Dual Role of the Adaptive Immune System in Liver Injury and Hepatocellular Carcinoma Development.
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M. Manns | R. Geffers | F. Reisinger | K. Unger | D. Haller | A. Vogel | T. Longerich | T. Clavel | M. Finegold | A. Saborowski | M. E. Healy | F. Limbourg | A. Weber | M. Heikenwälder | L. E. Buitrago-Molina | J. Endig | S. Marhenke | A. Misslitz | A. Michael | C. Könecke | J. Schütt | A. Schreder | M. Healy | L. E. Buitrago‐Molina
[1] Ji Luo,et al. NAFLD causes selective CD4+ T lymphocyte loss and promotes hepatocarcinogenesis , 2016, Nature.
[2] R. Moreau,et al. Acute-on-chronic liver failure: A new syndrome that will re-classify cirrhosis. , 2015, Journal of hepatology.
[3] B. Engelward,et al. Inflammation-Induced Cell Proliferation Potentiates DNA Damage-Induced Mutations In Vivo , 2015, PLoS genetics.
[4] M. Grompe,et al. Bipotential adult liver progenitors are derived from chronically injured mature hepatocytes. , 2014, Cell stem cell.
[5] H. Moch,et al. Metabolic activation of intrahepatic CD8+ T cells and NKT cells causes nonalcoholic steatohepatitis and liver cancer via cross-talk with hepatocytes. , 2014, Cancer cell.
[6] H. Mandel,et al. Cross-sectional study of 168 patients with hepatorenal tyrosinaemia and implications for clinical practice , 2014, Orphanet Journal of Rare Diseases.
[7] M. Manns,et al. Incidence and long‐term risk of de novo malignancies after liver transplantation with implications for prevention and detection , 2013, Liver transplantation : official publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society.
[8] Kristen Jepsen,et al. Identification of Liver Cancer Progenitors Whose Malignant Progression Depends on Autocrine IL-6 Signaling , 2013, Cell.
[9] M. Manns,et al. The degree of liver injury determines the role of p21 in liver regeneration and hepatocarcinogenesis in mice , 2013, Hepatology.
[10] B. Rehermann. Pathogenesis of chronic viral hepatitis: differential roles of T cells and NK cells , 2013, Nature Medicine.
[11] E. van de Steeg,et al. Complete OATP1B1 and OATP1B3 deficiency causes human Rotor syndrome by interrupting conjugated bilirubin reuptake into the liver. , 2012, The Journal of clinical investigation.
[12] T. Luedde,et al. Adaptive immunity suppresses formation and progression of diethylnitrosamine-induced liver cancer , 2012, Gut.
[13] T. Luedde,et al. Senescence surveillance of pre-malignant hepatocytes limits liver cancer development , 2011, Nature.
[14] J. Schug,et al. Prospective isolation of a bipotential clonogenic liver progenitor cell in adult mice. , 2011, Genes & development.
[15] D. Adams,et al. Mechanisms of immune-mediated liver injury. , 2010, Toxicological Sciences.
[16] M. Kurrer,et al. A lymphotoxin-driven pathway to hepatocellular carcinoma. , 2009, Cancer cell.
[17] M. Manns,et al. Rapamycin delays tumor development in murine livers by inhibiting proliferation of hepatocytes with DNA damage , 2009, Hepatology.
[18] S. Nedospasov,et al. T cell-derived lymphotoxin regulates liver regeneration. , 2009, Gastroenterology.
[19] M. Sporn,et al. Activation of nuclear factor E2‐related factor 2 in hereditary tyrosinemia type 1 and its role in survival and tumor development , 2008, Hepatology.
[20] Amar Deep Sharma,et al. Loss of p21 permits carcinogenesis from chronically damaged liver and kidney epithelial cells despite unchecked apoptosis. , 2008, Cancer cell.
[21] J. Marrero,et al. Diagnosis and treatment of hepatocellular carcinoma. , 2008, Gastroenterology.
[22] Wessel N. van Wieringen,et al. CGHcall: Calling aberrations for array CGH tumor profiles. , 2008 .
[23] Wessel N. van Wieringen,et al. CGHregions: Dimension Reduction for Array CGH Data with Minimal Information Loss , 2007 .
[24] M. A. van de Wiel,et al. CGHregions: Dimension Reduction for Array CGH Data with Minimal Information Loss , 2007, Cancer informatics.
[25] H. Blum,et al. Pathogenesis of hepatocellular carcinoma. , 2005, European journal of gastroenterology & hepatology.
[26] Y. Ben-Neriah,et al. NF-κB functions as a tumour promoter in inflammation-associated cancer , 2004, Nature.
[27] M. Grompe,et al. Myelomonocytic cells are sufficient for therapeutic cell fusion in liver , 2004, Nature Medicine.
[28] P. Charneau,et al. Bipotential mouse embryonic liver stem cell lines contribute to liver regeneration and differentiate as bile ducts and hepatocytes. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[29] M. Iordanov,et al. Chronic liver disease in murine hereditary tyrosinemia type 1 induces resistance to cell death , 2004, Hepatology.
[30] L. French,et al. Visualization of Lymphotoxin-β and Lymphotoxin-β Receptor Expression in Mouse Embryos1 , 2002, The Journal of Immunology.
[31] R. Tanguay,et al. Fumarylacetoacetate, the metabolite accumulating in hereditary tyrosinemia, activates the ERK pathway and induces mitotic abnormalities and genomic instability. , 2001, Human molecular genetics.
[32] F. Chisari,et al. Immune Pathogenesis of Hepatocellular Carcinoma , 1998, The Journal of experimental medicine.
[33] S. Lindstedt,et al. Pharmacological correction of neonatal lethal hepatic dysfunction in a murine model of hereditary tyrosinaemia type I , 1995, Nature Genetics.
[34] Philippe Soriano,et al. Loss of fumarylacetoacetate hydrolase is responsible for the neonatal hepatic dysfunction phenotype of lethal albino mice. , 1993, Genes & development.
[35] Rinat Abramovitch,et al. NF-kappaB functions as a tumour promoter in inflammation-associated cancer. , 2004, Nature.
[36] L. French,et al. Visualization of lymphotoxin-beta and lymphotoxin-beta receptor expression in mouse embryos. , 2002, Journal of immunology.