Mechanism of calsequestrin regulation of single cardiac ryanodine receptor in normal and pathological conditions
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Michael Fill | P. Volpe | Pompeo Volpe | S. Furlan | Sandor Gyorke | Sandra Furlan | Giorgia Valle | Haiyan Chen | Alma Nani | M. Fill | G. Valle | S. Gyorke | Haiyan Chen | A. Nani
[1] Amy D. Hanna,et al. Proteins within the intracellular calcium store determine cardiac RyR channel activity and cardiac output , 2012, Clinical and experimental pharmacology & physiology.
[2] R. Reithmeier,et al. Characterization of cardiac calsequestrin. , 1987, Biochemistry.
[3] R. Hinch,et al. A mathematical analysis of the generation and termination of calcium sparks. , 2004, Biophysical journal.
[4] I. Györke,et al. Regulation of the cardiac ryanodine receptor channel by luminal Ca2+ involves luminal Ca2+ sensing sites. , 1998, Biophysical journal.
[5] S. Priori,et al. Luminal Ca2+ Regulation of Single Cardiac Ryanodine Receptors: Insights Provided by Calsequestrin and its Mutants , 2008, The Journal of general physiology.
[6] Eric A Sobie,et al. Recovery of cardiac calcium release is controlled by sarcoplasmic reticulum refilling and ryanodine receptor sensitivity. , 2011, Cardiovascular research.
[7] R. Sitsapesan,et al. Regulation of the gating of the sheep cardiac sarcoplasmic reticulum ca2+-release channel by luminal Ca2+ , 1994, The Journal of Membrane Biology.
[8] D. Roden,et al. Casq 2 deletion causes sarcoplasmic reticulum volume increase , premature Ca 2 + release , and catecholaminergic polymorphic ventricular tachycardia , 2006 .
[9] Remo Guidieri. Res , 1995, RES: Anthropology and Aesthetics.
[10] M. Fill,et al. Streaming potentials reveal a short ryanodine-sensitive selectivity filter in cardiac Ca2+ release channel. , 1994, Biophysical journal.
[11] Silvia G Priori,et al. Inherited dysfunction of sarcoplasmic reticulum Ca2+ handling and arrhythmogenesis. , 2011, Circulation research.
[12] Heping Cheng,et al. RyR2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca2+ release (SOICR). , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[13] M. Cannell,et al. Termination of calcium-induced calcium release by induction decay: an emergent property of stochastic channel gating and molecular scale architecture. , 2013, Journal of molecular and cellular cardiology.
[14] D. Laver. Ca2+ STORES REGULATE RYANODINE RECEPTOR Ca2+ RELEASE CHANNELS VIA LUMINAL AND CYTOSOLIC Ca2+ SITES , 2007, Clinical and experimental pharmacology & physiology.
[15] A. Zahradníková,et al. Luminal Ca2+ controls activation of the cardiac ryanodine receptor by ATP , 2012, The Journal of general physiology.
[16] Donald M Bers,et al. How does stochastic ryanodine receptor-mediated Ca leak fail to initiate a Ca spark? , 2011, Biophysical journal.
[17] Michael Fill,et al. Ryanodine receptor calcium release channels. , 2002, Physiological reviews.
[18] José Jalife,et al. Arrhythmogenic Mechanisms in a Mouse Model of Catecholaminergic Polymorphic Ventricular Tachycardia , 2007, Circulation research.
[19] I. Györke,et al. Regulation of calcium release by calcium inside the sarcoplasmic reticulum in ventricular myocytes , 1996, Pflügers Archiv - European Journal of Physiology.
[20] J. H. Collins,et al. Complete amino acid sequence of canine cardiac calsequestrin deduced by cDNA cloning. , 1988, The Journal of biological chemistry.
[21] Sandor Györke,et al. The role of calsequestrin, triadin, and junctin in conferring cardiac ryanodine receptor responsiveness to luminal calcium. , 2004, Biophysical journal.
[22] D. Laver. Ca 2 + stores regulate ryanodine receptor Ca 2 + release channels via luminal and cytosolic Ca 2 + sites , 2007 .
[23] L. Jones,et al. Ca2+ binding effects on protein conformation and protein interactions of canine cardiac calsequestrin. , 1988, The Journal of biological chemistry.
[24] D. Gillespie,et al. Ryanodine Receptor Current Amplitude Controls Ca2+ Sparks in Cardiac Muscle , 2012, Circulation research.
[25] D. Bers. Macromolecular complexes regulating cardiac ryanodine receptor function. , 2004, Journal of molecular and cellular cardiology.
[26] M. Periasamy,et al. Functional interaction between calsequestrin and ryanodine receptor in the heart , 2012, Cellular and Molecular Life Sciences.
[27] S. Fleischer,et al. Isolation of canine cardiac sarcoplasmic reticulum. , 1988, Methods in enzymology.
