The inflammatory response to cardiopulmonary bypass
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[1] R. Bartlett,et al. A heparin-coated circuit reduces complement activation and the release of leukocyte inflammatory mediators during extracorporeal circulation in a rabbit. , 2008, Artificial organs.
[2] S. Thelin,et al. Heparin-coated equipment reduces complement activation during cardiopulmonary bypass in the pig. , 2008, Artificial organs.
[3] K M Taylor,et al. The platelet in cardiopulmonary bypass. , 1998, The Annals of thoracic surgery.
[4] K. Taylor,et al. Effects of cardiopulmonary bypass on leukocyte and endothelial adhesion molecules. , 1998, The Annals of thoracic surgery.
[5] A. Siegbahn,et al. Monocyte tissue factor expression, cell activation, and thrombin formation during cardiopulmonary bypass: a clinical study. , 1997, The Journal of thoracic and cardiovascular surgery.
[6] P. Toft,et al. The role of cytokines in cardiac surgery. , 1996, International journal of cardiology.
[7] J. Bion,et al. Intestinal permeability, gastric intramucosal pH, and systemic endotoxemia in patients undergoing cardiopulmonary bypass. , 1996, JAMA.
[8] H. O. Oudemans-van Straaten,et al. Myocardial performance in elderly patients after cardiopulmonary bypass is suppressed by tumor necrosis factor. , 1995, The Journal of thoracic and cardiovascular surgery.
[9] J. Maessen,et al. Influence of Duraflo II heparin-treated extracorporeal circuits on the systemic inflammatory response in patients having coronary bypass. , 1995, The Journal of thoracic and cardiovascular surgery.
[10] E. Fosse,et al. Complement and granulocyte activation in two different types of heparinized extracorporeal circuits. , 1995, The Journal of thoracic and cardiovascular surgery.
[11] G. Hill,et al. Aprotinin and methylprednisolone equally blunt cardiopulmonary bypass-induced inflammation in humans. , 1995, The Journal of thoracic and cardiovascular surgery.
[12] T. Ino,et al. Interleukin-8 and monocyte chemotactic activating factor responses to cardiopulmonary bypass. , 1995, The Journal of thoracic and cardiovascular surgery.
[13] M. Tönz,et al. The influence of leukocyte filtration during cardiopulmonary bypass on postoperative lung function. A clinical study. , 1995, The Journal of thoracic and cardiovascular surgery.
[14] W. Greeley,et al. Hemofiltration during Cardiopulmonary Bypass in Pediatric Cardiac Surgery: Effects on Hemostasis, Cytokines, and Complement Components , 1994, Anesthesiology.
[15] M. Dehoux,et al. Circulating cytokines in patients undergoing normothermic cardiopulmonary bypass. , 1994, The Journal of thoracic and cardiovascular surgery.
[16] L. Greenfield,et al. Relationship of the proinflammatory cytokines to myocardial ischemia and dysfunction after uncomplicated coronary revascularization. , 1994, The Journal of thoracic and cardiovascular surgery.
[17] E. Fosse,et al. Reduced complement and granulocyte activation with heparin-coated cardiopulmonary bypass. , 1994, The Annals of thoracic surgery.
[18] G. Hill,et al. Glucocorticoids blunt neutrophil CD11b surface glycoprotein upregulation during cardiopulmonary bypass in humans. , 1994, Anesthesia and analgesia.
[19] G. von Bernuth,et al. Complement, leukocytes, and leukocyte elastase in full-term neonates undergoing cardiac operation. , 1994, The Journal of thoracic and cardiovascular surgery.
[20] B. Risberg,et al. Hemofiltration modifies complement activation after extracorporeal circulation in infants. , 1993, The Annals of thoracic surgery.
[21] R. Linhardt,et al. Heparin oligosaccharides bind L- and P-selectin and inhibit acute inflammation. , 1993, Blood.
[22] M. Elliott,et al. Ultrafiltration and modified ultrafiltration in pediatric open heart operations. , 1993, The Annals of thoracic surgery.
[23] G. von Bernuth,et al. Complement activation during cardiopulmonary bypass in infants and children. Relation to postoperative multiple system organ failure. , 1993, The Journal of thoracic and cardiovascular surgery.
