Failed upregulation of TFAM protein and mitochondrial DNA in oxidatively deficient fibers of chronic obstructive pulmonary disease locomotor muscle

[1]  Robert W. Taylor,et al.  A novel immunofluorescent assay to investigate oxidative phosphorylation deficiency in mitochondrial myopathy: understanding mechanisms and improving diagnosis , 2015, Scientific Reports.

[2]  Robert W. Taylor,et al.  Mitochondrial and inflammatory changes in sporadic inclusion body myositis , 2015, Neuropathology and applied neurobiology.

[3]  Feng Liu,et al.  A systematic review and meta-analysis 2 , 2015 .

[4]  M. Meaney,et al.  Human skeletal muscle biopsy procedures using the modified Bergström technique. , 2014, Journal of visualized experiments : JoVE.

[5]  Thierry Troosters,et al.  An official American Thoracic Society/European Respiratory Society statement: update on limb muscle dysfunction in chronic obstructive pulmonary disease. , 2014, American journal of respiratory and critical care medicine.

[6]  E. Barreiro.  Protein carbonylation and muscle function in COPD and other conditions. , 2014, Mass spectrometry reviews.

[7]  E. Barreiro,et al.  The Muscle Oxidative Regulatory Response to Acute Exercise Is Not Impaired in Less Advanced COPD Despite a Decreased Oxidative Phenotype , 2014, PloS one.

[8]  Yana Konokhova.  Mitochondrial DNA deletions correspond to high levels of oxidative DNA damage in skeletal muscle of patients with chronic obstructive pulmonary disease , 2014 .

[9]  C. Leeuwenburgh,et al.  Aging and Calorie Restriction Oppositely Affect Mitochondrial Biogenesis through TFAM Binding at Both Origins of Mitochondrial DNA Replication in Rat Liver , 2013, PloS one.

[10]  C. Prefaut,et al.  Reference values for vastus lateralis fiber size and type in healthy subjects over 40 years old: a systematic review and metaanalysis. , 2013, Journal of applied physiology.

[11]  P. Diemunsch,et al.  Skeletal muscle mitochondrial dysfunction during chronic obstructive pulmonary disease: central actor and therapeutic target , 2013, Experimental physiology.

[12]  M. Kelders,et al.  Loss of quadriceps muscle oxidative phenotype and decreased endurance in patients with mild-to-moderate COPD. , 2013, Journal of applied physiology.

[13]  D. Turnbull,et al.  Mitochondrial DNA deletions and depletion within paraspinal muscles , 2012, Neuropathology and applied neurobiology.

[14]  M. Polkey,et al.  Pathways associated with reduced quadriceps oxidative fibres and endurance in COPD , 2012, European Respiratory Journal.

[15]  Robert W. Taylor,et al.  Cytochrome c oxidase-intermediate fibres: Importance in understanding the pathogenesis and treatment of mitochondrial myopathy , 2012, Neuromuscular Disorders.

[16]  J. Roca,et al.  Does oxidative stress modulate limb muscle atrophy in severe COPD patients? , 2012, European Respiratory Journal.

[17]  Stephen A. Foulis,et al.  Age-related changes in oxidative capacity differ between locomotory muscles and are associated with physical activity behavior. , 2012, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.

[18]  A. Lezza.  Mitochondrial transcription factor A (TFAM): one actor for different roles , 2012, Frontiers in Biology.

[19]  D. Turnbull,et al.  Habitual Physical Activity in Mitochondrial Disease , 2011, PloS one.

[20]  A. Vainshtein,et al.  Mechanisms of exercise-induced mitochondrial biogenesis in skeletal muscle: implications for health and disease. , 2011, Comprehensive Physiology.

[21]  A. Agustí,et al.  Effects of exercise on mitochondrial DNA content in skeletal muscle of patients with COPD , 2010, Thorax.

[22]  V. Peinado,et al.  Cigarette smoke-induced oxidative stress: A role in chronic obstructive pulmonary disease skeletal muscle dysfunction. , 2010, American journal of respiratory and critical care medicine.

[23]  Laura C. Greaves,et al.  Mitochondrial DNA defects and selective extraocular muscle involvement in CPEO. , 2010, Investigative ophthalmology & visual science.

[24]  P. Chinnery,et al.  OPA1 mutations cause cytochrome c oxidase deficiency due to loss of wild-type mtDNA molecules , 2010, Human molecular genetics.

[25]  G. Couper,et al.  Defective DNA Replication Impairs Mitochondrial Biogenesis In Human Failing Hearts , 2010, Circulation research.

[26]  A. Agustí,et al.  Abnormal mitochondrial function in locomotor and respiratory muscles of COPD patients , 2009, European Respiratory Journal.

[27]  H. Magnussen,et al.  Physical activity in patients with COPD , 2008, European Respiratory Journal.

[28]  J. Bourbeau,et al.  The mitochondrial phenotype of peripheral muscle in chronic obstructive pulmonary disease: disuse or dysfunction? , 2008, American journal of respiratory and critical care medicine.

[29]  F. Maltais,et al.  The major limitation to exercise performance in COPD is lower limb muscle dysfunction. , 2008, Journal of applied physiology.

[30]  J. Gregory,et al.  DNA digestion to deoxyribonucleoside: a simplified one-step procedure. , 2008, Analytical biochemistry.

[31]  R. Haller,et al.  The effect of training on the expression of mitochondrial biogenesis- and apoptosis-related proteins in skeletal muscle of patients with mtDNA defects. , 2007, American journal of physiology. Endocrinology and metabolism.

