Monomeric serum calcitonin and bone turnover during anticonvulsant treatment and in congenital hypothyroidism.

[1]  U. Lerner,et al.  Influence of diphenylhydantoin on lysosomal enzyme release during bone resorption in vitro. , 2009, Acta pharmacologica et toxicologica.

[2]  O. Tørring,et al.  Gel chromatography of immunoextracted plasma calcitonin in response to the calcium clamp in healthy males. , 1985, Acta endocrinologica.

[3]  C. Reiners,et al.  [Separate measurements of compact and spongy bone density using a transverse rotation scanner. Determination of mineral content of the radius using a least squares algorithm]. , 1985, RoFo : Fortschritte auf dem Gebiete der Rontgenstrahlen und der Nuklearmedizin.

[4]  J. Body,et al.  Estimates of circulating monomeric calcitonin: physiological studies in normal and thyroidectomized man. , 1983, The Journal of clinical endocrinology and metabolism.

[5]  M. Dambacher,et al.  Identification and characterization of calcitonin forms in plasma and urine of normal subjects and medullary carcinoma patients. , 1983, The Journal of clinical endocrinology and metabolism.

[6]  M. Mcdermott,et al.  Reduced bone mineral content in totally thyroidectomized patients: possible effect of calcitonin deficiency. , 1983, The Journal of clinical endocrinology and metabolism.

[7]  H. Wolfe Calcitonin: perspectives in current concepts , 1982, Journal of endocrinological investigation.

[8]  K. Kruse,et al.  Urinary adenosine 3',5'-monophosphate excretion in childhood. , 1981, Journal of Clinical Endocrinology and Metabolism.

[9]  U. Schubart,et al.  Effect of trifluoperazine, D600, and phenytoin on depolarization- and thyrotropin-releasing hormone-induced thyrotropin release from rat pituitary tissue. , 1981, Endocrinology.

[10]  M. Aladjem,et al.  ENHANCED RENAL TUBULAR CALCIUM REABSORPTION INDEPENDENT OF PARATHORMONE ACTIVITY, IN CHILDREN ON LONG TERM ANTICONVULSANT THERAPY , 1980, Acta paediatrica Scandinavica.

[11]  K. Jones,et al.  Calcitonin secretion in congenital nongoitrous cretinism. , 1980, The Journal of clinical investigation.

[12]  T. Hahn 6 Drug-induced disorders of vitamin D and mineral metabolism , 1980 .

[13]  J. Wark,et al.  CHRONIC DIPHENYLHYDANTOIN THERAPY DOES NOT REDUCE PLASMA 25‐HYDROXY‐VITAMIN D , 1979, Clinical endocrinology.

[14]  L. Deftos,et al.  Calcitonin secretion in normal human subjects. , 1978, The Journal of clinical endocrinology and metabolism.

[15]  L. Mosekilde,et al.  The interrelationships between serum 25-hydroxycholecalciferol, serum parathyroid hormone and bone changes in anticonvulsant osteomalacia. , 1977, Acta endocrinologica.

[16]  O. Bijvoet,et al.  NOMOGRAM FOR DERIVATION OF RENAL THRESHOLD PHOSPHATE CONCENTRATION , 1975, The Lancet.

[17]  Rimoin Dl Familial metaphyseal dysplasia. , 1970, Lancet.

[18]  R. Kruse [Osteopathies in antiepileptic long-term therapy (preliminary report)]. , 1968, Monatsschrift fur Kinderheilkunde.

[19]  W. Rick,et al.  Optimal conditions for the determination of serum alkaline phosphatase by a new kinetic method , 1967 .

[20]  J. Coindre,et al.  Bone loss in hypothyroidism with hormone replacement. A histomorphometric study. , 1986, Archives of internal medicine.

[21]  R. Ziegler,et al.  Calcitonin in human pathophysiology. , 1984, Hormone research.

[22]  R. Delorenzo Calmodulin systems in neuronal excitability: a molecular approach to epilepsy. , 1984, Annals of neurology.

[23]  K. Kruse,et al.  Die Hydroxyprolin-Ausscheidung im Morgen-Urin: ein geeigneter Parameter des Knochen-Umsatzes im Kindesalter , 1983 .

[24]  S. A. Grubb,et al.  Evidence for an important physiological role for calcitonin. , 1980, Proceedings of the National Academy of Sciences of the United States of America.

[25]  S. Glick,et al.  Diphenylhydantoin inhibition of calcitonin secretion in the pig. , 1973, Endocrinology.