[24] J. Weiler,et al. Cytokine and complement levels in patients undergoing cardiopulmonary bypass. , 1993, The Journal of thoracic and cardiovascular surgery.
[25] R. Wakusawa,et al. Elevation of cytokines during open heart surgery with cardiopulmonary bypass: participation of interleukin 8 and 6 in reperfusion injury , 1993, Canadian journal of anaesthesia = Journal canadien d'anesthesie.
[26] J. Levy,et al. Inflammation and cardiopulmonary bypass , 1993, Canadian journal of anaesthesia = Journal canadien d'anesthesie.
[27] K. Matsushima,et al. Prevention of lung reperfusion injury in rabbits by a monoclonal antibody against interleukin-8 , 1993, Nature.
[28] W. Baumgartner,et al. Neutrophil adhesion molecule expression during cardiopulmonary bypass with bubble and membrane oxygenators. , 1993, The Annals of thoracic surgery.
[29] L. Bossaert,et al. Interleukin-8 production in patients undergoing cardiopulmonary bypass. The influence of pretreatment with methylprednisolone. , 1993, The American review of respiratory disease.
[30] R. Colman,et al. Aprotinin inhibits the contact, neutrophil, and platelet activation systems during simulated extracorporeal perfusion. , 1993, The Journal of thoracic and cardiovascular surgery.
[31] C. Dinarello,et al. Anticytokine strategies in the treatment of the systemic inflammatory response syndrome. , 1993, JAMA.
[32] J. M. Lipton,et al. Perioperative measurements of interleukin-6 and alpha-melanocyte-stimulating hormone in cardiac transplant patients. , 1993, Journal of cardiothoracic and vascular anesthesia.
[33] S Westaby,et al. Inflammatory response to cardiopulmonary bypass. , 1993, The Annals of thoracic surgery.
[34] M. Elliott,et al. Interleukin-8 release and neutrophil degranulation after pediatric cardiopulmonary bypass. , 1993, The Journal of thoracic and cardiovascular surgery.
[35] L. Cohn,et al. Complete prevention of myocardial stunning, contracture, low-reflow, and edema after heart transplantation by blocking neutrophil adhesion molecules during reperfusion , 1992 .
[36] G. Weissmann,et al. A Mechanism for the Antiinflammatory Effects of Corticosteroids , 1993, Journal of Pediatric Gastroenterology and Nutrition.
[37] W. van Oeveren,et al. Endotoxin release and tumor necrosis factor formation during cardiopulmonary bypass. , 1992, The Annals of thoracic surgery.
[38] T. Standiford,et al. Anoxia-hyperoxia induces monocyte-derived interleukin-8. , 1992, The Journal of clinical investigation.
[39] J. M. Lipton,et al. DOES APROTININ INHIBIT CARDIOPULMONARY BYPASSINDUCED INFLAMMATION , 1992 .
[40] P. Cooper. Inhibition by dexamethasone of the reperfusion phenomena in cardiopulmonary bypass. , 1991, The Journal of thoracic and cardiovascular surgery.
[41] J. Gaer,et al. Intraabdominal complications after cardiopulmonary bypass. , 1991, The Annals of thoracic surgery.
[42] M. Oppermann,et al. Formation of C5a during cardiopulmonary bypass: inhibition by precoating with heparin. , 1991, The Annals of thoracic surgery.
[43] P. Kubes,et al. Nitric oxide: an endogenous modulator of leukocyte adhesion. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[44] V. Videm,et al. Biocompatibility of extracorporeal circulation. In vitro comparison of heparin-coated and uncoated oxygenator circuits. , 1991, The Journal of thoracic and cardiovascular surgery.
[45] F. Sebening,et al. Influence of high-dose aprotinin treatment on blood loss and coagulation patterns in patients undergoing myocardial revascularization. , 1990, Anesthesiology.
[46] D. Noonan,et al. C5a-mediated release of interleukin 6 by human monocytes. , 1990, Clinical immunology and immunopathology.
[47] L. Kulander,et al. Endotoxins in cardiopulmonary bypass. , 1990, The Journal of thoracic and cardiovascular surgery.
[48] C. Dinarello,et al. Recombinant C5a stimulates transcription rather than translation of interleukin-1 (IL-1) and tumor necrosis factor: translational signal provided by lipopolysaccharide or IL-1 itself. , 1990, Blood.
