Cortical hyperexcitability and epileptogenesis: Understanding the mechanisms of epilepsy – Part 1
暂无分享,去创建一个
Richard A.L. Macdonell | R. Badawy | A. Harvey | R. Macdonell | Radwa A.B. Badawy | Anthony S. Harvey
[1] G F Cahill,et al. Brain metabolism during fasting. , 1967, The Journal of clinical investigation.
[2] R. Naquet,et al. Effects of opiate-like peptides, morphine, and naloxone in the photosensitive baboon, papio papio , 1979, Brain Research.
[3] R. Nicoll,et al. Epileptiform burst afterhyperolarization: calcium-dependent potassium potential in hippocampal CA1 pyramidal cells. , 1980, Science.
[4] L. Sokoloff,et al. Relationships among local functional activity, energy metabolism, and blood flow in the central nervous system. , 1981, Federation proceedings.
[5] R K Wong,et al. Cellular factors influencing GABA response in hippocampal pyramidal cells. , 1982, Journal of neurophysiology.
[6] B. Connors,et al. Electrophysiological properties of neocortical neurons in vitro. , 1982, Journal of neurophysiology.
[7] B. Connors. Initiation of synchronized neuronal bursting in neocortex , 1984, Nature.
[8] J. Noebels,et al. A single gene error of noradrenergic axon growth synchronizes central neurones , 1984, Nature.
[9] W. Löscher,et al. Evidence for impaired GABAergic activity in the substantia nigra of amygdaloid kindled rats , 1985, Brain Research.
[10] R. Macdonald,et al. Anticonvulsant drug mechanisms of action. , 1985, Federation proceedings.
[11] W. Löscher,et al. Low Levels of γ‐Aminobutyric Acid in Cerebrospinal Fluid of Dogs with Epilepsy , 1986 .
[12] P. Schwartzkroin,et al. Spontaneous Rhythmic Synchronous Activity in Epileptic Human and Normal Monkey Temporal Lobe , 1986, Epilepsia.
[13] F. Andermann,et al. Progressive myoclonus epilepsies: specific causes and diagnosis. , 1986, The New England journal of medicine.
[14] W. Löscher,et al. Low levels of gamma-aminobutyric acid in cerebrospinal fluid of dogs with epilepsy. , 1986, Journal of neurochemistry.
[15] Y. Mayanagi,et al. Decreased dopamine level in the epileptic focus. , 1987, Research communications in chemical pathology and pharmacology.
[16] Oxcarbazepine , 1989, The Lancet.
[17] D. McCormick,et al. GABA as an inhibitory neurotransmitter in human cerebral cortex. , 1989, Journal of neurophysiology.
[18] D. Prince,et al. Specific petit mal anticonvulsants reduce calcium currents in thalamic neurons , 1989, Neuroscience Letters.
[19] D. Prince,et al. Characterization of ethosuximide reduction of low‐threshold calcium current in thalamic neurons , 1989, Annals of neurology.
[20] J. McNamara,et al. Excitatory amino acid receptors in epilepsy. , 1990, Trends in pharmacological sciences.
[21] J. Ebersole. Electrophysiologic Methods of Evaluating Epilepsy , 1990, Seminars in neurology.
[22] P. D. De Deyn,et al. Epilepsy and the GABA-hypothesis a brief review and some examples. , 1990, Acta neurologica Belgica.
[23] Alan A. Wilson,et al. Quantification of mu and non–mu opiate receptors in temporal lobe epilepsy using positron emission tomography , 1991, Annals of neurology.
[24] J. Sikela,et al. A novel gamma subunit of the GABAA receptor identified using the polymerase chain reaction. , 1991, FEBS letters.
[25] J. Sikela,et al. A novel γ subunit of the GABAA receptor identified using the polymerase chain reaction , 1991 .
[26] Margaret Robertson,et al. Identification of a mutation in the gene causing hyperkalemic periodic paralysis , 1991, Cell.
[27] E J Speckmann,et al. The neurophysiological basis of epileptic activity: a condensed overview. , 1991, Epilepsy research. Supplement.
[28] Speckmann Ej,et al. The neurophysiological basis of epileptic activity: a condensed overview. , 1991 .
[29] M. Avoli,et al. Excitatory synaptic transmission mediated by NMDA and non-NMDA receptors in the superficial/middle layers of the epileptogenic human neocortex maintained in vitro , 1992, Neuroscience Letters.
