Eotaxin Triggers Eosmophil-Selective Chemotaxis and Calcium Flux via a Distinct Receptor and Induces Pulmonary Eosinophilia in the Presence of Interleukin 5 in Mice
暂无分享,去创建一个
M. van de Rijn | P. Leder | J. Bonventre | A. Luster | R. Ownbey | M. Rothenberg | H. Oettgen | P. Loiselle | P. Mehlhop | Robert T. Ownbey | P. M. Loiselle
[1] P. Leder,et al. Human eotaxin is a specific chemoattractant for eosinophil cells and provides a new mechanism to explain tissue eosinophilia , 1996, Nature Medicine.
[2] W Newman,et al. Cloning of the human eosinophil chemoattractant, eotaxin. Expression, receptor binding, and functional properties suggest a mechanism for the selective recruitment of eosinophils. , 1996, The Journal of clinical investigation.
[3] P. Foster,et al. Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model , 1996, The Journal of experimental medicine.
[4] N. Copeland,et al. Mouse Eotaxin expression parallels eosinophil accumulation during lung allergic inflammation but it is not restricted to a Th2-type response. , 1996, Immunity.
[5] P. Hodgkin,et al. IL-5-deficient mice have a developmental defect in CD5+ B-1 cells and lack eosinophilia but have normal antibody and cytotoxic T cell responses. , 1996, Immunity.
[6] A. Luster,et al. Molecular characterization of two murine eosinophil beta chemokine receptors. , 1995, Journal of immunology.
[7] T. Williams,et al. Cooperation between interleukin-5 and the chemokine eotaxin to induce eosinophil accumulation in vivo , 1995, The Journal of experimental medicine.
[8] P. Leder,et al. Murine eotaxin: an eosinophil chemoattractant inducible in endothelial cells and in interleukin 4-induced tumor suppression. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[9] C. Power,et al. Molecular Cloning and Functional Expression of a Novel CC Chemokine Receptor cDNA from a Human Basophilic Cell Line (*) , 1995, The Journal of Biological Chemistry.
[10] C. Combadière,et al. Cloning, chromosomal localization, and RNA expression of a human beta chemokine receptor-like gene. , 1995, DNA and cell biology.
[11] C. Combadière,et al. Cloning and Functional Expression of a Human Eosinophil CC Chemokine Receptor (*) , 1995, The Journal of Biological Chemistry.
[12] I. van Ark,et al. Eosinophil infiltration precedes development of airway hyperreactivity and mucosal exudation after intranasal administration of interleukin-5 to mice. , 1995, The Journal of allergy and clinical immunology.
[13] P. Leder,et al. The IP-10 chemokine binds to a specific cell surface heparan sulfate site shared with platelet factor 4 and inhibits endothelial cell proliferation , 1995, The Journal of experimental medicine.
[14] M. Baggiolini,et al. Monocyte chemotactic protein MCP-2 activates human basophil and eosinophil leukocytes similar to MCP-3. , 1995, Journal of immunology.
[15] P. Leder,et al. Constitutive and allergen-induced expression of eotaxin mRNA in the guinea pig lung , 1995, The Journal of experimental medicine.
[16] J. Gong,et al. Antagonists of monocyte chemoattractant protein 1 identified by modification of functionally critical NH2-terminal residues , 1995, Journal of Experimental Medicine.
[17] I. Adcock,et al. Eotaxin: cloning of an eosinophil chemoattractant cytokine and increased mRNA expression in allergen-challenged guinea-pig lungs. , 1994, Biochemical and biophysical research communications.
[18] D. Largaespada,et al. Lymphotactin: a cytokine that represents a new class of chemokine. , 1994, Science.
[19] T. Schall,et al. Biologic activities of the murine beta-chemokine TCA3. , 1994, Journal of immunology.
[20] B. Sykes,et al. Neutrophil activation by monomeric interleukin-8. , 1994, Science.
[21] B. Rollins,et al. Structure/activity analysis of human monocyte chemoattractant protein-1 (MCP-1) by mutagenesis. Identification of a mutated protein that inhibits MCP-1-mediated monocyte chemotaxis. , 1994, The Journal of biological chemistry.
[22] S. Coughlin,et al. Molecular cloning and functional expression of two monocyte chemoattractant protein 1 receptors reveals alternative splicing of the carboxyl-terminal tails. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[23] J. Hsuan,et al. Eotaxin: a potent eosinophil chemoattractant cytokine detected in a guinea pig model of allergic airways inflammation , 1994, The Journal of experimental medicine.
[24] M. Baggiolini,et al. CC chemokines in allergic inflammation. , 1994, Immunology today.
[25] A. Minty,et al. Monocyte chemotactic protein 3 is a most effective basophil- and eosinophil-activating chemokine , 1994, The Journal of experimental medicine.
[26] B. Dewald,et al. Interleukin-8 and related chemotactic cytokines--CXC and CC chemokines. , 1994, Advances in immunology.
[27] P. Murphy. The molecular biology of leukocyte chemoattractant receptors. , 1994, Annual review of immunology.
[28] T. Schall,et al. Molecular cloning, functional expression, and signaling characteristics of a C-C chemokine receptor , 1993, Cell.
[29] S. Georas,et al. Eosinophil transendothelial migration induced by cytokines. I. Role of endothelial and eosinophil adhesion molecules in IL-1 beta-induced transendothelial migration. , 1992, Journal of immunology.
[30] T. Schall,et al. RANTES and macrophage inflammatory protein 1 alpha induce the migration and activation of normal human eosinophil granulocytes , 1992, The Journal of experimental medicine.
[31] S. Stafford,et al. Macrophage inflammatory protein-1 alpha activates basophils and mast cells , 1992, The Journal of experimental medicine.
[32] C. Sanderson. Interleukin-5, Eosinophils, and Disease , 1992 .
[33] Hongyung Choi,et al. A murine model of allergic bronchopulmonary aspergillosis with elevated eosinophils and IgE. , 1992, Journal of immunology.
[34] A. Wardlaw,et al. Interleukin-5 selectively enhances the chemotactic response of eosinophils obtained from normal but not eosinophilic subjects. , 1992, Blood.
[35] T. Brunner,et al. Monocyte chemotactic protein 1 is a potent activator of human basophils , 1992, The Journal of experimental medicine.
[36] R. Alam,et al. Monocyte chemotactic and activating factor is a potent histamine-releasing factor for basophils. , 1992, The Journal of clinical investigation.
[37] T. Suda,et al. Analysis of the survival of mature human eosinophils: interleukin-5 prevents apoptosis in mature human eosinophils. , 1991, Blood.
[38] R. Hajjar,et al. Noncyclooxygenase metabolites of arachidonic acid amplify the vasopressin-induced Ca2+ signal in glomerular mesangial cells by releasing Ca2+ from intracellular stores. , 1991, The Journal of biological chemistry.
[39] M. Strath,et al. Eosinophilia in transgenic mice expressing interleukin 5 , 1990, The Journal of experimental medicine.
[40] W. Owen,et al. IL-5-dependent conversion of normodense human eosinophils to the hypodense phenotype uses 3T3 fibroblasts for enhanced viability, accelerated hypodensity, and sustained antibody-dependent cytotoxicity. , 1989, Journal of immunology.
[41] P. Kallós,et al. Experimental asthma in guinea pigs revisited. , 1984, International archives of allergy and applied immunology.
[42] H. Schild. Receptor Classification with Special Reference to β-Adrenergic Receptors , 1973 .