Estrogen sulfotransferase ablation sensitizes mice to sepsis

[1]  B. Wei,et al.  Activation of vascular endothelial growth factor receptor-3 in macrophages restrains TLR4-NF-κB signaling and protects against endotoxin shock. , 2014, Immunity.

[2]  J. Gong,et al.  An efficient method to isolate and culture mouse Kupffer cells. , 2014, Immunology letters.

[3]  M. Jiang,et al.  Hepatic Overexpression of Steroid Sulfatase Ameliorates Mouse Models of Obesity and Type 2 Diabetes through Sex-specific Mechanisms* , 2014, The Journal of Biological Chemistry.

[4]  D. Stolz,et al.  Cellular‐specific role of toll‐like receptor 4 in hepatic ischemia‐reperfusion injury in mice , 2013, Hepatology.

[5]  Simon C Watkins,et al.  Lipopolysaccharide Clearance, Bacterial Clearance, and Systemic Inflammatory Responses Are Regulated by Cell Type–Specific Functions of TLR4 during Sepsis , 2013, The Journal of Immunology.

[6]  N. Gaikwad Ultra performance liquid chromatography-tandem mass spectrometry method for profiling of steroid metabolome in human tissue. , 2013, Analytical chemistry.

[7]  Xiuyu Wang,et al.  Oxymatrine protects against myocardial injury via inhibition of JAK2/STAT3 signaling in rat septic shock. , 2013, Molecular medicine reports.

[8]  P. Kubes,et al.  Sterile inflammation in the liver. , 2012, Gastroenterology.

[9]  S. Baldus,et al.  Depletion of neutrophil extracellular traps in vivo results in hypersusceptibility to polymicrobial sepsis in mice , 2012, Critical Care.

[10]  B. Schröppel,et al.  Toll-like receptors in transplantation: sensing and reacting to injury. , 2012, Kidney international.

[11]  J. Rubin,et al.  Estrogen sulfotransferase inhibits adipocyte differentiation. , 2011, Molecular endocrinology.

[12]  T. Sueyoshi,et al.  Garlic Extract Diallyl Sulfide (DAS) Activates Nuclear Receptor CAR to Induce the Sult1e1 Gene in Mouse Liver , 2011, PloS one.

[13]  C. Wood,et al.  Triclosan is a potent inhibitor of estradiol and estrone sulfonation in sheep placenta. , 2010, Environment international.

[14]  S. Wakil,et al.  Activation of nuclear receptor CAR ameliorates diabetes and fatty liver disease , 2009, Proceedings of the National Academy of Sciences.

[15]  I. Marriott,et al.  Estrogens augment cell surface TLR4 expression on murine macrophages and regulate sepsis susceptibility in vivo. , 2009, Endocrinology.

[16]  Yonggong Zhai,et al.  The Constitutive Androstane Receptor Is an Anti-obesity Nuclear Receptor That Improves Insulin Sensitivity* , 2009, The Journal of Biological Chemistry.

[17]  Kent Doi,et al.  Pre-existing renal disease promotes sepsis-induced acute kidney injury and worsens outcome. , 2008, Kidney international.

[18]  D. DeFranco,et al.  Glucocorticoids antagonize estrogens by glucocorticoid receptor-mediated activation of estrogen sulfotransferase. , 2008, Cancer research.

[19]  Lesly A. Dossett,et al.  High levels of endogenous estrogens are associated with death in the critically injured adult. , 2008, The Journal of trauma.

[20]  Jie Zhou,et al.  Estrogen deprivation and inhibition of breast cancer growth in vivo through activation of the orphan nuclear receptor liver X receptor. , 2007, Molecular endocrinology.

[21]  R. Evans,et al.  A Novel Pregnane X Receptor-mediated and Sterol Regulatory Element-binding Protein-independent Lipogenic Pathway* , 2006, Journal of Biological Chemistry.

[22]  J. Lawson,et al.  Spontaneous fetal loss caused by placental thrombosis in estrogen sulfotransferase—deficient mice , 2005, Nature Medicine.

[23]  C. Falany,et al.  TRICLOSAN AS A SUBSTRATE AND INHIBITOR OF 3′-PHOSPHOADENOSINE 5′-PHOSPHOSULFATE-SULFOTRANSFERASE AND UDP-GLUCURONOSYL TRANSFERASE IN HUMAN LIVER FRACTIONS , 2004, Drug Metabolism and Disposition.

[24]  A. De Maio,et al.  Genetic background conditions the effect of sex steroids on the inflammatory response during endotoxic shock , 2003, Critical care medicine.

[25]  W. Song Biochemistry and Reproductive Endocrinology of Estrogen Sulfotransferase , 2001, Annals of the New York Academy of Sciences.

[26]  T. Lawrence,et al.  Possible new role for NF-κB in the resolution of inflammation , 2001, Nature Medicine.

[27]  P. Godowski,et al.  Cell activation and apoptosis by bacterial lipoproteins through toll-like receptor-2. , 1999, Science.

[28]  I. Rusyn,et al.  Glycine and uridine prevent d‐galactosamine hepatotoxicity in the rat: Role of kupffer cells , 1999, Hepatology.

[29]  P. Ricciardi-Castagnoli,et al.  Defective LPS signaling in C3H/HeJ and C57BL/10ScCr mice: mutations in Tlr4 gene. , 1998, Science.

[30]  D. Brenner,et al.  Estrogen increases sensitivity of hepatic Kupffer cells to endotoxin. , 1998, American journal of physiology. Gastrointestinal and liver physiology.

[31]  E. Morgan Regulation of cytochromes P450 during inflammation and infection. , 1997, Drug metabolism reviews.

[32]  M. Gossen,et al.  Doxycycline-mediated quantitative and tissue-specific control of gene expression in transgenic mice. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[33]  S. Zuckerman,et al.  In vivo modulation of murine serum tumour necrosis factor and interleukin-6 levels during endotoxemia by oestrogen agonists and antagonists. , 1995, Immunology.

[34]  G. Bishop,et al.  CpG motifs in bacterial DNA trigger direct B-cell activation , 1995, Nature.

[35]  D. Rittirsch,et al.  Immunodesign of experimental sepsis by cecal ligation and puncture , 2008, Nature Protocols.

[36]  T. Lawrence,et al.  Possible new role for NF-kappaB in the resolution of inflammation. , 2001, Nature medicine.

[37]  X. Sun,et al.  Printed in U.S.A. Copyright © 2001 by The Endocrine Society Targeted Disruption of the Mouse Estrogen Sulfotransferase Gene Reveals a Role of Estrogen Metabolism in Intracrine and Paracrine Estrogen Regulation , 2022 .