[30] T. Jentsch,et al. Regions involved in the opening of CIC-2 chloride channel by voltage and cell volume , 1992, Nature.
[31] M. Schmutz,et al. GABAB receptors in various in vitro and in vivo models of epilepsy: A study with the GABAB receptor blocker CGP 35348 , 1992, Neuroscience.
[32] T. Serikawa,et al. Decreased Dopamine and Increased Norepinephrine Levels in the Spontaneously Epileptic Rat, a Double Mutant Rat , 1993, Epilepsia.
[33] R. Traub,et al. Are there unifying principles underlying the generation of epileptic afterdischarges in vitro? , 1994, Progress in brain research.
[34] D. Schmidt. Clobazam for Treatment of Intractable Epilepsy: A Critical Assessment , 1994, Epilepsia.
[35] B. Bourgeois. Felbamate in the Treatment of Partial‐Onset Seizures , 1994, Epilepsia.
[36] M. Kasuga,et al. Inhibition of the translocation of GLUT1 and GLUT4 in 3T3-L1 cells by the phosphatidylinositol 3-kinase inhibitor, wortmannin. , 1994, The Biochemical journal.
[37] C. Remy. Clobazam in the Treatment of Epilepsy: A Review of the Literature , 1994, Epilepsia.
[38] J. McNamara,et al. Autoantibodies to glutamate receptor GluR3 in Rasmussen's encephalitis. , 1994, Science.
[39] C. Marsden,et al. Autosomal dominant frontal epilepsy misdiagnosed as sleep disorder , 1994, The Lancet.
[40] C. B. Davis,et al. Topiramate: Preclinical Evaluation of a Structurally Novel Anticonvulsant , 1994, Epilepsia.
[41] J G Jefferys,et al. Experimental neurobiology of epilepsies. , 1994, Current opinion in neurology.
[42] JO McNamara,et al. Cellular and molecular basis of epilepsy , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[43] M. Scanziani,et al. Role of excitatory amino acid and GABAB receptors in the generation of epileptiform activity in disinhibited hippocampal slice cultures , 1994, Neuroscience.
[44] B. Meldrum. The role of glutamate in epilepsy and other CNS disorders , 1994, Neurology.
[45] P. Carlen,et al. Modulation of gap junctional mechanisms during calcium-free induced field burst activity: a possible role for electrotonic coupling in epileptogenesis , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[46] W. Catterall,et al. Role of calcium channel subtypes in calcium transients in hippocampal CA3 neurons , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[47] A. Coenen,et al. Role of L‐Type Calcium Channel Modulation in Nonconvulsive Epilepsy in Rats , 1995, Epilepsia.
[48] A. Coenen,et al. Kappa opioid receptor agonists suppress absence seizures in WAG/Rij rats , 1995, Neuroscience Letters.
[49] U. Misgeld,et al. A physiological role for GABAB receptors and the effects of baclofen in the mammalian central nervous system , 1995, Progress in Neurobiology.
[50] K. Staley,et al. Differential expression of an inwardly rectifying chloride conductance in rat brain neurons: a potential mechanism for cell-specific modulation of postsynaptic inhibition , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[51] C. Marsden,et al. Autosomal dominant nocturnal frontal lobe epilepsy. A distinctive clinical disorder. , 1995, Brain : a journal of neurology.
[52] O. Snead,et al. Basic mechanisms of generalized absence seizures , 1995, Annals of neurology.
[53] J. Aicardi,et al. Vigabatrin as Initial Therapy for Infantile Spasms: A European Retrospective Survey , 1996, Epilepsia.
[54] P. Mangan,et al. Profound disturbances of pre- and postsynaptic GABAB-receptor-mediated processes in region CA1 in a chronic model of temporal lobe epilepsy. , 1996, Journal of neurophysiology.
[55] B. Meldrum. Update on the Mechanism of Action of Antiepileptic Drugs , 1996, Epilepsia.
[56] M. Johnston. Developmental Aspects of Epileptogenesis , 1996, Epilepsia.
[57] C. Wilcox,et al. Alteration of GABAA Receptor Function Following Gene Transfer of the CLC-2 Chloride Channel , 1996, Neuron.
[58] Richard Hawkes,et al. Absence Epilepsy in Tottering Mutant Mice Is Associated with Calcium Channel Defects , 1996, Cell.
[59] Y. Jan,et al. Voltage‐gated and inwardly rectifying potassium channels , 1997, The Journal of physiology.
