Intrahepatic STAT-3 activation and acute phase gene expression predict outcome after CLP sepsis in the rat.
暂无分享,去创建一个
[1] C. Deutschman,et al. Sepsis-induced depression of rat glucose-6-phosphatase gene expression and activity. , 1997, American journal of physiology. Regulatory, integrative and comparative physiology.
[2] E. Shiloni,et al. Interleukin-6 secretion in mice is associated with reduced glucose-6-phosphatase and liver glycogen levels. , 1997, The American journal of physiology.
[3] R. Piekorz,et al. Members of the family of IL-6-type cytokines activate Stat5a in various cell types. , 1997, Biochemical and biophysical research communications.
[4] J. Darnell,et al. The state of the STATs: recent developments in the study of signal transduction to the nucleus. , 1997, Current opinion in cell biology.
[5] C. Deutschman,et al. ALTERED HEPATIC GENE EXPRESSION IN FECAL PERITONITIS: CHANGES IN TRANSCRIPTION OF GLUCONEOGENIC, β‐OXIDATIVE, AND UREAGENIC GENES , 1997, Shock.
[6] J. Peschon,et al. Initiation of liver growth by tumor necrosis factor: deficient liver regeneration in mice lacking type I tumor necrosis factor receptor. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[7] C. Deutschman,et al. Acute-phase gene expression correlates with intrahepatic tumor necrosis factor-alpha abundance but not with plasma tumor necrosis factor concentrations during sepsis/systemic inflammatory response syndrome in the rat. , 1996, Critical care medicine.
[8] E. Furth,et al. Liver Failure and Defective Hepatocyte Regeneration in Interleukin-6-Deficient Mice , 1996, Science.
[9] David Baltimore,et al. An Essential Role for NF-κB in Preventing TNF-α-Induced Cell Death , 1996, Science.
[10] Marty W. Mayo,et al. TNF- and Cancer Therapy-Induced Apoptosis: Potentiation by Inhibition of NF-κB , 1996, Science.
[11] A. Sauaia,et al. The inflammatory profile of interleukin-6, interleukin-8, and soluble intercellular adhesion molecule-1 in postinjury multiple organ failure , 1996 .
[12] T. Standiford,et al. Balance of inflammatory cytokines related to severity and mortality of murine sepsis , 1996, Infection and immunity.
[13] C. Deutschman,et al. Increased expression of cytokine-induced neutrophil chemoattractant in septic rat liver , 1996 .
[14] J. Christman,et al. Sepsis and cytokines: current status. , 1996, British journal of anaesthesia.
[15] Jens Schneider-Mergener,et al. Differential Activation of Acute Phase Response Factor/STAT3 and STAT1 via the Cytoplasmic Domain of the Interleukin 6 Signal Transducer gp130 , 1996, The Journal of Biological Chemistry.
[16] I. Chaudry,et al. Mechanism of hepatocellular dysfunction during hyperdynamic sepsis. , 1996, The American journal of physiology.
[17] G. Regel,et al. Pattern of Organ Failure following Severe Trauma , 1996, World Journal of Surgery.
[18] Dongxiao Zhang,et al. STAT3 Participates in Transcriptional Activation of the C-reactive Protein Gene by Interleukin-6 (*) , 1996, The Journal of Biological Chemistry.
[19] T. Yen,et al. Perioperative plasma concentrations of tumor necrosis factor-alpha and interleukin-6 in infected patients. , 1996, Critical care medicine.
[20] C. Deutschman,et al. Increased expression of cytokine-induced neutrophil chemoattractant in septic rat liver. , 1995, The American journal of physiology.
[21] J. Darnell,et al. Maximal activation of transcription by statl and stat3 requires both tyrosine and serine phosphorylation , 1995, Cell.
[22] J. Sjölin,et al. Plasma levels of cytokines in primary septic shock in humans: correlation with disease severity. , 1995, The Journal of infectious diseases.
[23] J. Christman,et al. Strategies for blocking the systemic effects of cytokines in the sepsis syndrome. , 1995, Critical care medicine.
[24] P. Heinrich,et al. Inhibition by recombinant human interleukin‐6 of the glucagon‐dependent induction of phosphoenolpyruvate carboxykinase and of the insulin‐dependent induction of glucokinase gene expression in cultured rat hepatocytes: Regulation of gene transcription and messenger RNA degradation , 1994, Hepatology.
[25] M. Brennan,et al. Systemic and liver cytokine activation. Implications for liver regeneration and posthepatectomy endotoxemia and sepsis. , 1994, Archives of surgery.
[26] J. Gauldie,et al. The acute phase response. , 1994, Immunology today.
[27] F. Cerra,et al. Multiple organ failure syndrome in the 1990s. Systemic inflammatory response and organ dysfunction. , 1994, JAMA.
[28] R. Bone,et al. Plasma Cytokine and Endotoxin Levels Correlate with Survival in Patients with the Sepsis Syndrome , 1993, Annals of Internal Medicine.
[29] E. Deitch,et al. Multiple organ failure. Pathophysiology and potential future therapy. , 1992, Annals of surgery.
[30] P Franchimont,et al. Cytokine serum level during severe sepsis in human IL-6 as a marker of severity. , 1992, Annals of surgery.
[31] B. Hazenberg,et al. Patterns of cytokines, plasma endotoxin, plasminogen activator inhibitor, and acute‐phase proteins during the treatment of severe sepsis in humans , 1992, Critical care medicine.
[32] I. Chaudry,et al. Evaluation of factors affecting mortality rate after sepsis in a murine cecal ligation and puncture model. , 1983, Surgery.
[33] I. Chaudry,et al. Sepsis and septic shock--a review of laboratory models and a proposal. , 1980, The Journal of surgical research.
[34] M. Pinsky. Clinical studies on cytokines in sepsis: role of serum cytokines in the development of multiple-systems organ failure. , 1994, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.