[49] R. Cotran,et al. The role of endothelial cells in inflammation. , 1990, Transplantation.
[50] Timothy A. Springer,et al. Adhesion receptors of the immune system , 1990, Nature.
[51] W. Fraser,et al. Response of serum interleukin-6 in patients undergoing elective surgery of varying severity. , 1990, Clinical science.
[52] L. Osborn,et al. Leukocyte adhesion to endothelium in inflammation , 1990, Cell.
[53] W. van Oeveren,et al. Aprotinin protects platelets against the initial effect of cardiopulmonary bypass. , 1990, The Journal of thoracic and cardiovascular surgery.
[54] M. Arnaout,et al. Structure and function of the leukocyte adhesion molecules CD11/CD18 , 1990 .
[55] P. Libby,et al. Proliferating or interleukin 1-activated human vascular smooth muscle cells secrete copious interleukin 6. , 1990, The Journal of clinical investigation.
[56] Rodger P. McEver,et al. Rapid neutrophil adhesion to activated endothelium mediated by GMP-140 , 1990, Nature.
[57] J. Cavaillon,et al. Recombinant C5a enhances interleukin 1 and tumor necrosis factor release by lipopolysaccharide‐stimulated monocytes and macrophages , 1990, European journal of immunology.
[58] A. Carpentier,et al. Induction of interleukin-1 production in patients undergoing cardiopulmonary bypass. , 1989, The Journal of thoracic and cardiovascular surgery.
[59] I. von Zabern,et al. Non-enzymic activation of the fifth component of human complement, by oxygen radicals. Some properties of the activation product, C5b-like C5. , 1989, Molecular immunology.
[60] D. Wagner,et al. PADGEM protein: A receptor that mediates the interaction of activated platelets with neutrophils and monocytes , 1989, Cell.
[61] M. Baggiolini,et al. Neutrophil-activating peptide-1/interleukin 8, a novel cytokine that activates neutrophils. , 1989, The Journal of clinical investigation.
[62] I. Ginsburg,et al. Endothelial cell killing by neutrophils. Synergistic interaction of oxygen products and proteases. , 1989, The American journal of pathology.
[63] L. W. Andersen,et al. Endotoxemia and enhanced generation of oxygen radicals by neutrophils from patients undergoing cardiopulmonary bypass. , 1989, The Journal of thoracic and cardiovascular surgery.
[64] G. Schreiner,et al. Interleukin 1 and tumor necrosis factor inhibit cardiac myocyte beta-adrenergic responsiveness. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[65] T. Kishimoto,et al. The biology of interleukin-6. , 1989, Blood.
[66] P. Sims,et al. Stimulated secretion of endothelial von Willebrand factor is accompanied by rapid redistribution to the cell surface of the intracellular granule membrane protein GMP-140. , 1989, The Journal of biological chemistry.
[67] Michael Loran Dustin,et al. Functional cloning of ICAM-2, a cell adhesion ligand for LFA-1 homologous to ICAM-1 , 1989, Nature.
[68] N. Hogg. The leukocyte integrins. , 1989, Immunology today.
[69] R. Cook,et al. Cloning of GMP-140, a granule membrane protein of platelets and endothelium: Sequence similarity to proteins involved in cell adhesion and inflammation , 1989, Cell.
[70] B. Seed,et al. Endothelial leukocyte adhesion molecule 1: an inducible receptor for neutrophils related to complement regulatory proteins and lectins. , 1989, Science.
[71] D. Cosgrove,et al. Reduction in blood loss and blood use after cardiopulmonary bypass with high dose aprotinin (Trasylol). , 1989, The Journal of thoracic and cardiovascular surgery.
[72] R. Howard,et al. Effects of cardiopulmonary bypass on pulmonary leukostasis and complement activation. , 1988, Archives of surgery.
[73] J. Pober. Warner-Lambert/Parke-Davis award lecture. Cytokine-mediated activation of vascular endothelium. Physiology and pathology. , 1988, The American journal of pathology.
[74] S. Ogoshi,et al. Complement activation in cardiopulmonary bypass, with special reference to anaphylatoxin production in membrane and bubble oxygenators. , 1988, The Annals of thoracic surgery.
