Lack of Galectin-3 Prevents Cardiac Fibrosis and Effective Immune Responses in a Murine Model of Trypanosoma cruzi Infection.
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[1] M. Fresno,et al. Interactions of human galectins with Trypanosoma cruzi: binding profile correlate with genetic clustering of lineages. , 2015, Glycobiology.
[2] J. Butler,et al. The Emerging Role of Galectin-3 and ST2 in Heart Failure: Practical Considerations and Pitfalls Using Novel Biomarkers , 2013, Current Heart Failure Reports.
[3] A. Vargas-Berenguel,et al. Human galectin-3 selective and high affinity inhibitors. Present state and future perspectives. , 2013, Current medicinal chemistry.
[4] R. Gazzinelli,et al. Differential Use of TLR2 and TLR9 in the Regulation of Immune Responses during the Infection with Trypanosoma cruzi , 2013, PloS one.
[5] Bertram Pitt,et al. Genetic and Pharmacological Inhibition of Galectin-3 Prevents Cardiac Remodeling by Interfering With Myocardial Fibrogenesis , 2013, Circulation. Heart failure.
[6] H. Gabius,et al. Galectin‐3 binds Neisseria meningitidis and increases interaction with phagocytic cells , 2012, Cellular microbiology.
[7] V. Rodrigues,et al. In Situ Expression of Regulatory Cytokines by Heart Inflammatory Cells in Chagas' Disease Patients with Heart Failure , 2012, Clinical & developmental immunology.
[8] Riyao Yang,et al. Galectins in acute and chronic inflammation , 2012, Annals of the New York Academy of Sciences.
[9] D. Spray,et al. Reversion of gene expression alterations in hearts of mice with chronic chagasic cardiomyopathy after transplantation of bone marrow cells , 2011, Cell cycle.
[10] F. Tzelepis,et al. Impaired Innate Immunity in Tlr4 −/− Mice but Preserved CD8+ T Cell Responses against Trypanosoma cruzi in Tlr4-, Tlr2-, Tlr9- or Myd88-Deficient Mice , 2010, PLoS pathogens.
[11] A. Pshezhetsky,et al. Neu1 desialylation of sialyl alpha-2,3-linked beta-galactosyl residues of TOLL-like receptor 4 is essential for receptor activation and cellular signaling. , 2010, Cellular signalling.
[12] D. J. Veldhuisen,et al. Galectin‐3: a novel mediator of heart failure development and progression , 2009, European journal of heart failure.
[13] N. Henderson,et al. The regulation of inflammation by galectin‐3 , 2009, Immunological reviews.
[14] J. Scharfstein,et al. Proteolytic generation of kinins in tissues infected by Trypanosoma cruzi depends on CXC chemokine secretion by macrophages activated via Toll‐like 2 receptors , 2009, Journal of leukocyte biology.
[15] M. Nolte,et al. Inflammatory signals in dendritic cell activation and the induction of adaptive immunity , 2009, Immunological reviews.
[16] M. Fresno,et al. Inducible nitric oxide synthase and arginase expression in heart tissue during acute Trypanosoma cruzi infection in mice: arginase I is expressed in infiltrating CD68+ macrophages. , 2008, The Journal of infectious diseases.
[17] S. Akira,et al. Selective synergy in anti-inflammatory cytokine production upon cooperated signaling via TLR4 and TLR2 in murine conventional dendritic cells. , 2008, Molecular immunology.
[18] O. Carretero,et al. Novel anti-inflammatory mechanisms of N-Acetyl-Ser-Asp-Lys-Pro in hypertension-induced target organ damage. , 2008, American journal of physiology. Heart and circulatory physiology.
[19] Majid Ezzati,et al. Measuring the Burden of Neglected Tropical Diseases: The Global Burden of Disease Framework , 2007, PLoS neglected tropical diseases.
[20] S. Sauleda,et al. Development of a real-time PCR assay for Trypanosoma cruzi detection in blood samples. , 2007, Acta tropica.
[21] R. Tarleton. Immune system recognition of Trypanosoma cruzi. , 2007, Current opinion in immunology.
[22] A. Henriques-Pons,et al. Fas ligand-dependent inflammatory regulation in acute myocarditis induced by Trypanosoma cruzi infection. , 2007, The American journal of pathology.
[23] J. Correale,et al. Differential glycosylation of TH1, TH2 and TH-17 effector cells selectively regulates susceptibility to cell death , 2007, Nature Immunology.
[24] S. Akira,et al. TLR-Dependent Induction of IFN-β Mediates Host Defense against Trypanosoma cruzi1 , 2006, The Journal of Immunology.
[25] D. Hsu,et al. Galectin-3 Induces Death of Candida Species Expressing Specific β-1,2-Linked Mannans1 , 2006, The Journal of Immunology.
