Detection of KIR3DS1 on the Cell Surface of Peripheral Blood NK Cells Facilitates Identification of a Novel Null Allele and Assessment of KIR3DS1 Expression during HIV-1 Infection12
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M. Altfeld | M. Carrington | J. Metcalf | M. Baseler | J. Adelsberger | G. Alter | S. Anderson | Maureen P. Martin | D. McVicar | V. Pascal | Eriko Yamada
[1] M. Carrington,et al. Lack of KIR3DS1 binding to MHC class I Bw4 tetramers in complex with CD8+ T cell epitopes. , 2007, AIDS research and human retroviruses.
[2] Des C. Jones,et al. Allelic expression patterns of KIR3DS1 and 3DL1 using the Z27 and DX9 antibodies , 2007, European journal of immunology.
[3] G. Davies,et al. Identification of bidirectional promoters in the human KIR genes , 2007, Genes and Immunity.
[4] D. Nixon,et al. Cutting Edge: KIR3DS1, a Gene Implicated in Resistance to Progression to AIDS, Encodes a DAP12-Associated Receptor Expressed on NK Cells That Triggers NK Cell Activation1 , 2007, The Journal of Immunology.
[5] P. Parham,et al. Functional Polymorphism of the KIR3DL1/S1 Receptor on Human NK Cells1 , 2007, The Journal of Immunology.
[6] M. Carrington,et al. Cutting Edge: Allele-Specific and Peptide-Dependent Interactions between KIR3DL1 and HLA-A and HLA-B12 , 2007, The Journal of Immunology.
[7] P. Parham. Taking license with natural killer cell maturation and repertoire development , 2006, Immunological reviews.
[8] S. Anderson,et al. Regulation of class I major histocompatibility complex receptor expression in natural killer cells: one promoter is not enough! , 2006, Immunological reviews.
[9] E. Thiel,et al. The anti-lymphoma effect of antibody-mediated immunotherapy is based on an increased degranulation of peripheral blood natural killer (NK) cells. , 2006, Experimental hematology.
[10] E. Kistner,et al. Reprogramming of CTLs into natural killer–like cells in celiac disease , 2006, The Journal of experimental medicine.
[11] J. Lubkowski,et al. The Structure of the Extracellular Domain of Triggering Receptor Expressed on Myeloid Cells Like Transcript-1 and Evidence for a Naturally Occurring Soluble Fragment* , 2006, Journal of Biological Chemistry.
[12] P. Parham,et al. Roles for HLA and KIR polymorphisms in natural killer cell repertoire selection and modulation of effector function , 2006, The Journal of experimental medicine.
[13] M. Carrington,et al. The impact of variation at the KIR gene cluster on human disease. , 2006, Current topics in microbiology and immunology.
[14] A. Fauci,et al. NK cells in HIV infection: paradigm for protection or targets for ambush , 2005, Nature Reviews Immunology.
[15] Hendrik Streeck,et al. Sequential deregulation of NK cell subset distribution and function starting in acute HIV-1 infection. , 2005, Blood.
[16] Eric Vivier,et al. Recognition of peptide-MHC class I complexes by activating killer immunoglobulin-like receptors. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[17] A. Poggi,et al. Regulation of γδ T cell survival by soluble HLA‐I: Involvement of CD8 and activating killer Ig‐like receptors , 2005 .
[18] L. Rodrigo,et al. Protective effect of the HLA-Bw4I80 epitope and the killer cell immunoglobulin-like receptor 3DS1 gene against the development of hepatocellular carcinoma in patients with hepatitis C virus infection. , 2005, Journal of Infectious Diseases.
[19] R. Solana,et al. CD8 T cells expressing NK associated receptors are increased in melanoma patients and display an effector phenotype , 2005, Cancer Immunology, Immunotherapy.
[20] Andrew Glass,et al. Hierarchy of resistance to cervical neoplasia mediated by combinations of killer immunoglobulin-like receptor and human leukocyte antigen loci , 2005 .
[21] J. Dillner,et al. A population-based cohort study of KIR genes and genotypes in relation to cervical intraepithelial neoplasia. , 2005, Tissue antigens.
[22] A. Fauci,et al. Characterization of CD56-/CD16+ natural killer (NK) cells: a highly dysfunctional NK subset expanded in HIV-infected viremic individuals. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[23] Lewis L Lanier,et al. NK cell recognition. , 2005, Annual review of immunology.
[24] Jessica M Malenfant,et al. Increased Natural Killer Cell Activity in Viremic HIV-1 Infection1 , 2004, The Journal of Immunology.
[25] P. Leibson,et al. Stimulatory Killer Ig-Like Receptors Modulate T Cell Activation through DAP12-Dependent and DAP12-Independent Mechanisms1 , 2004, The Journal of Immunology.
[26] Salim I. Khakoo,et al. HLA and NK Cell Inhibitory Receptor Genes in Resolving Hepatitis C Virus Infection , 2004, Science.
