Localization of a small genomic region associated with elevated ACE.

[1]  K. Saku,et al.  The D allele of the angiotensin-converting enzyme gene and reperfusion-induced ventricular arrhythmias in patients with acute myocardial infarction. , 2001, Japanese circulation journal.

[2]  John A. Todd,et al.  The genetically isolated populations of Finland and Sardinia may not be a panacea for linkage disequilibrium mapping of common disease genes , 2000, Nature Genetics.

[3]  Pui-Yan Kwok,et al.  Juxtaposed regions of extensive and minimal linkage disequilibrium in human Xq25 and Xq28 , 2000, Nature Genetics.

[4]  Sarah Parish,et al.  Large-scale test of hypothesised associations between the angiotensin-converting-enzyme insertion/deletion polymorphism and myocardial infarction in about 5000 cases and 6000 controls , 1999, The Lancet.

[5]  G. Lathrop,et al.  Fine-mapping of an ancestral recombination breakpoint in DCP1 , 1999, Nature Genetics.

[6]  K. Kontula,et al.  Angiotensin converting enzyme gene insertion/deletion polymorphism, angiotensinogen gene polymorphisms, family history of hypertension, and childhood blood pressure. , 1999, American journal of hypertension.

[7]  R. Gershoni-baruch,et al.  The D allele of the angiotensin-converting enzyme gene contributes towards blood LDL-cholesterol levels and the presence of hypertension. , 1999, Atherosclerosis.

[8]  M. Rieder,et al.  Sequence variation in the human angiotensin converting enzyme , 1999, Nature Genetics.

[9]  K. Crandall The evolution of HIV , 1999 .

[10]  M Farrall,et al.  Measured haplotype analysis of the angiotensin-I converting enzyme gene. , 1998, Human molecular genetics.

[11]  D. Ganten,et al.  Alternative splicing of the mRNA coding for the human endothelial angiotensin-converting enzyme: a new mechanism for solubilization. , 1998, Biochemical and biophysical research communications.

[12]  K K Kidd,et al.  A global haplotype analysis of the myotonic dystrophy locus: implications for the evolution of modern humans and for the origin of myotonic dystrophy mutations. , 1998, American journal of human genetics.

[13]  H. Schunkert Polymorphism of the angiotensin-converting enzyme gene and cardiovascular disease , 1997, Journal of Molecular Medicine.

[14]  J. Ott Genetic data analysis II , 1997 .

[15]  C. Brayne,et al.  Analysis of the apo E/apo C-I, angiotensin converting enzyme and methylenetetrahydrofolate reductase genes as candidates affecting human longevity. , 1997, Atherosclerosis.

[16]  Daniel L. McGee,et al.  The prevalence of hypertension in seven populations of west African origin. , 1997, American journal of public health.

[17]  M. Kobusiak-Prokopowicz [Angiotensin converting enzyme inhibitors for hypertension]. , 1997, Polski merkuriusz lekarski : organ Polskiego Towarzystwa Lekarskiego.

[18]  W. G. Hill,et al.  Genetic Data Analysis II . By Bruce S. Weir, Sunderland, Massachusetts. Sinauer Associates, Inc.445 pages. ISBN 0-87893-902-4. , 1996 .

[19]  D. Nickerson,et al.  Single-well genotyping of diallelic sequence variations by a two-color ELISA-based oligonucleotide ligation assay. , 1996, Nucleic acids research.

[20]  L. Tarnow,et al.  Development of enzyme-linked immunoassays for human angiotensin I converting enzyme suitable for large-scale studies , 1996, Journal of hypertension.

[21]  L. Tiret,et al.  Identification of new polymorphisms of the angiotensin I-converting enzyme (ACE) gene, and study of their relationship to plasma ACE levels by two-QTL segregation-linkage analysis. , 1996, American journal of human genetics.

[22]  S. Tishkoff,et al.  Global Patterns of Linkage Disequilibrium at the CD4 Locus and Modern Human Origins , 1996, Science.

[23]  G. Lathrop,et al.  Segregation and linkage analysis of serum angiotensin I-converting enzyme levels: evidence for two quantitative-trait loci. , 1995, American journal of human genetics.

[24]  A J Pakstis,et al.  Evolution of haplotypes at the DRD2 locus. , 1995, American journal of human genetics.

[25]  N. Risch,et al.  A comparison of linkage disequilibrium measures for fine-scale mapping. , 1995, Genomics.

[26]  L. Excoffier,et al.  Maximum-likelihood estimation of molecular haplotype frequencies in a diploid population. , 1995, Molecular biology and evolution.

[27]  N. Risch,et al.  Genetic analysis of idiopathic torsion dystonia in Ashkenazi Jews and their recent descent from a small founder population , 1995, Nature Genetics.

[28]  B. Waeber,et al.  Angiotensin-converting enzyme inhibitors in hypertension , 1995 .

[29]  Angiotensin I converting enzyme gene: regulation, polymorphism and implications in cardiovascular diseases. , 1994, European heart journal.

[30]  Eric S. Lander,et al.  The diastrophic dysplasia gene encodes a novel sulfate transporter: Positional cloning by fine-structure linkage disequilibrium mapping , 1994, Cell.

[31]  F. Cambien,et al.  Polymorphisms of the angiotensin-converting-enzyme gene in subjects who die from coronary heart disease. , 1994, The Quarterly journal of medicine.

[32]  C. Sing,et al.  A cladistic analysis of phenotypic associations with haplotypes inferred from restriction endonuclease mapping and DNA sequence data. III. Cladogram estimation. , 1992, Genetics.

[33]  L. Tiret,et al.  Evidence, from combined segregation and linkage analysis, that a variant of the angiotensin I-converting enzyme (ACE) gene controls plasma ACE levels. , 1992, American journal of human genetics.

[34]  H. Schunkert,et al.  Increased rat cardiac angiotensin converting enzyme activity and mRNA expression in pressure overload left ventricular hypertrophy. Effects on coronary resistance, contractility, and relaxation. , 1990, The Journal of clinical investigation.

[35]  P Corvol,et al.  An insertion/deletion polymorphism in the angiotensin I-converting enzyme gene accounting for half the variance of serum enzyme levels. , 1990, The Journal of clinical investigation.

[36]  A. Clark,et al.  Inference of haplotypes from PCR-amplified samples of diploid populations. , 1990, Molecular biology and evolution.

[37]  G. Mugesh,et al.  Converting-enzyme inhibitors in the treatment of hypertension. , 2011, The New England journal of medicine.

[38]  L. Chambless,et al.  The cardiovascular risk factor profile in the study area Augsburg. Results from the first MONICA survey 1984/85. , 2009, Acta medica Scandinavica. Supplementum.

[39]  C. Sing,et al.  A cladistic analysis of phenotype associations with haplotypes inferred from restriction endonuclease mapping. II. The analysis of natural populations. , 1988, Genetics.

[40]  E. Boerwinkle,et al.  A cladistic analysis of phenotypic associations with haplotypes inferred from restriction endonuclease mapping. I. Basic theory and an analysis of alcohol dehydrogenase activity in Drosophila. , 1987, Genetics.

[41]  A. Templeton The general relationship between average effect and average excess. , 1987, Genetical research.

[42]  G. Thomson,et al.  Measuring the strength of associations between HLA antigens and diseases. , 1981, Tissue antigens.

[43]  J. Neter,et al.  Applied linear statistical models : regression, analysis of variance, and experimental designs , 1974 .