[60] D. Hanahan,et al. Epilepsy in mice deficient in the 65-kDa isoform of glutamic acid decarboxylase. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[61] P. Coumel,et al. A novel mutation in the potassium channel gene KVLQT1 causes the Jervell and Lange-Nielsen cardioauditory syndrome , 1997, Nature Genetics.
[62] M. Vergnes,et al. Opposite effects of GABAB receptor antagonists on absences and convulsive seizures. , 1997, European journal of pharmacology.
[63] T. Jentsch,et al. Chloride channels: An emerging molecular picture , 1997, BioEssays : news and reviews in molecular, cellular and developmental biology.
[64] W Zieglgänsberger,et al. Presynaptic and postsynaptic GABAB receptors of neocortical neurons of the rat in vitro: Differences in pharmacology and ionic mechanisms , 1997, Synapse.
[65] I. Scheffer,et al. Generalized epilepsy with febrile seizures plus. A genetic disorder with heterogeneous clinical phenotypes. , 1997, Brain : a journal of neurology.
[66] B. Gähwiler,et al. Either N- or P-type Calcium Channels Mediate GABA Release at Distinct Hippocampal Inhibitory Synapses , 1997, Neuron.
[67] S. Jordt,et al. Molecular dissection of gating in the ClC‐2 chloride channel , 1997, The EMBO journal.
[68] M. Privitera. Topiramate: A New Antiepileptic Drug , 1997, The Annals of pharmacotherapy.
[69] R. Ophoff,et al. P/Q-type Ca2+ channel defects in migraine, ataxia and epilepsy. , 1998, Trends in pharmacological sciences.
[70] Samuel F. Berkovic,et al. Febrile seizures and generalized epilepsy associated with a mutation in the Na+-channel ß1 subunit gene SCN1B , 1998, Nature Genetics.
[71] M. Schuelke,et al. Epilepsia partialis continua associated with a homoplasmic mitochondrial tRNASer(UCN) mutation , 1998, Annals of neurology.
[72] E. Ben-Menachem,et al. Zonisamide for progressive myoclonus epilepsy: long-term observations in seven patients , 1998, Epilepsy Research.
[73] P. Halász,et al. The 5-HT1A agonist 8-OH-DPAT increases the number of spike-wave discharges in a genetic rat model of absence epilepsy , 1998, Brain Research.
[74] B. Gähwiler,et al. L-Type Ca2+ channels mediate the slow Ca2+-dependent afterhyperpolarization current in rat CA3 pyramidal cells in vitro. , 1998, Journal of neurophysiology.
[75] Douglas C. Wallace,et al. Radicals r'aging , 1998, Nature Genetics.
[76] C. Marsden,et al. Autosomal dominant nocturnal frontal-lobe epilepsy: genetic heterogeneity and evidence for a second locus at 15q24. , 1998, American journal of human genetics.
[77] D. Pongratz,et al. Progressive myoclonus epilepsy and mitochondrial myopathy associated with mutations in the tRNAser(UCN) gene , 1998, Annals of neurology.
[78] D. Bertrand,et al. Properties of neuronal nicotinic acetylcholine receptor mutants from humans suffering from autosomal dominant nocturnal frontal lobe epilepsy , 1998, British journal of pharmacology.
[79] M. Schuelke,et al. Epilepsia partialis continua associated with a homoplasmic mitochondrial tRNA(Ser(UCN)) mutation. , 1998, Annals of neurology.
[80] S Clark,et al. Mechanisms of epileptogenesis. , 1999, Advances in neurology.
[81] R. Deisz. The GABAB receptor antagonist CGP 55845A reduces presynaptic GABAB actions in neocortical neurons of the rat in vitro , 1999, Neuroscience.
[82] L. Ellerby,et al. Release of caspase-9 from mitochondria during neuronal apoptosis and cerebral ischemia. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[83] J. McNamara. Emerging insights into the genesis of epilepsy , 1999, Nature.
[84] R. Dingledine,et al. The glutamate receptor ion channels. , 1999, Pharmacological reviews.
[85] J. Rho,et al. The Pharmacologic Basis of Antiepileptic Drug Action , 1999, Epilepsia.
[86] R. Naviaux,et al. Mitochondrial DNA polymerase γ deficiency and mtDNA depletion in a child with Alpers' syndrome , 1999 .
[87] R. Köhling,et al. Contribution of L-type calcium channels to epileptiform activity in hippocampal and neocortical slices of guinea-pigs , 1999, Neuroscience.
