Neuroleptics cause stimulation of dopamine D1 receptors and their desensitization after chronic treatment.

[1]  G. Gessa,et al.  Does dopamine exert a tonic inhibitory control on the release of striatal acetylcholine in vivo? , 1994, European journal of pharmacology.

[2]  G. Gessa,et al.  Evidence that neuroleptics increase striatal acetylcholine release through stimulation of dopamine D1 receptors. , 1993, The Journal of pharmacology and experimental therapeutics.

[3]  L. Hansen,et al.  Effect of chronic treatment with NNC 756, a new D-1 receptor antagonist, or raclopride, a D-2 receptor antagonist, in drug-naive Cebus monkeys: dystonia, dyskinesia and D-1/D-2 supersensitivity , 1993, Journal of psychopharmacology.

[4]  B. Bunney,et al.  Depolarization inactivation of dopamine neurons: Terminal release characteristics , 1993, Synapse.

[5]  G. Lahoste,et al.  Dopamine supersensitivity and D1/D2 synergism are unrelated to changes in striatal receptor density , 1992, Synapse.

[6]  A. Imperato,et al.  Repeated stressful experiences differently affect limbic dopamine release during and following stress , 1992, Brain Research.

[7]  H. Groenewegen,et al.  Regulation of the activity of striatal cholinergic neurons by dopamine , 1992, Neuroscience.

[8]  H. Fibiger,et al.  Dopaminergic regulation of striatal acetylcholine release: importance of D1 and N-methyl-D-aspartate receptors. , 1991, The Journal of pharmacology and experimental therapeutics.

[9]  H. Fibiger,et al.  Dopamine D1 receptor stimulation increases striatal acetylcholine release in the rat. , 1990, European journal of pharmacology.

[10]  T. Chase,et al.  D1 dopamine receptor activation required for postsynaptic expression of D2 agonist effects. , 1987, Science.

[11]  T. Chase,et al.  Obligatory D-1/D-2 receptor interaction in the generation of dopamine agonist related behaviors. , 1986, European journal of pharmacology.

[12]  K. Neve,et al.  The effects of denervation and chronic haloperidol treatment on neostriatal dopamine receptor density are not additive in the rat , 1984, Neuroscience Letters.

[13]  G. Di Chiara,et al.  Trans-striatal dialysis coupled to reverse phase high performance liquid chromatography with electrochemical detection: a new method for the study of the in vivo release of endogenous dopamine and metabolites , 1984, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[14]  S. Snyder,et al.  Antischizophrenic drugs: chronic treatment elevates dopamine receptor binding in brain. , 1977, Science.

[15]  R. Roth,et al.  Dopaminergic neurons: similar biochemical and histochemical effects of gamma-hydroxybutyrate and acute lesions of the nigro-neostriatal pathway. , 1973, The Journal of pharmacology and experimental therapeutics.

[16]  R. Roth,et al.  Dopaminergic neurons: effect of antipsychotic drugs and amphetamine on single cell activity. , 1973, The Journal of pharmacology and experimental therapeutics.

[17]  B. Westerink,et al.  A microdialysis and automated on-line analysis approach to study central cholinergic transmission in vivo , 1991 .

[18]  G. Di Chiara,et al.  Dopamine release and metabolism in awake rats after systemic neuroleptics as studied by trans-striatal dialysis. , 1985, The Journal of neuroscience : the official journal of the Society for Neuroscience.