A peripheral signature of Alzheimer’s disease featuring microbiota-gut-brain axis markers

[1]  D. Holtzman,et al.  ApoE isoform– and microbiota-dependent progression of neurodegeneration in a mouse model of tauopathy , 2023, Science.

[2]  M. Willmann,et al.  Signature of Alzheimer’s Disease in Intestinal Microbiome: Results From the AlzBiom Study , 2022, Frontiers in Neuroscience.

[3]  Miguel Ángel Cuevas-Budhart,et al.  Relationship Between the Gut Microbiota and Alzheimer's Disease: A Systematic Review. , 2022, Journal of Alzheimer's disease : JAD.

[4]  L. Wold,et al.  Influence of the Microbiota-Gut-Brain Axis on Cognition in Alzheimer's Disease. , 2022, Journal of Alzheimer's disease : JAD.

[5]  Ying Han,et al.  Combination of gut microbiota and plasma amyloid-β as a potential index for identifying preclinical Alzheimer’s disease: a cross-sectional analysis from the SILCODE study , 2022, Alzheimer's research & therapy.

[6]  B. Tang,et al.  Crosstalk Between the Gut Microbiota and Epithelial Cells Under Physiological and Infectious Conditions , 2022, Frontiers in Cellular and Infection Microbiology.

[7]  K. Ye,et al.  Gut microbiota regulate Alzheimer’s disease pathologies and cognitive disorders via PUFA-associated neuroinflammation , 2022, Gut.

[8]  Heleen M A Hendriksen,et al.  Gut Microbiota Composition Is Related to AD Pathology , 2022, Frontiers in Immunology.

[9]  A. Macpherson,et al.  Microbiota-derived acetate enables the metabolic fitness of the brain innate immune system during health and disease. , 2021, Cell metabolism.

[10]  M. Dichgans,et al.  Microbiota-derived short chain fatty acids modulate microglia and promote Aβ plaque deposition , 2021, eLife.

[11]  L. Morbidelli,et al.  Endothelium as a Source and Target of H2S to Improve Its Trophism and Function , 2021, Antioxidants.

[12]  M. Simons,et al.  The quiescent endothelium: signalling pathways regulating organ-specific endothelial normalcy , 2021, Nature Reviews Cardiology.

[13]  Jun Peng,et al.  Gut Microbiome Features of Chinese Patients Newly Diagnosed with Alzheimer's Disease or Mild Cognitive Impairment. , 2021, Journal of Alzheimer's Disease.

[14]  G. Frisoni,et al.  Short-Chain Fatty Acids and Lipopolysaccharide as Mediators Between Gut Dysbiosis and Amyloid Pathology in Alzheimer's Disease. , 2020, Journal of Alzheimer's disease : JAD.

[15]  G. Frisoni,et al.  Comparison of Bioinformatics Pipelines and Operating Systems for the Analyses of 16S rRNA Gene Amplicon Sequences in Human Fecal Samples , 2020, Frontiers in Microbiology.

[16]  R. Frozza,et al.  The Role of Short-Chain Fatty Acids From Gut Microbiota in Gut-Brain Communication , 2020, Frontiers in Endocrinology.

[17]  Sandy L. Torres,et al.  Neuronal Mitochondria Modulation of LPS-Induced Neuroinflammation , 2020, The Journal of Neuroscience.

[18]  S. Niida,et al.  The relationship between the gut microbiome and mild cognitive impairment in patients without dementia: a cross-sectional study conducted in Japan , 2019, Scientific Reports.

[19]  Huidong Tang,et al.  Mild cognitive impairment has similar alterations as Alzheimer's disease in gut microbiota , 2019, Alzheimer's & Dementia.

[20]  Lanjuan Li,et al.  Altered microbiomes distinguish Alzheimer’s disease from amnestic mild cognitive impairment and health in a Chinese cohort , 2019, Brain, Behavior, and Immunity.