[26] A. Sher,et al. Cutting Edge: TLR9 and TLR2 Signaling Together Account for MyD88-Dependent Control of Parasitemia in Trypanosoma cruzi Infection1 , 2006, The Journal of Immunology.
[27] F. Kierszenbaum. Where do we stand on the autoimmunity hypothesis of Chagas disease? , 2005, Trends in parasitology.
[28] C. Petersen,et al. Toll-Like Receptor 2 Regulates Interleukin-1β-Dependent Cardiomyocyte Hypertrophy Triggered by Trypanosoma cruzi , 2005, Infection and Immunity.
[29] C. Ropert,et al. Regulatory role of Toll-like receptor 2 during infection with Trypanosoma cruzi , 2004, Journal of endotoxin research.
[30] Yigal M. Pinto,et al. Galectin-3 Marks Activated Macrophages in Failure-Prone Hypertrophied Hearts and Contributes to Cardiac Dysfunction , 2004, Circulation.
[31] M. Lima,et al. Human Galectin-3 Promotes Trypanosoma cruzi Adhesion to Human Coronary Artery Smooth Muscle Cells , 2004, Infection and Immunity.
[32] P. Ricciardi-Castagnoli,et al. Up-regulation of galectin-3 and its ligands by Trypanosoma cruzi infection with modulation of adhesion and migration of murine dendritic cells. , 2004, Glycobiology.
[33] G. Rabinovich,et al. Galectin-3 Mediates IL-4-Induced Survival and Differentiation of B Cells: Functional Cross-Talk and Implications during Trypanosoma cruzi Infection 1 , 2004, The Journal of Immunology.
[34] G. Caron,et al. The Trypanosoma cruzi Tc52-Released Protein Induces Human Dendritic Cell Maturation, Signals Via Toll-Like Receptor 2, and Confers Protection Against Lethal Infection1 , 2002, The Journal of Immunology.
[35] S. Akira,et al. Glycosylphosphatidylinositol‐anchored mucin‐like glycoproteins isolated from Trypanosoma cruzi trypomastigotes induce in vivo leukocyte recruitment dependent on MCP‐1 production by IFN‐γ‐primed‐macrophages , 2002, Journal of leukocyte biology.
[36] R. Ribeiro‐dos‐Santos,et al. The pathogenesis of Chagas' disease: when autoimmune and parasite-specific immune responses meet. , 2001, Anais da Academia Brasileira de Ciencias.
[37] R. Tarleton. Parasite persistence in the aetiology of Chagas disease. , 2001, International journal for parasitology.
[38] T. Eberlein,et al. Expression and function of galectin‐3, a β‐galactoside‐binding protein in activated T lymphocytes , 2001, Journal of Leukocyte Biology.
[39] J. Dennis,et al. Negative regulation of T-cell activation and autoimmunity by Mgat5 N-glycosylation , 2001, Nature.
[40] A. Aderem,et al. The repertoire for pattern recognition of pathogens by the innate immune system is defined by cooperation between toll-like receptors. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[41] I. C. Almeida,et al. Highly purified glycosylphosphatidylinositols from Trypanosoma cruzi are potent proinflammatory agents , 2000, The EMBO journal.
[42] F. Villalta,et al. Novel mechanism that Trypanosoma cruzi uses to adhere to the extracellular matrix mediated by human galectin‐3 , 2000, FEBS letters.
[43] R. Tarleton,et al. Parasite persistence correlates with disease severity and localization in chronic Chagas' disease. , 1999, The Journal of infectious diseases.
[44] S. Reed,et al. Regulation of Trypanosoma cruzi infection in mice by gamma interferon and interleukin 10: role of NK cells , 1996, Infection and immunity.
[45] M. Levin,et al. Humoral autoimmune response in Chagas' disease: Trypanosoma cruzi ribosomal antigens as immunizing agents. , 1993, FEMS immunology and medical microbiology.
[46] J. F. Fernandes,et al. Differentiation of Trypanosoma cruzi in culture. , 1967, The Journal of protozoology.
[47] S. Akira,et al. TLR-dependent induction of IFN-beta mediates host defense against Trypanosoma cruzi. , 2006, Journal of immunology.
[48] M. Fresno,et al. Trypanosoma cruzi-induced molecular mimicry and Chagas' disease. , 2005, Current topics in microbiology and immunology.
[49] M. Fresno,et al. Etiology of Chagas disease myocarditis: autoimmunity, parasite persistence, or both? , 2003, Trends in parasitology.
[50] T. Eberlein,et al. Expression and function of galectin-3, a beta-galactoside-binding protein in activated T lymphocytes. , 2001, Journal of leukocyte biology.