[88] R. Naviaux,et al. Mitochondrial DNA polymerase gamma deficiency and mtDNA depletion in a child with Alpers' syndrome. , 1999, Annals of neurology.
[89] S. Dimauro,et al. Mitochondrial genes for generalized epilepsies. , 1999, Advances in neurology.
[90] M. Vreugdenhil,et al. Modulation of Sodium Currents in Rat CA1 Neurons by Carbamazepine and Valproate After Kindling Epileptogenesis , 1999, Epilepsia.
[91] R. Fisher,et al. Oxcarbazepine in a monotherapy trial for partial seizures--placebo-controlled studies in neurology: where do they stop? , 1999, Neurology.
[92] M. Krug,et al. Effects of nicardipine, an antagonist of l-type voltage-dependent calcium channels, on kindling development, kindling-induced learning deficits and hippocampal potentiation phenomena , 1999, Neuropharmacology.
[93] S. Sombati,et al. Cellular Actions of Topiramate: Blockade of Kainate‐Evoked Inward Currents in Cultured Hippocampal Neurons , 2000, Epilepsia.
[94] W. Catterall,et al. From Ionic Currents to Molecular Mechanisms The Structure and Function of Voltage-Gated Sodium Channels , 2000, Neuron.
[95] T. Mori,et al. Carbon-monoxide poisoning presenting as an afebrile seizure. , 2000, Pediatric neurology.
[96] W. Catterall. Structure and regulation of voltage-gated Ca2+ channels. , 2000, Annual review of cell and developmental biology.
[97] R. D'Hooge,et al. Pathophysiology of epilepsy. , 2000, Acta neurologica Belgica.
[98] A. Marson,et al. Zonisamide for drug-resistant partial epilepsy. , 2000, The Cochrane database of systematic reviews.
[99] Christopher Miller. An overview of the potassium channel family , 2000, Genome Biology.
[100] M. Berger,et al. Colocalization and coassembly of two human brain M-type potassium channel subunits that are mutated in epilepsy. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[101] C. Dravet. Severe Myoclonic Epilepsy in Infants and its Related Syndromes , 1999, Epilepsia.
[102] D. Gilbert,et al. The Ketogenic Diet: Seizure Control Correlates Better With Serum β-Hydroxybutyrate Than With Urine Ketones , 2000, Journal of child neurology.
[103] N. Nishiyama,et al. L-type Ca2+ channel blocker inhibits mossy fiber sprouting and cognitive deficits following pilocarpine seizures in immature mice , 2000, Neuroscience.
[104] F. Ashcroft. The Yin and Yang of the K(ATP) channel. , 2000, The Journal of physiology.
[105] S. Gultekin,et al. Paraneoplastic limbic encephalitis: neurological symptoms, immunological findings and tumour association in 50 patients. , 2000, Brain : a journal of neurology.
[106] A. Brooks-Kayal,et al. Role of excitatory amino acids in developmental epilepsies. , 2001, Mental Retardation and Developmental Disabilities Research Reviews.
[107] H. Lerche,et al. Ion channels and epilepsy. , 2001, American journal of medical genetics.
[108] J. Stephenson. Anoxic seizures: self-terminating syncopes. , 2001, Epileptic disorders : international epilepsy journal with videotape.
[109] John Gordon Ralph Jefferys,et al. Ca2+ entry through l-type Ca2+ channels helps terminate epileptiform activity by activation of a Ca2+ dependent afterhyperpolarisation in hippocampal CA3. , 2001, Neuroscience.
[110] D. Bertrand,et al. Neuronal nicotinic receptors: from protein structure to function , 2001, FEBS letters.
[111] E. Faught,et al. Pharmacokinetic Considerations in Prescribing Antiepileptic Drugs , 2001, Epilepsia.
[112] D. McCormick,et al. On the cellular and network bases of epileptic seizures. , 2001, Annual review of physiology.
[113] D. Lowenstein,et al. Hippocampal Heterotopia Lack Functional Kv4.2 Potassium Channels in the Methylazoxymethanol Model of Cortical Malformations and Epilepsy , 2001, The Journal of Neuroscience.
[114] J. Dichgans,et al. GluR3 antibodies: Prevalence in focal epilepsy but no specificity for Rasmussen’s encephalitis , 2001, Neurology.
[115] Michel Baulac,et al. First genetic evidence of GABAA receptor dysfunction in epilepsy: a mutation in the γ2-subunit gene , 2001, Nature Genetics.