[32]  P. Schrauwen,et al.  Peroxisome proliferator-activated receptor expression is reduced in skeletal muscle in COPD , 2007, European Respiratory Journal.

[33]  M. Hesselink,et al.  Reduced mitochondrial density in the vastus lateralis muscle of patients with COPD , 2007, European Respiratory Journal.

[34]  P. Wagner.  Skeletal muscles in chronic obstructive pulmonary disease: Deconditioning, or myopathy? , 2006, Respirology.

[35]  K. Rabe.  Guidelines for chronic obstructive pulmonary disease treatment and issues of implementation. , 2006, Proceedings of the American Thoracic Society.

[36]  S. Salamat,et al.  Mitochondrial DNA-deletion mutations accumulate intracellularly to detrimental levels in aged human skeletal muscle fibers. , 2006, American journal of human genetics.

[37]  C. Prefaut,et al.  From muscle disuse to myopathy in COPD: potential contribution of oxidative stress , 2005, European Respiratory Journal.

[38]  Thierry Troosters,et al.  Activity monitoring for assessment of physical activities in daily life in patients with chronic obstructive pulmonary disease. , 2005, Archives of physical medicine and rehabilitation.

[39]  Margaret A. Johnson,et al.  Methods of microphotometric assay of succinate dehydrogenase and cytochromec oxidase activities for use on human skeletal muscle , 1989, The Histochemical Journal.

[40]  F. Maltais,et al.  Peripheral muscle endurance and the oxidative profile of the quadriceps in patients with COPD , 2004, Thorax.

[41]  Robert W. Taylor,et al.  The diagnosis of mitochondrial muscle disease , 2004, Neuromuscular Disorders.

[42]  E. Wouters,et al.  Myopathological features in skeletal muscle of patients with chronic obstructive pulmonary disease , 2003, European Respiratory Journal.

[43]  S. Hussain,et al.  Nitric oxide synthases and protein oxidation in the quadriceps femoris of patients with chronic obstructive pulmonary disease. , 2003, American journal of respiratory cell and molecular biology.

[44]  François Maltais,et al.  Exercise-induced quadriceps oxidative stress and peripheral muscle dysfunction in patients with chronic obstructive pulmonary disease. , 2003, American journal of respiratory and critical care medicine.

[45]  R. Ross,et al.  ATS/ACCP statement on cardiopulmonary exercise testing. , 2003, American journal of respiratory and critical care medicine.

[46]  S. Dimauro,et al.  The spectrum of exercise tolerance in mitochondrial myopathies: a study of 40 patients. , 2003, Brain : a journal of neurology.

[47]  Y. Lacasse,et al.  Midthigh muscle cross-sectional area is a better predictor of mortality than body mass index in patients with chronic obstructive pulmonary disease. , 2002, American journal of respiratory and critical care medicine.

[48]  Robert W. Taylor,et al.  Detection and quantification of mitochondrial DNA deletions in individual cells by real-time PCR. , 2002, Nucleic acids research.

[49]  H. van Mameren,et al.  Skeletal muscle fibre-type shifting and metabolic profile in patients with chronic obstructive pulmonary disease , 2002, European Respiratory Journal.

[50]  E. Shoubridge,et al.  Aerobic conditioning in patients with mitochondrial myopathies: Physiological, biochemical, and genetic effects , 2001, Annals of neurology.

[51]  F. Maltais,et al.  Oxidative enzyme activities of the vastus lateralis muscle and the functional status in patients with COPD , 2000, Thorax.

[52]  Margaret A. Johnson,et al.  Role of mitochondrial DNA mutations in human aging: Implications for the central nervous system and muscle , 1998, Annals of neurology.

[53]  E. Bonilla,et al.  Cytochemistry and immunocytochemistry of mitochondria in tissue sections. , 1996, Methods in enzymology.

[54]  D. A. Clayton,et al.  Low levels of mitochondrial transcription factor A in mitochondrial DNA depletion. , 1994, Biochemical and biophysical research communications.

[55]  S. Dimauro,et al.  Distribution of wild-type and common deletion forms of mtDNA in normal and respiration-deficient muscle fibers from patients with mitochondrial myopathy. , 1994, Human molecular genetics.

[56]  A. Tjønneland,et al.  Oxidative DNA damage estimated by 8-hydroxydeoxyguanosine excretion in humans: influence of smoking, gender and body mass index. , 1992, Carcinogenesis.

[57]  B. Ames,et al.  Oxidative damage to DNA during aging: 8-hydroxy-2'-deoxyguanosine in rat organ DNA and urine. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[58]  S. Dimauro,et al.  Detection of "deleted" mitochondrial genomes in cytochrome-c oxidase-deficient muscle fibers of a patient with Kearns-Sayre syndrome. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[59]  M. Fairbarn,et al.  Prediction of maximal oxygen uptake and power during cycle ergometry in subjects older than 55 years of age. , 1989, The American review of respiratory disease.

[60]  M. Dizdaroglu,et al.  Characterization of free radical-induced base damage in DNA at biologically relevant levels. , 1986, Analytical biochemistry.

[61]  S. Nishimura,et al.  Hydroxylation of deoxyguanosine at the C-8 position by ascorbic acid and other reducing agents. , 1984, Nucleic acids research.

[62]  J. Henriksson,et al.  Time course of changes in human skeletal muscle succinate dehydrogenase and cytochrome oxidase activities and maximal oxygen uptake with physical activity and inactivity. , 1977, Acta physiologica Scandinavica.