 expression is associated with the development of vein graft intimal hyperplasia. , 1997, Arteriosclerosis, thrombosis, and vascular biology.

[38]  I. Ishikawa,et al.  Relationship between serum antibody levels and subgingival colonization of Porphyromonas gingivalis in patients with various types of periodontitis. , 1997, Journal of periodontology.

[39]  A. Takeshita,et al.  CD14-mediated signal pathway of Porphyromonas gingivalis lipopolysaccharide in human gingival fibroblasts , 1996, Infection and immunity.

[40]  A. Itai,et al.  The effects of pharmacological PAI-1 inhibition on thrombus formation and neointima formation after arterial injury. , 2008, Expert opinion on therapeutic targets.

[41]  稲垣 裕 Ultrasound-microbubble-mediated NF-κB decoy transfection attenuates neointimal formation after arterial injury in mice , 2008 .

[42]  E. Lien,et al.  Toll-like receptors. , 2002, Critical care medicine.

[43]  H. Pahl,et al.  Activators and target genes of Rel/NF-kappaB transcription factors. , 1999, Oncogene.