CCR4 involvement in expansion of Th17 cells in a mouse model of psoriasis.

[1]  Cheng Chang,et al.  In-depth serum proteomics reveals biomarkers of psoriasis severity and response to traditional Chinese medicine , 2019, Theranostics.

[2]  W. Boehncke,et al.  The IL-17 Family of Cytokines in Psoriasis: IL-17A and Beyond , 2018, Front. Immunol..

[3]  Shinya Yamamoto,et al.  A CCR4 antagonist enhances DC activation and homing to the regional lymph node and shows potent vaccine adjuvant activity through the inhibition of regulatory T-cell recruitment. , 2018, Journal of pharmacological sciences.

[4]  J. Renauld,et al.  Ccr6 Is Dispensable for the Development of Skin Lesions Induced by Imiquimod despite its Effect on Epidermal Homing of IL-22-Producing Cells. , 2017, The Journal of investigative dermatology.

[5]  R. Förster,et al.  Dendritic cell migration in health and disease , 2016, Nature Reviews Immunology.

[6]  M. Diani,et al.  T Helper Cell Subsets in Clinical Manifestations of Psoriasis , 2016, Journal of immunology research.

[7]  A. Kawada,et al.  CCR4 is critically involved in effective antitumor immunity in mice bearing intradermal B16 melanoma. , 2016, Cancer letters.

[8]  S. Kusunoki,et al.  C-C chemokine receptor type 4 antagonist Compound 22 ameliorates experimental autoimmune encephalomyelitis , 2016, Journal of Neuroimmunology.

[9]  W. Boehncke.  Etiology and Pathogenesis of Psoriasis. , 2015, Rheumatic diseases clinics of North America.

[10]  A. Luster,et al.  Chemokine-guided cell positioning in the lymph node orchestrates the generation of adaptive immune responses. , 2015, Current opinion in cell biology.

[11]  K. Matsushima,et al.  CCR4 and its ligands: from bench to bedside. , 2015, International immunology.

[12]  A. Mantovani,et al.  International Union of Basic and Clinical Pharmacology. LXXXIX. Update on the Extended Family of Chemokine Receptors and Introducing a New Nomenclature for Atypical Chemokine Receptors , 2014, Pharmacological Reviews.

[13]  N. Mehta,et al.  Modulation of cardiometabolic pathways in skin and serum from patients with psoriasis , 2013, Journal of Translational Medicine.

[14]  Ido D. Weiss,et al.  CCR6 is required for epidermal trafficking of γδ T cells in an IL-23-induced model of psoriasiform dermatitis , 2012, The Journal of investigative dermatology.

[15]  A. Zlotnik,et al.  The chemokine superfamily revisited. , 2012, Immunity.

[16]  J. Renauld,et al.  IL-22 Is Required for Imiquimod-Induced Psoriasiform Skin Inflammation in Mice , 2011, The Journal of Immunology.

[17]  T. Mabuchi,et al.  Chemokine receptors in the pathogenesis and therapy of psoriasis. , 2012, Journal of dermatological science.

[18]  C. Johansen,et al.  Kinetics and differential expression of the skin‐related chemokines CCL27 and CCL17 in psoriasis, atopic dermatitis and allergic contact dermatitis , 2011, Experimental dermatology.

[19]  J. Xiang,et al.  A Distinct Role of CD4+ Th17- and Th17-Stimulated CD8+ CTL in the Pathogenesis of Type 1 Diabetes and Experimental Autoimmune Encephalomyelitis , 2011, Journal of Clinical Immunology.

[20]  P. Hwu,et al.  T helper 17 cells promote cytotoxic T cell activation in tumor immunity. , 2009, Immunity.

[21]  J. Farber,et al.  CCR6 is required for IL-23-induced psoriasis-like inflammation in mice. , 2009, The Journal of clinical investigation.

[22]  M. Röcken,et al.  Angiogenesis drives psoriasis pathogenesis , 2009, International journal of experimental pathology.

[23]  F. Nestle,et al.  The IL-23/Th17 axis in the immunopathogenesis of psoriasis. , 2009, The Journal of investigative dermatology.

[24]  L. Boon,et al.  Imiquimod-Induced Psoriasis-Like Skin Inflammation in Mice Is Mediated via the IL-23/IL-17 Axis1 , 2009, The Journal of Immunology.

[25]  V. Kuchroo,et al.  Role of Th1 and Th17 cells in organ-specific autoimmunity. , 2008, Journal of autoimmunity.

[26]  D. Jarrossay,et al.  Surface phenotype and antigenic specificity of human interleukin 17–producing T helper memory cells , 2007, Nature Immunology.

[27]  J. Carucci,et al.  Major differences in inflammatory dendritic cells and their products distinguish atopic dermatitis from psoriasis. , 2007, The Journal of allergy and clinical immunology.

[28]  K. McIntyre,et al.  Core exploration in optimization of chemokine receptor CCR4 antagonists. , 2007, Bioorganic & medicinal chemistry letters.

[29]  Kathleen M. Smith,et al.  IL-23 stimulates epidermal hyperplasia via TNF and IL-20R2–dependent mechanisms with implications for psoriasis pathogenesis , 2006, The Journal of experimental medicine.

[30]  Donald Y M Leung,et al.  Amendment history : Erratum ( April 2004 ) New insights into atopic dermatitis , 2018 .

[31]  K. Matsushima,et al.  Macrophage-derived chemokine (MDC/CCL22) and CCR4 are involved in the formation of T lymphocyte-dendritic cell clusters in human inflamed skin and secondary lymphoid tissue. , 2001, The American journal of pathology.

[32]  C. Caux,et al.  Up-Regulation of Macrophage Inflammatory Protein-3α/CCL20 and CC Chemokine Receptor 6 in Psoriasis1 , 2000, The Journal of Immunology.

[33]  K. Matsushima,et al.  Selective recruitment of CCR4-bearing Th2 cells toward antigen-presenting cells by the CC chemokines thymus and activation-regulated chemokine and macrophage-derived chemokine. , 1999, International immunology.