[21]  Vanni Bucci,et al.  Alzheimer’s Disease Microbiome Is Associated with Dysregulation of the Anti-Inflammatory P-Glycoprotein Pathway , 2019, mBio.

[22]  Francesco Asnicar,et al.  QIIME 2: Reproducible, interactive, scalable, and extensible microbiome data science , 2018 .

[23]  J. Hoffman,et al.  Lipopolysaccharide endotoxemia induces amyloid-β and p-tau formation in the rat brain. , 2018, American journal of nuclear medicine and molecular imaging.

[24]  F. Jessen,et al.  Characterization and clinical use of inflammatory cerebrospinal fluid protein markers in Alzheimer’s disease , 2018, Alzheimer's Research & Therapy.

[25]  F. Sharp,et al.  Lipopolysaccharide Associates with Amyloid Plaques, Neurons and Oligodendrocytes in Alzheimer’s Disease Brain: A Review , 2018, Front. Aging Neurosci..

[26]  G. Pasinetti,et al.  Protective roles of intestinal microbiota derived short chain fatty acids in Alzheimer’s disease-type beta-amyloid neuropathological mechanisms , 2018, Expert review of neurotherapeutics.

[27]  Sterling C. Johnson,et al.  Gut microbiome alterations in Alzheimer’s disease , 2017, Scientific Reports.

[28]  C. Pepine,et al.  Increased human intestinal barrier permeability plasma biomarkers zonulin and FABP2 correlated with plasma LPS and altered gut microbiome in anxiety or depression , 2017, Gut.

[29]  G. Rossi,et al.  Microbiota modulation counteracts Alzheimer’s disease progression influencing neuronal proteolysis and gut hormones plasma levels , 2017, Scientific Reports.

[30]  M. Prinz,et al.  Microbiology: Gut microbes augment neurodegeneration , 2017, Nature.

[31]  Henrik Zetterberg,et al.  Association of Plasma Neurofilament Light With Neurodegeneration in Patients With Alzheimer Disease , 2017, JAMA neurology.

[32]  G. Frisoni,et al.  Reduction of Abeta amyloid pathology in APPPS1 transgenic mice in the absence of gut microbiota , 2017, Scientific Reports.

[33]  G. Frisoni,et al.  Association of brain amyloidosis with pro-inflammatory gut bacterial taxa and peripheral inflammation markers in cognitively impaired elderly , 2017, Neurobiology of Aging.

[34]  P. Pasqualetti,et al.  Assessment of the Incremental Diagnostic Value of Florbetapir F 18 Imaging in Patients With Cognitive Impairment: The Incremental Diagnostic Value of Amyloid PET With [18F]-Florbetapir (INDIA-FBP) Study. , 2016, JAMA neurology.

[35]  M. Khazaei,et al.  Lipopolysaccharide-Induced Spatial Memory and Synaptic Plasticity Impairment Is Preventable by Captopril , 2016, Advances in medicine.

[36]  E. Masliah,et al.  Exposure to the Functional Bacterial Amyloid Protein Curli Enhances Alpha-Synuclein Aggregation in Aged Fischer 344 Rats and Caenorhabditis elegans , 2016, Scientific Reports.

[37]  T. Krahnke,et al.  Alzheimer’s Disease Assessment Scale–Cognitive subscale variants in mild cognitive impairment and mild Alzheimer’s disease: change over time and the effect of enrichment strategies , 2016, Alzheimer's Research & Therapy.

[38]  Seongho Kim.  ppcor: An R Package for a Fast Calculation to Semi-partial Correlation Coefficients. , 2015, Communications for statistical applications and methods.

[39]  J. Choo,et al.  Sample storage conditions significantly influence faecal microbiome profiles , 2015, Scientific Reports.

[40]  E. Dejana,et al.  A gut-vascular barrier controls the systemic dissemination of bacteria , 2015, Science.

[41]  M. Buttmann,et al.  Soluble VCAM-1 impairs human brain endothelial barrier integrity via integrin α-4-transduced outside-in signalling , 2015, Acta Neuropathologica.