[75] C. Dinarello,et al. C5a stimulates secretion of tumor necrosis factor from human mononuclear cells in vitro. Comparison with secretion of interleukin 1 beta and interleukin 1 alpha , 1988, The Journal of experimental medicine.
[76] S. Khuri,et al. The Effects of Complement Activation During Cardiopulmonary Bypass: Attenuation by Hypothermia, Heparin, and Hemodilution , 1988, Annals of surgery.
[77] W. Hörl,et al. Plasma levels of main granulocyte components during cardiopulmonary bypass. , 1988, The Journal of thoracic and cardiovascular surgery.
[78] S. Clark,et al. Multiple actions of interleukin 6 within a cytokine network. , 1988, Immunology today.
[79] B. Seed,et al. ICAM, an adhesion ligand of LFA-1, is homologous to the neural cell adhesion molecule NCAM , 1988, Nature.
[80] K. M. Taylor,et al. EFFECT OF APROTININ ON NEED FOR BLOOD TRANSFUSION AFTER REPEAT OPEN-HEART SURGERY , 1987, The Lancet.
[81] C. Hack,et al. SERUM LEVELS OF INTERLEUKIN-6 AND ACUTE PHASE RESPONSES , 1987, The Lancet.
[82] C. Dinarello,et al. C5a induction of human interleukin 1. Synergistic effect with endotoxin or interferon-gamma. , 1987, Journal of immunology.
[83] J. Cavaillon,et al. C3a(C3adesArg) induces production and release of interleukin 1 by cultured human monocytes. , 1987, Journal of immunology.
[84] S. Gaffin,et al. Endotoxemia associated with cardiopulmonary bypass. , 1987, The Journal of thoracic and cardiovascular surgery.
[85] Richard O. Hynes,et al. Integrins: A family of cell surface receptors , 1987, Cell.
[86] B. Beutler,et al. Cachectin: more than a tumor necrosis factor. , 1987, The New England journal of medicine.
[87] H. Colten,et al. Cachectin/tumor necrosis factor regulates hepatic acute-phase gene expression. , 1986, The Journal of clinical investigation.
[88] A. Marty,et al. Anaphylactic Reactions in Anesthesia and Intensive Care , 1986 .
[89] W. W. Bailey,et al. The effects of methylprednisolone on complement-mediated neutrophil activation during cardiopulmonary bypass. , 1986, Surgery.
[90] J. Gamble,et al. Stimulation of neutrophils by tumor necrosis factor. , 1986, Journal of immunology.
[91] R. Colman,et al. Purified plasma factor XIIa aggregates human neutrophils and causes degranulation. , 1986, Blood.
[92] J. Fleming,et al. Increased production of peroxidation products associated with cardiac operations. Evidence for free radical generation. , 1986, The Journal of thoracic and cardiovascular surgery.
[93] G. Deeb,et al. Complement activation during cardiopulmonary bypass. Comparison of bubble and membrane oxygenators. , 1986, The Journal of thoracic and cardiovascular surgery.
[94] E. Blackstone,et al. Effects of protamine administration after cardiopulmonary bypass on complement, blood elements, and the hemodynamic state. , 1986, The Annals of thoracic surgery.
[95] H. Schaff,et al. Evidence for complement activation by protamine-heparin interaction after cardiopulmonary bypass. , 1985, Surgery.
[96] J. Hogg,et al. The effect of body temperature on leukocyte kinetics during cardiopulmonary bypass. , 1985, The Journal of thoracic and cardiovascular surgery.
[97] A. Carpentier,et al. Deleterious effects of cardiopulmonary bypass. A prospective study of bubble versus membrane oxygenation. , 1985, The Journal of thoracic and cardiovascular surgery.
[98] C. Dang,et al. Disorganization of cultured vascular endothelial cell monolayers by fibrinogen fragment D. , 1985, Science.
[99] H. Verbrugh,et al. Cardiopulmonary bypass and host defense functions in human beings: I. Serum levels and role of immunoglobulins and complement in phagocytosis. , 1985, The Annals of thoracic surgery.
[100] J. Harlan. Leukocyte-endothelial interactions. , 1985, Blood.
[101] D. Magilligan. Indications for ultrafiltration in the cardiac surgical patient. , 1985, The Journal of thoracic and cardiovascular surgery.