[116] A. George,et al. Molecular Basis of an Inherited Epilepsy , 2002, Neuron.
[117] I. Scheffer,et al. Sodium-channel defects in benign familial neonatal-infantile seizures , 2002, The Lancet.
[118] Wei-Yang Lu,et al. Mutation of GABRA1 in an autosomal dominant form of juvenile myoclonic epilepsy , 2002, Nature Genetics.
[119] R. Köhling. Neuroscience. GABA becomes exciting. , 2002, Science.
[120] T. Voit,et al. Facilitated glucose transporter protein type 1 (GLUT1) deficiency syndrome: impaired glucose transport into brain – a review , 2002, European Journal of Pediatrics.
[121] T. Baram,et al. Developmental Febrile Seizures Modulate Hippocampal Gene Expression of Hyperpolarization-Activated Channels in an Isoform- and Cell-Specific Manner , 2002, The Journal of Neuroscience.
[122] R. Spreafico,et al. Antibodies against GluR3 peptides are not specific for Rasmussen's encephalitis but are also present in epilepsy patients with severe, early onset disease and intractable seizures , 2002, Journal of Neuroimmunology.
[123] Steven Petrou,et al. Truncation of the GABA(A)-receptor gamma2 subunit in a family with generalized epilepsy with febrile seizures plus. , 2002, American journal of human genetics.
[124] D. Bertrand,et al. Nicotinic receptors in circuit excitability and epilepsy. , 2002, Journal of neurobiology.
[125] W. Kunz. The role of mitochondria in epileptogenesis , 2002, Current opinion in neurology.
[126] Rüdiger Köhling,et al. GABA Becomes Exciting , 2002, Science.
[127] R. Deisz. Cellular mechanisms of pharmacoresistance in slices from epilepsy surgery. , 2002, Novartis Foundation symposium.
[128] W. Hauser,et al. Mutations in LGI1 cause autosomal-dominant partial epilepsy with auditory features , 2002, Nature Genetics.
[129] H. Lester,et al. Five ADNFLE Mutations Reduce the Ca2+ Dependence of the Mammalian α4β2 Acetylcholine Response , 2003 .
[130] H. Lester,et al. Five ADNFLE mutations reduce the Ca2+ dependence of the mammalian alpha4beta2 acetylcholine response. , 2003, The Journal of physiology.
[131] S. Horvath,et al. Mutations in CLCN2 encoding a voltage-gated chloride channel are associated with idiopathic generalized epilepsies , 2003, Nature Genetics.
[132] H. Beck,et al. Enhanced Expression of a Specific Hyperpolarization-Activated Cyclic Nucleotide-Gated Cation Channel (HCN) in Surviving Dentate Gyrus Granule Cells of Human and Experimental Epileptic Hippocampus , 2003, The Journal of Neuroscience.
[133] O. Devinsky,et al. Epilepsy-Associated Dysfunction in the Voltage-Gated Neuronal Sodium Channel SCN1A , 2003, The Journal of Neuroscience.
[134] T. Freund,et al. Loss of interneurons innervating pyramidal cell dendrites and axon initial segments in the CA1 region of the hippocampus following pilocarpine‐induced seizures , 2003, The Journal of comparative neurology.
[135] I. Scheffer,et al. Phenotypic Comparison of Two Scottish Families with Mutations in Different Genes Causing Autosomal Dominant Nocturnal Frontal Lobe Epilepsy , 2003, Epilepsia.
[136] Zhijian Yao,et al. Association between genetic variation of CACNA1H and childhood absence epilepsy , 2003, Annals of neurology.
[137] D. Bertrand,et al. A New Chrna4 Mutation with Low Penetrance in Nocturnal Frontal Lobe Epilepsy , 2003, Epilepsia.
[138] Simon Shorvon,et al. Treatment of Epilepsy , 2004 .
[139] B. Boeve,et al. Potentially reversible autoimmune limbic encephalitis with neuronal potassium channel antibody , 2004, Neurology.
[140] K. Metrakos,et al. Plasma amino acids in 3/sec spike-wave epilepsy , 1980, Neurochemical Research.
[141] M. Rossor,et al. Potassium channel antibody-associated encephalopathy: a potentially immunotherapy-responsive form of limbic encephalitis. , 2004, Brain : a journal of neurology.
[142] D. Johnston,et al. Seizure-Induced Plasticity of h Channels in Entorhinal Cortical Layer III Pyramidal Neurons , 2004, Neuron.