[27] S. Lockett,et al. Identification of probabilistic transcriptional switches in the Ly49 gene cluster: a eukaryotic mechanism for selective gene activation. , 2004, Immunity.
[28] T. Chun,et al. Expression of chemokine and inhibitory receptors on natural killer cells: effect of immune activation and HIV viremia. , 2004, The Journal of infectious diseases.
[29] R. Solana,et al. Selective Depletion of CD56dim NK Cell Subsets and Maintenance of CD56bright NK Cells in Treatment-Naive HIV-1-Seropositive Individuals , 2002, Journal of Clinical Immunology.
[30] P. Parham,et al. The Protein Made from a Common Allele of KIR3DL1 (3DL1*004) Is Poorly Expressed at Cell Surfaces due to Substitution at Positions 86 in Ig Domain 0 and 182 in Ig Domain 1 1 , 2003, The Journal of Immunology.
[31] M. Daucher,et al. Natural killer cells in HIV-1 infection: Dichotomous effects of viremia on inhibitory and activating receptors and their functional correlates , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[32] K. Hsu,et al. The killer cell immunoglobulin‐like receptor (KIR) genomic region: gene‐order, haplotypes and allelic polymorphism , 2002, Immunological reviews.
[33] Keith Hoots,et al. Epistatic interaction between KIR3DS1 and HLA-B delays the progression to AIDS , 2002, Nature Genetics.
[34] Ashok Kumar,et al. Normalization of natural killer cell function and phenotype with effective anti-HIV therapy and the role of IL-10 , 2002, AIDS.
[35] S. Topino,et al. Distribution of the natural killer-related receptor for HLA-C during highly active antiretroviral therapy for human immunodeficiency virus infection. , 2001, Human immunology.
[36] R. Morisset,et al. Modulation of expression of the MHC class I‐binding natural killer cell receptors, and NK activity in relation to viral load in HIV‐infected/AIDS patients , 2001, Journal of medical virology.
[37] Michael J. Wilson,et al. The genomic context of natural killer receptor extended gene families , 2001, Immunological reviews.
[38] D. Burshtyn,et al. Intracellular Signaling by the Killer Immunoglobulin-Like Receptors and Ly49 , 2001, Science's STKE.
[39] William H. Carr,et al. Different NK Cell Surface Phenotypes Defined by the DX9 Antibody Are Due to KIR3DL1 Gene Polymorphism1 , 2001, The Journal of Immunology.
[40] M. Bonneville,et al. Regulation of Inhibitory and Activating Killer-Cell Ig-Like Receptor Expression Occurs in T Cells After Termination of TCR Rearrangements1 , 2001, The Journal of Immunology.
[41] R. Biassoni,et al. Activating receptors and coreceptors involved in human natural killer cell-mediated cytolysis. , 2001, Annual review of immunology.
[42] J. Yen,et al. Killer Cell Activating Receptors Function as Costimulatory Molecules on CD4+CD28null T Cells Clonally Expanded in Rheumatoid Arthritis1 , 2000, The Journal of Immunology.
[43] S. Beck,et al. Plasticity in the organization and sequences of human KIR/ILT gene families. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[44] Eric O Long,et al. A Human Histocompatibility Leukocyte Antigen (HLA)-G–specific Receptor Expressed on All Natural Killer Cells , 1999, The Journal of experimental medicine.
[45] É. Vivier,et al. Differential regulation of killer cell Ig‐like receptors and CD94 lectin‐like dimers on NK and T lymphocytes from HIV‐1‐infected individuals , 1999, European journal of immunology.
[46] Eric O Long,et al. Regulation of immune responses through inhibitory receptors. , 1999, Annual review of immunology.
[47] J V Giorgi,et al. Natural killer cell immunodeficiency in HIV disease is manifest by profoundly decreased numbers of CD16+CD56+ cells and expansion of a population of CD16dimCD56- cells with low lytic activity. , 1995, Journal of acquired immune deficiency syndromes and human retrovirology : official publication of the International Retrovirology Association.
[48] A. Landay,et al. Evidence of a selective depletion of a CD16+ CD56+ CD8+ natural killer cell subset during HIV infection. , 1995, Cytometry.
[49] P. Parham,et al. The Bw4 public epitope of HLA-B molecules confers reactivity with natural killer cell clones that express NKB1, a putative HLA receptor , 1995, The Journal of experimental medicine.
[50] G. Ferrara,et al. NK3-specific natural killer cells are selectively inhibited by Bw4- positive HLA alleles with isoleucine 80 , 1994, The Journal of experimental medicine.
[51] Arthur Weiss,et al. The cytoplasmic domain of the T cell receptor ζ chain is sufficient to couple to receptor-associated signal transduction pathways , 1991, Cell.
[52] H. Ljunggren,et al. In search of the 'missing self': MHC molecules and NK cell recognition. , 1990, Immunology today.