[102] C. Dollery,et al. Dexamethasone inhibits the production of thromboxane B2 and leukotriene B4 by human alveolar and peritoneal macrophages in culture. , 1984, Clinical science.
[103] C. Dinarello. Interleukin-1 and the pathogenesis of the acute-phase response. , 1984, The New England journal of medicine.
[104] D. Vergani,et al. Pathways to complement activation during cardiopulmonary bypass. , 1984 .
[105] P. Henson,et al. Neutrophil-endothelial cell interactions. Modulation of neutrophil adhesiveness induced by complement fragments C5a and C5a des arg and formyl-methionyl-leucyl-phenylalanine in vitro. , 1984, The Journal of clinical investigation.
[106] S Westaby,et al. Complement and the damaging effects of cardiopulmonary bypass. , 1983, The Journal of thoracic and cardiovascular surgery.
[107] R. Colman,et al. Human plasma kallikrein releases neutrophil elastase during blood coagulation. , 1983, The Journal of clinical investigation.
[108] F. Zaimi,et al. Perioperative changes in complement associated with cardiopulmonary bypass. , 1982, British journal of anaesthesia.
[109] R. Colman,et al. Purified human plasma kallikrein aggregates human blood neutrophils. , 1982, The Journal of clinical investigation.
[110] W. Rating,et al. Effects of Dexamethason on the Early Postoperative Course after Coronary Artery Bypass Surgery , 1982, The Thoracic and cardiovascular surgeon.
[111] H. Jacob,et al. Complement-induced granulocyte aggregation in vivo. , 1981, The American journal of pathology.
[112] E. Blackstone,et al. Intracardiac surgery in infants under age 3 months: incremental risk factors for hospital mortality. , 1981, The American journal of cardiology.
[113] E. Townsend,et al. Pulmonary Dysfunction Associated with Cardiopulmonary Bypass: A Comparison of Bubble and Membrane Oxygenators , 1981, Circulation.
[114] White Jv. Complement activation during cardiopulmonary bypass. , 1981 .
[115] S. Dahlén,et al. Leukotrienes promote plasma leakage and leukocyte adhesion in postcapillary venules: in vivo effects with relevance to the acute inflammatory response. , 1981, Proceedings of the National Academy of Sciences of the United States of America.
[116] E. Blackstone,et al. Complement activation during cardiopulmonary bypass: evidence for generation of C3a and C5a anaphylatoxins. , 1981, The New England journal of medicine.
[117] M. J. Moore,et al. Complement-induced granulocyte aggregation: an unsuspected mechanism of disease. , 1980, The New England journal of medicine.
[118] L. Joyce,et al. Effects of Glucocorticosteriods in Patients Undergoing Coronary Artery Bypass Surgery , 1979 .
[119] R. Byrick,et al. Postperfusion lung syndrome. Comparison of Travenol bubble and membrane oxygenators. , 1978, The Journal of thoracic and cardiovascular surgery.
[120] T. Hugli,et al. Demonstration of specific C5a receptor on intact human polymorphonuclear leukocytes. , 1978, Proceedings of the National Academy of Sciences of the United States of America.
[121] H. Jacob,et al. Oxygen radicals mediate endothelial cell damage by complement-stimulated granulocytes. An in vitro model of immune vascular damage. , 1978, The Journal of clinical investigation.
[122] J. White,et al. Complement (C5-a)-induced granulocyte aggregation in vitro. A possible mechanism of complement-mediated leukostasis and leukopenia. , 1977, The Journal of clinical investigation.
[123] G. Machiedo,et al. Prostaglandin influence on pulmonary intravascular leukocytic aggregation during cardiopulmonary bypass. , 1977, The Journal of thoracic and cardiovascular surgery.
[124] G. Noon,et al. Clinical experience with the Teflo disposable membrane oxygenator. , 1976, The Annals of thoracic surgery.
[125] S. Shapiro,et al. ABO BLOOD TYPES IN CONGENITAL HEART-DISEASE , 1975, The Lancet.
[126] M. Debakey,et al. Membrane vs Bubble Oxygenator: Clinical Comparsion , 1975, Annals of surgery.
[127] J. Wilson. The pulmonary cellular and subcellular alterations of extracorporeal circulation. , 1974, The Surgical clinics of North America.
