Herpes simplex virus latency analyzed by in situ hybridization.
暂无分享,去创建一个
[1] W. Flanagan,et al. The herpes simplex virus latency-associated transcript is spliced during the latent phase of infection , 1988, Journal of virology.
[2] R. Javier,et al. A herpes simplex virus transcript abundant in latently infected neurons is dispensable for establishment of the latent state. , 1988, Virology.
[3] E. Wagner,et al. Prominence of the herpes simplex virus latency-associated transcript in trigeminal ganglia from seropositive humans. , 1988, The Journal of infectious diseases.
[4] E. Romanowski,et al. RNA complementary to herpes simplex virus type 1 ICP0 gene demonstrated in neurons of human trigeminal ganglia , 1988, Journal of virology.
[5] W. Flanagan,et al. Physical characterization of the herpes simplex virus latency-associated transcript in neurons , 1988, Journal of virology.
[6] E. Lavi,et al. Latent herpes simplex virus type 1 transcripts in peripheral and central nervous system tissues of mice map to similar regions of the viral genome , 1988, Journal of virology.
[7] J. Smialek,et al. Latent herpes simplex virus in human trigeminal ganglia. Detection of an immediate early gene "anti-sense" transcript by in situ hybridization. , 1987, The New England journal of medicine.
[8] A. Nesburn,et al. Detection of latency-related viral RNAs in trigeminal ganglia of rabbits latently infected with herpes simplex virus type 1 , 1987, Journal of virology.
[9] N. Fraser,et al. Detection of herpes simplex virus type 1 transcripts during latent infection in mice , 1987, Journal of virology.
[10] E. Wagner,et al. RNA complementary to a herpesvirus alpha gene mRNA is prominent in latently infected neurons. , 1987, Science.
[11] D. Rock,et al. Localization of herpes simplex virus in the trigeminal and olfactory systems of the mouse central nervous system during acute and latent infections by in situ hybridization. , 1984, Laboratory investigation; a journal of technical methods and pathology.
[12] A. Haase,et al. Detection of Viral Nucleic Acids by in Situ Hybridization , 1984 .
[13] A. Haase,et al. Detection of hepatitis B virus DNA in hepatocytes, bile duct epithelium, and vascular elements by in situ hybridization. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[14] A. Haase,et al. Visna DNA synthesis and the tempo of infection in vitro. , 1982, Virology.
[15] R. Tenser,et al. Detection of herpes simplex virus mRNA in latently infected trigeminal ganglion neurons by in situ hybridization , 1982, Annals of neurology.
[16] D. Galloway,et al. Limited transcription of the herpes simplex virus genome when latent in human sensory ganglia , 1982, Journal of virology.
[17] G. Darby,et al. Herpes simplex virus latency: the cellular location of virus in dorsal root ganglia and the fate of the infected cell following virus activation. , 1980, The Journal of general virology.
[18] J. G. Stevens,et al. Latent characteristics of selected herpesviruses. , 1978, Advances in cancer research.
[19] J. G. Stevens,et al. Evidence that Neurons Harbor Latent Herpes Simplex Virus , 1974, Infection and immunity.
[20] J. G. Stevens,et al. LATENT HERPES SIMPLEX VIRUS IN THE CENTRAL NERVOUS SYSTEM OF RABBITS AND MICE , 1973, The Journal of experimental medicine.
[21] J. G. Stevens,et al. Latent Herpes Simplex Virus in Spinal Ganglia of Mice , 1971, Science.