Dysregulation of glucose, insulin, triglyceride, blood pressure, and oxidative stress after an oral glucose tolerance test in men with abdominal obesity.

[1]  M. Matsuda,et al.  Insulin sensitivity indices obtained from oral glucose tolerance testing: comparison with the euglycemic insulin clamp. , 1999, Diabetes care.

[2]  S. Kohno,et al.  Evaluation of glucose tolerance, post-prandial hyperglycemia and hyperinsulinemia influencing the incidence of coronary heart disease. , 2007, Internal medicine.

[3]  N. Kohno,et al.  A protective effect of adiponectin against oxidative stress in Japanese Americans: the association between adiponectin or leptin and urinary isoprostane. , 2005, Metabolism: clinical and experimental.

[4]  E. Bonora,et al.  Postprandial blood glucose as a risk factor for cardiovascular disease in Type II diabetes: the epidemiological evidence , 2001, Diabetologia.

[5]  Amir Peleg,et al.  Power spectral analysis of heart rate variability during the 100-g oral glucose tolerance test in pregnant women. , 2006, Diabetes care.

[6]  Y. Tabara,et al.  Serum resistin is reduced by glucose and meal loading in healthy human subjects. , 2008, Metabolism: clinical and experimental.

[7]  B. Nordestgaard,et al.  Nonfasting triglycerides and risk of myocardial infarction, ischemic heart disease, and death in men and women. , 2007, JAMA.

[8]  Alan Rees,et al.  Postprandial glucose - a potential therapeutic target to reduce cardiovascular mortality. , 2009, Current vascular pharmacology.

[9]  J. Després,et al.  Abdominal obesity and metabolic syndrome , 2006, Nature.

[10]  Acute fructose administration decreases the glycemic response to an oral glucose tolerance test in normal adults. , 2000, The Journal of clinical endocrinology and metabolism.

[11]  R. Paschke,et al.  Hormonal regulation of adiponectin gene expression in 3T3-L1 adipocytes. , 2002, Biochemical and biophysical research communications.

[12]  A Sekikawa,et al.  Impaired glucose tolerance is a risk factor for cardiovascular disease, but not impaired fasting glucose. The Funagata Diabetes Study. , 1999, Diabetes care.

[13]  W. W. Lautt,et al.  Postprandial insulin resistance as an early predictor of cardiovascular risk , 2007, Therapeutics and clinical risk management.

[14]  J. O’Keefe,et al.  Postprandial hyperglycemia/hyperlipidemia (postprandial dysmetabolism) is a cardiovascular risk factor. , 2007, The American journal of cardiology.

[15]  T. Funahashi,et al.  Metabolic syndrome: Clinical concept and molecular basis , 2007, Annals of medicine.

[16]  T. Funahashi,et al.  cDNA cloning and expression of a novel adipose specific collagen-like factor, apM1 (AdiPose Most abundant Gene transcript 1). , 1996, Biochemical and biophysical research communications.

[17]  K. Yagi,et al.  A simple fluorometric assay for lipoperoxide in blood plasma. , 1976, Biochemical medicine.

[18]  G. Yoshino,et al.  Serum concentration of small dense low-density lipoprotein-cholesterol during oral glucose tolerance test and oral fat tolerance test. , 2008, Clinica chimica acta; international journal of clinical chemistry.

[19]  M. S. Lee,et al.  Postprandial hypertriglyceridemia impairs endothelial function by enhanced oxidant stress. , 2001, Atherosclerosis.

[20]  T Nakamura,et al.  Paradoxical decrease of an adipose-specific protein, adiponectin, in obesity. , 1999, Biochemical and biophysical research communications.

[21]  K. Kugiyama,et al.  Hyperglycemia rapidly suppresses flow-mediated endothelium-dependent vasodilation of brachial artery. , 1999, Journal of the American College of Cardiology.

[22]  R. Testa,et al.  Glucose "peak" and glucose "spike": Impact on endothelial function and oxidative stress. , 2008, Diabetes research and clinical practice.

[23]  E. Feskens,et al.  Glucose tolerance and cardiovascular mortality: comparison of fasting and 2-hour diagnostic criteria. , 2001, Archives of internal medicine.

[24]  T. Fujita The metabolic syndrome in Japan , 2008, Nature Clinical Practice Cardiovascular Medicine.

[25]  H. Iso,et al.  Serum triglycerides and risk of coronary heart disease among Japanese men and women. , 2001, American journal of epidemiology.

[26]  Eiji Oda,et al.  New criteria for 'obesity disease' in Japan. , 2002, Circulation journal : official journal of the Japanese Circulation Society.

[27]  M. Suchard,et al.  Alterations in the dynamics of circulating ghrelin, adiponectin, and leptin in human obesity. , 2004, Proceedings of the National Academy of Sciences of the United States of America.

[28]  P. Ridker,et al.  Fasting compared with nonfasting triglycerides and risk of cardiovascular events in women. , 2007, JAMA.

[29]  G. Paolisso,et al.  Oxidative Stress and Diabetic Vascular Complications , 1996, Diabetes Care.

[30]  T. Rabelink,et al.  Endothelial function in the post-prandial state. , 2002, Atherosclerosis. Supplements.

[31]  K. Node,et al.  Postprandial hyperglycemia as an etiological factor in vascular failure , 2009, Cardiovascular diabetology.

[32]  P. Zimmet,et al.  Definition, diagnosis and classification of diabetes mellitus and its complications. Part 1: diagnosis and classification of diabetes mellitus. Provisional report of a WHO Consultation , 1998, Diabetic medicine : a journal of the British Diabetic Association.

[33]  R. Heine,et al.  Two consecutive high‐fat meals affect endothelial‐dependent vasodilation, oxidative stress and cellular microparticles in healthy men , 2006, Journal of thrombosis and haemostasis : JTH.

[34]  Morihiro Matsuda,et al.  Increased oxidative stress in obesity and its impact on metabolic syndrome. , 2004, The Journal of clinical investigation.

[35]  B. Frei,et al.  Obesity and oxidative stress: a direct link to CVD? , 2003, Arteriosclerosis, thrombosis, and vascular biology.

[36]  D. B. Zilversmit Atherogenesis: a postprandial phenomenon. , 1979, Circulation.

[37]  M. Matsuda,et al.  Insulin resistance and coronary artery disease , 1996, Diabetologia.

[38]  H. Matsuoka,et al.  Endothelial dysfunction associated with oxidative stress in human. , 2001, Diabetes research and clinical practice.

[39]  John P H Wilding,et al.  Plasma adiponectin increases postprandially in obese, but not in lean, subjects. , 2003, Obesity research.

[40]  M. Horowitz,et al.  The effects of drinks made from simple sugars on blood pressure in healthy older people. , 2005, The British journal of nutrition.

[41]  H. Hayashi,et al.  Post-challenge hyperinsulinaemia rather than hyperglycaemia is associated with the severity of coronary artery disease in patients without a previous diagnosis of diabetes mellitus , 2005, Heart.