[128] N. Ratliff,et al. Pulmonary injury secondary to extracorporeal circulation. An ultrastructural study. , 1973, The Journal of thoracic and cardiovascular surgery.
[129] I. Gigli,et al. The complement system of man. 3. , 1972, The New England journal of medicine.
[130] Karp Rb,et al. Changes in serum complement and immunoglobulins following cardiopulmonary bypass. , 1972 .
[131] A. Janoff. Mediators of tissue damage in leukocyte lysosomes. X. Further studies on human granulocyte elastase. , 1970, Laboratory investigation; a journal of technical methods and pathology.
[132] H. Müller-Eberhard,et al. THE ROLE OF SERUM COMPLEMENT IN CHEMOTAXIS OF LEUKOCYTES IN VITRO , 1965, The Journal of experimental medicine.
[133] F. Spencer,et al. INCREASED PULMONARY VASCULAR RESISTANCE FOLLOWING PROLONGED PUMP OXYGENATION. , 1965, The Journal of thoracic and cardiovascular surgery.
[134] A. Galloway,et al. Heparin bonding of bypass circuits reduces cytokine release during cardiopulmonary bypass. , 1995, The Annals of thoracic surgery.
[135] M. Elliott,et al. Cardiopulmonary bypass in neonates, infants and young children , 1994 .
[136] P. Menasché,et al. A potential mechanism of vasodilation after warm heart surgery. The temperature-dependent release of cytokines. , 1994, The Journal of thoracic and cardiovascular surgery.
[137] W. Baumgartner,et al. Inhibition of neutrophil adhesion during cardiopulmonary bypass. , 1994, The Annals of thoracic surgery.
[138] R. Colman,et al. Upregulation of Mac-1 surface expression on neutrophils during simulated extracorporeal circulation. , 1993, The Journal of laboratory and clinical medicine.
[139] J. Levy. 4 – Complement and Contact Activation , 1992 .
[140] W. van Oeveren,et al. The role of different types of corticosteroids on the inflammatory mediators in cardiopulmonary bypass. , 1991, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.
[141] C W Smith,et al. Effect of venous shear stress on CD18-mediated neutrophil adhesion to cultured endothelium. , 1990, Blood.
[142] C. Dinarello,et al. Correlations and interactions in the production of interleukin-6 (IL-6), IL-1, and tumor necrosis factor (TNF) in human blood mononuclear cells: IL-6 suppresses IL-1 and TNF. , 1990, Blood.
[143] R. Colman,et al. Platelet and neutrophil activation in cardiopulmonary bypass. , 1990, The Annals of thoracic surgery.
[144] P. Venge,et al. Inflammatory system activation during cardiopulmonary bypass as an indicator of biocompatibility: a randomized comparison of bubble and membrane oxygenators. , 1990, Scandinavian journal of thoracic and cardiovascular surgery.
[145] P. Venge,et al. Activation of inflammatory systems during cardiopulmonary bypass. , 1988, Scandinavian journal of thoracic and cardiovascular surgery.
[146] M. Hemler,et al. Adhesive protein receptors on hematopoietic cells. , 1988, Immunology today.
[147] U. Kucich,et al. Human neutrophil degranulation during extracorporeal circulation , 1987 .
[148] L. W. Andersen,et al. Presence of circulating endotoxins during cardiac operations. , 1987, The Journal of thoracic and cardiovascular surgery.
[149] Ford-Hutchinson Aw. Leukotrienes: their formation and role as inflammatory mediators. , 1985, Federation proceedings.
[150] P. Curreri,et al. Platelet and neutrophil sequestration after fragment D-induced respiratory distress. , 1982, Circulatory shock.
[151] P. Ward,et al. Role of oxygen-derived free radicals and metabolites in leukocyte-dependent inflammatory reactions. , 1982, The American journal of pathology.
[152] Haslam Pl,et al. Complement activation during cardiopulmonary bypass , 1980 .
[153] F. Mckenzie,et al. The membrane lung in routine bloodless open heart surgery. , 1977, Advances in experimental medicine and biology.
[154] H. Gewurz,et al. Complement activation by interaction of polyanions and polycations. I. Heparin-protamine induced consumption of complement. , 1975, Journal of immunology.
[155] A. Janoff. Neutrophil proteases in inflammation. , 1972, Annual review of medicine.