[28] O. H. Lowry,et al. Protein measurement with the Folin phenol reagent. , 1951, The Journal of biological chemistry.
[29] Eric A. Sobie,et al. Dynamics of calcium sparks and calcium leak in the heart. , 2011, Biophysical journal.
[30] S. Fleischer,et al. [9] Isolation of canine cardiac sarcoplasmic reticulum , 1988 .
[31] C. Reggiani,et al. Reorganized stores and impaired calcium handling in skeletal muscle of mice lacking calsequestrin‐1 , 2007, The Journal of physiology.
[32] S. Priori,et al. Clinical Phenotype and Functional Characterization of CASQ2 Mutations Associated With Catecholaminergic Polymorphic Ventricular Tachycardia , 2006, Circulation.
[33] M. Varsányi,et al. Regulation of ryanodine receptors by calsequestrin: effect of high luminal Ca2+ and phosphorylation. , 2005, Biophysical journal.
[34] D. Maclennan,et al. Isolation of a calcium-sequestering protein from sarcoplasmic reticulum. , 1971, Proceedings of the National Academy of Sciences of the United States of America.
[35] T. Shannon,et al. Flux regulation of cardiac ryanodine receptor channels , 2010, The Journal of general physiology.
[36] D. Roden,et al. Flecainide prevents catecholaminergic polymorphic ventricular tachycardia in mice and humans , 2009, Nature Medicine.
[37] D. Roden,et al. Casq2 deletion causes sarcoplasmic reticulum volume increase, premature Ca2+ release, and catecholaminergic polymorphic ventricular tachycardia. , 2006, The Journal of clinical investigation.
[38] A. Zima,et al. Single ryanodine receptor channel basis of caffeine's action on Ca2+ sparks. , 2011, Biophysical journal.
[39] D. Lancet,et al. A missense mutation in a highly conserved region of CASQ2 is associated with autosomal recessive catecholamine-induced polymorphic ventricular tachycardia in Bedouin families from Israel. , 2001, American journal of human genetics.
[40] J J Rice,et al. Modeling gain and gradedness of Ca2+ release in the functional unit of the cardiac diadic space. , 1999, Biophysical journal.
[41] G. G. Stokes. "J." , 1890, The New Yale Book of Quotations.
[42] D. Terentyev,et al. Protein–protein interactions between triadin and calsequestrin are involved in modulation of sarcoplasmic reticulum calcium release in cardiac myocytes , 2007, The Journal of physiology.
[43] K. Otsu,et al. Regulation of sarcoplasmic reticulum gene expression during cardiac and skeletal muscle development. , 1992, The American journal of physiology.
[44] A. Dunker,et al. Polymerization of Calsequestrin , 2003, The Journal of Biological Chemistry.
[45] D. Lancet,et al. A missense mutation in a highly conserved region of CASQ2 is associated with autosomal recessive catecholamine-induced polymorphic ventricular tachycardia in Bedouin families from Israel. , 2001, Cold Spring Harbor symposia on quantitative biology.
[46] D. Jiang,et al. Loss of luminal Ca2+ activation in the cardiac ryanodine receptor is associated with ventricular fibrillation and sudden death , 2007, Proceedings of the National Academy of Sciences.
[47] Heping Cheng,et al. RyR 2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca 2 release ( SOICR ) , 2004 .
[48] Pernicious attrition and inter-RyR2 CICR current control in cardiac muscle. , 2013, Journal of molecular and cellular cardiology.
[49] S. Priori,et al. Viral Gene Transfer Rescues Arrhythmogenic Phenotype and Ultrastructural Abnormalities in Adult Calsequestrin-Null Mice With Inherited Arrhythmias , 2012, Circulation research.
[50] P. Volpe,et al. Ryanodine receptor luminal Ca2+ regulation: swapping calsequestrin and channel isoforms. , 2009, Biophysical journal.
[51] S. Priori,et al. Unexpected Structural and Functional Consequences of the R33Q Homozygous Mutation in Cardiac Calsequestrin: A Complex Arrhythmogenic Cascade in a Knock In Mouse Model , 2008, Circulation research.
[52] D. Clegg,et al. Identification and developmental expression of a chicken calsequestrin homolog. , 1990, Developmental biology.
[53] David W Piston,et al. Flecainide inhibits arrhythmogenic Ca2+ waves by open state block of ryanodine receptor Ca2+ release channels and reduction of Ca2+ spark mass. , 2010, Journal of molecular and cellular cardiology.
[54] H. Duff,et al. Carvedilol and its new analogs suppress arrhythmogenic store overload–induced Ca2+ release , 2011, Nature Medicine.
[55] R. Reithmeier,et al. Amino acid sequence of rabbit fast-twitch skeletal muscle calsequestrin deduced from cDNA and peptide sequencing. , 1987, Proceedings of the National Academy of Sciences of the United States of America.