[143] D. Johnston,et al. Acquired Dendritic Channelopathy in Temporal Lobe Epilepsy , 2004, Science.
[144] C. Woods,et al. Homozygosity for a missense mutation in the 67 kDa isoform of glutamate decarboxylase in a family with autosomal recessive spastic cerebral palsy: parallels with Stiff-Person Syndrome and other movement disorders , 2004, BMC neurology.
[145] R. Vannucci,et al. Glucose transport in developing rat brain: Glucose transporter proteins, rate constants and cerebral glucose utilization , 1994, Molecular and Cellular Biochemistry.
[146] Ying-Hui Fu,et al. Channels and disease: past, present, and future. , 2004, Archives of neurology.
[147] I. Scheffer,et al. Benign familial neonatal‐infantile seizures: Characterization of a new sodium channelopathy , 2004, Annals of neurology.
[148] Luigi Ferini-Strambi,et al. Autosomal dominant nocturnal frontal lobe epilepsy , 2004, Journal of Neurology.
[149] M. Cunnington. The International Lamotrigine Pregnancy Registry Update for the Epilepsy Foundation , 2004, Epilepsia.
[150] S. Moshé,et al. Malic enzyme 2 may underlie susceptibility to adolescent-onset idiopathic generalized epilepsy. , 2005, American journal of human genetics.
[151] C. Elger,et al. Pathogenesis, diagnosis and treatment of Rasmussen encephalitis: a European consensus statement. , 2005, Brain : a journal of neurology.
[152] T. Freund,et al. Surviving CA1 pyramidal cells receive intact perisomatic inhibitory input in the human epileptic hippocampus. , 2004, Brain : a journal of neurology.
[153] A. Vincent,et al. Autoimmune channelopathies , 2005, Nature Clinical Practice Neurology.
[154] R. Olsen,et al. GABAA Receptor-Associated Protein Regulates GABAA Receptor Cell-Surface Number in Xenopus laevis Oocytes , 2005, Molecular Pharmacology.
[155] Mark Gardiner,et al. Genetics of Idiopathic Generalized Epilepsies , 2005, Epilepsia.
[156] J. Kantrowitz,et al. Synaptic depolarizing GABA Response in adults is excitatory and proconvulsive when GABAB receptors are blocked. , 2005, Journal of neurophysiology.
[157] P. Murphy. Use of the ketogenic diet as a treatment for epilepsy refractory to drug treatment , 2005, Expert review of neurotherapeutics.
[158] T. Baram,et al. Formation of heteromeric hyperpolarization-activated cyclic nucleotide-gated (HCN) channels in the hippocampus is regulated by developmental seizures , 2005, Neurobiology of Disease.
[159] G. Lu,et al. Protein Kinase C-Independent Effects of Protein Kinase D3 in Glucose Transport in L6 Myotubes , 2005, Molecular Pharmacology.
[160] E. Kobayashi,et al. Familial Temporal Lobe Epilepsy with Auditory Features , 2005, Epilepsia.
[161] D. Binder,et al. Functional changes in astroglial cells in epilepsy , 2006, Glia.
[162] B. Fakler,et al. The Epilepsy-Linked Lgi1 Protein Assembles into Presynaptic Kv1 Channels and Inhibits Inactivation by Kvβ1 , 2006, Neuron.
[163] Samuel F. Berkovic,et al. Human epilepsies: interaction of genetic and acquired factors , 2006, Trends in Neurosciences.
[164] M. Avoli,et al. Synaptic hyperexcitability of deep layer neocortical cells in a genetic model of absence seizures , 2005, Genes, brain, and behavior.
[165] M. Leppert,et al. Andreas Rett and benign familial neonatal convulsions revisited , 2006, Neurology.
[166] I. Scheffer,et al. Extended spectrum of idiopathic generalized epilepsies associated with CACNA1H functional variants , 2007, Annals of neurology.
[167] E. Kossoff,et al. The Ketogenic Diet: One Decade Later , 2007, Pediatrics.
[168] V. Napolioni,et al. Genetic polymorphisms and idiopathic generalized epilepsies. , 2007, Pediatric neurology.
[169] M. Seeck,et al. Epilepsy and cerebellar ataxia associated with anti-glutamic acid decarboxylase antibodies , 2009, BMJ Case Reports.
[170] Barbara C. Jobst,et al. Handbook of Epilepsy Treatment By Simon Shorvon 2010 Wiley–Blackwell , 2011, Epilepsy & Behavior.