[42]  H. Drummond,et al.  The Impact of Different DNA Extraction Kits and Laboratories upon the Assessment of Human Gut Microbiota Composition by 16S rRNA Gene Sequencing , 2014, PloS one.

[43]  L. Gioglio,et al.  Can a bacterial endotoxin be a key factor in the kinetics of amyloid fibril formation? , 2014, Journal of Alzheimer's disease : JAD.

[44]  K. Fassbender,et al.  Molecular links between endothelial dysfunction and neurodegeneration in Alzheimer's disease. , 2014, Current Alzheimer research.

[45]  C. Jack,et al.  Biomarker Modeling of Alzheimer’s Disease , 2013, Neuron.

[46]  M. Milovanovic,et al.  P-selectin paradox and dementia of the Alzheimer type: Circulating P-selectin is increased but platelet-bound P-selectin after agonist provocation is compromised , 2013, Scandinavian journal of clinical and laboratory investigation.

[47]  K. Honda,et al.  Intestinal commensal microbes as immune modulators. , 2012, Cell host & microbe.

[48]  R. Coleman,et al.  Cerebral PET with florbetapir compared with neuropathology at autopsy for detection of neuritic amyloid-β plaques: a prospective cohort study , 2012, The Lancet Neurology.

[49]  C. Huttenhower,et al.  Metagenomic biomarker discovery and explanation , 2011, Genome Biology.

[50]  Thomas E Lane,et al.  Inflammation induced by infection potentiates tau pathological features in transgenic mice. , 2011, The American journal of pathology.

[51]  M. McGrath,et al.  Circulating endotoxin and systemic immune activation in sporadic amyotrophic lateral sclerosis (sALS) , 2009, Journal of Neuroimmunology.

[52]  A. Passaro,et al.  Markers of endothelial dysfunction in older subjects with late onset Alzheimer's disease or vascular dementia , 2008, Journal of the Neurological Sciences.

[53]  Jae Woong Lee,et al.  Journal of Neuroinflammation Neuro-inflammation Induced by Lipopolysaccharide Causes Cognitive Impairment through Enhancement of Beta-amyloid Generation , 2022 .

[54]  E. Londos,et al.  Soluble adhesion molecules and angiotensin-converting enzyme in dementia , 2007, Neurobiology of Disease.

[55]  M. Michalopoulou,et al.  Serum Levels of Soluble Intercellular Adhesion Molecule-1 and Soluble Endothelial Leukocyte Adhesion Molecule-1 in Alzheimer’s Disease , 2004, Journal of geriatric psychiatry and neurology.

[56]  L. Puricelli,et al.  Neural cell adhesion molecule in human serum. Increased levels in dementia of the Alzheimer type , 2004, Neurobiology of Disease.

[57]  Hilde van der Togt,et al.  Publisher's Note , 2003, J. Netw. Comput. Appl..

[58]  A. Mohsenin,et al.  PECAM‐1 shedding during apoptosis generates a membrane‐anchored truncated molecule with unique signaling characteristics , 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[59]  G. Wenk,et al.  LPS‐induced neuroinflammatory effects do not recover with time , 2000, Neuroreport.

[60]  F. Hayden,et al.  Efficacy of tremacamra, a soluble intercellular adhesion molecule 1, for experimental rhinovirus infection: a randomized clinical trial. , 1999, JAMA.

[61]  M. Schachner,et al.  The Neural Cell Adhesion Molecule Is a Receptor for Rabies Virus , 1998, Journal of Virology.

[62]  J. Kuiper,et al.  The influence of cytokines on the integrity of the blood-brain barrier in vitro , 1996, Journal of Neuroimmunology.

[63]  G. Forloni,et al.  Expression of amyloid precursor protein mRNAs in endothelial, neuronal and glial cells: modulation by interleukin-1. , 1992, Brain research. Molecular brain research.