Repeated stimulation of the ventral tegmental area sensitizes the hyperlocomotor response to amphetamine

[1]  J. B. Justice,et al.  Sensitization and individual differences to IP amphetamine, cocaine, or caffeine following repeated intracranial amphetamine infusions , 1992, Pharmacology Biochemistry and Behavior.

[2]  T. Borowski,et al.  Long-term influence of d-amphetamine on mesolimbic brain-stimulation reward: Comparison to chronic haloperidol and naloxone effects , 1992, Pharmacology Biochemistry and Behavior.

[3]  D. Segal,et al.  In vivo microdialysis reveals a diminished amphetamine-induced DA response corresponding to behavioral sensitization produced by repeated amphetamine pretreatment , 1992, Brain Research.

[4]  M. Wolf,et al.  Repeated administration of MK-801 produces sensitization to its own locomotor stimulant effects but blocks sensitization to amphetamine , 1991, Brain Research.

[5]  P. Verbanck,et al.  Acute amphetamine-induced subsensitivity of A10 dopamine autoreceptors in vitro , 1991, Brain Research.

[6]  H. Ujike,et al.  Co-administration of either a selective D1 or D2 dopamine antagonist with methamphetamine prevents methamphetamine-induced behavioral sensitization and neurochemical change, studied by in vivo intracerebral dialysis , 1991, Brain Research.

[7]  S. L. Patrick,et al.  Concomitant sensitization of amphetamine-induced behavioral stimulation and in vivo dopamine release from rat caudate nucleus , 1991, Brain Research.

[8]  A. Phillips,et al.  Amphetamine-like stimulus properties produced by electrical stimulation of reward sites in the ventral tegmental area , 1990, Behavioural Brain Research.

[9]  P. Kalivas,et al.  Amphetamine injection into the ventral mesencephalon sensitizes rats to peripheral amphetamine and cocaine. , 1988, The Journal of pharmacology and experimental therapeutics.

[10]  T. Robinson,et al.  Enduring changes in brain and behavior produced by chronic amphetamine administration: A review and evaluation of animal models of amphetamine psychosis , 1986, Brain Research Reviews.

[11]  A. Crane,et al.  Metabolic mapping of the brain during rewarding self-stimulation. , 1984, Science.

[12]  P. Milner,et al.  Response involvement in brain stimulation reward , 1981, Physiology & Behavior.

[13]  E. Ellinwood,et al.  Chronic D-amphetamine in nucleus accumbens: lack of tolerance or reverse tolerance of locomotor activity. , 1981, Life sciences.

[14]  R. Post.  Intermittent versus continuous stimulation: effect of time interval on the development of sensitization or tolerance. , 1980, Life sciences.

[15]  S. Antelman,et al.  Sensitization to amphetamine and stress may involve nucleus accumbens and medial frontal cortex , 1979, Brain Research.

[16]  S. Iversen,et al.  Selective 60HDA-induced destruction of mesolimbic dopamine neurons: Abolition of psychostimulant-induced locomotor activity in rats , 1976 .

[17]  S. Iversen,et al.  Amphetamine and apomorphine responses in the rat following 6-OHDA lesions of the nucleus accumbens septi and corpus striatum , 1975, Brain Research.

[18]  J. Rossum,et al.  Stimulation of locomotor activity following injection of dopamine into the nucleus accumbens , 1973, The Journal of pharmacy and pharmacology.

[19]  J. V. van Rossum,et al.  Ergometrine induced locomotor activity following intracerebral injection into the nucleus accumbens. , 1973, Brain research.

[20]  H. Pakkenberg,et al.  Behavioral effects of dopamine and p-hydroxyamphetamine injected into corpus striatum of rats. , 1971, Experimental neurology.

[21]  F. J. White,et al.  Differential development of autoreceptor subsensitivity and enhanced dopamine release during amphetamine sensitization. , 1993, The Journal of pharmacology and experimental therapeutics.

[22]  R. Kirkby,et al.  Alterations in amphetamine-induced locomotor activity and stereotypy after electrical stimulation of the nucleus accumbens and neostriatum. , 1989, Life sciences.

[23]  L. Pellegrino,et al.  stereotaxic atlas of the rat brain , 1967 .