Acute blood pressure elevation and ventricular fibrillation threshold during coronary occlusion and reperfusion in the dog.

[1]  R. Verrier,et al.  Differing mechanisms for ventricular vulnerability during coronary artery occlusion and release. , 1976, American heart journal.

[2]  B Lown,et al.  Neural activity and ventricular fibrillation. , 1976, The New England journal of medicine.

[3]  R. Verrier,et al.  Muscarinic Effects of Vagosympathetic Trunk Stimulation on the Repetitive Extrasystole (RE) Threshold , 1976, Circulation.

[4]  R. Verrier,et al.  Vulnerability to ventricular fibrillation during acute coronary arterial occlusion and release. , 1975, The American journal of cardiology.

[5]  J. Tamargo,et al.  Interaction of Sequential Stimuli Applied during the Relative Refractory Period in Relation to Determination of Fibrillation Threshold in the Canine Ventricle , 1975, Circulation research.

[6]  R. Verrier,et al.  The Effect of Vagus Nerve Stimulation upon Vulnerability of the Canine Ventricle: Role of Sympathetic‐Parasympathetic Interactions , 1975, Circulation.

[7]  J. .. Abildskov,et al.  Influence of sympathetic tone on ventricular fibrillation threshold during experimental coronary occlusion. , 1975, The American journal of cardiology.

[8]  K. Engelman,et al.  Vagally mediated suppression of premature ventricular contractions in man. , 1975, American heart journal.

[9]  J. Spear,et al.  Ventricular fibrillation threshold; its physiological and pharmacological importance. , 1975, Archives of internal medicine.

[10]  D. Jacobowitz,et al.  Cholinergic Innervation of the Canine and Human Ventricular Conducting System: Anatomic and Electrophysiologic Correlations , 1974, Circulation.

[11]  E. Downar,et al.  Phenylephrine (Neo‐synephrine) Terminated Ventricular Tachycardia , 1974, Circulation.

[12]  H. Levine,et al.  Distinctive time course of ventricular vulnerability to fibrillation during and after release of coronary ligation. , 1974, The American journal of cardiology.

[13]  R. Verrier,et al.  Effect of acute blood pressure elevation on the ventricular fibrillation threshold. , 1974, The American journal of physiology.

[14]  K. Kent,et al.  Electrical Stability of Acutely Ischemic Myocardium: Influences of Heart Rate and Vagal Stimulation , 1973, Circulation.

[15]  R. M. Conant,et al.  Autonomic Disturbance at Onset of Acute Myocardial Infarction , 1972, British medical journal.

[16]  R. Verrier,et al.  Protective zone for ventricular fibrillation , 1972 .

[17]  J. Staszewska-Barczak,et al.  The reflex stimulation of catecholamine secretion during the acute stage of myocardial infarction in the dog. , 1971, Clinical science.

[18]  R. Gillis,et al.  Role of the nervous system in the arrhythmias produced by coronary occlusion in the cat. , 1971, American heart journal.

[19]  J. Covell,et al.  Factors Influencing Infarct Size Following Experimental Coronary Artery Occlusions , 1971, Circulation.

[20]  J. Han,et al.  Ventricular vulnerability during acute coronary occlusion. , 1969, The American journal of cardiology.

[21]  A Malliani,et al.  A sympathetic reflex elicited by experimental coronary occlusion. , 1969, The American journal of physiology.

[22]  W. Daggett,et al.  Selective cardiac vagotomy. Surgical preparation and techniques of verification. , 1966, The Journal of thoracic and cardiovascular surgery.

[23]  A. S. Harris,et al.  Changes in infarction arrhythmias resulting from changes in arterial pressure. , 1966, The American journal of physiology.

[24]  L. Costantin,et al.  Extracardiac factors contributing to hypotension during coronary occlusion. , 1963, The American journal of cardiology.

[25]  R. Gorlin,et al.  A new therapeutic approach to acute coronary occlusion: II. Opening dormant coronary collateral channels by counterpulsation , 1963 .

[26]  M. Szentiványi,et al.  Localisation of the receptors of the coronary chemoreflex in the dog. , 1961, Archives internationales de pharmacodynamie et de therapie.

[27]  B. C. Abbott,et al.  Inotropic Effects of Trains of Impulses Applied during the Contraction of Cardiac Muscle , 1960, The Journal of general physiology.

[28]  E. Corday,et al.  Vasopressor therapy in the cardiac arrhythmias. , 1959, The New England journal of medicine.

[29]  J. Dudel,et al.  Das Aktionspotential und Mechanogramm des Herzmuskels unter dem Einfluß der Dehnung , 1954 .

[30]  G. Dawes,et al.  Respiratory and cardiovascular reflexes from the heart and lungs , 1951, The Journal of physiology.

[31]  G. Moe,et al.  The role of arterial pressure in the induction of idioventricular rhythms under cyclopropane anesthesia. , 1948, The Journal of pharmacology and experimental therapeutics.

[32]  G. Dawes.  Studies on veratrum alkaloids; receptor areas in the coronary arteries and elsewhere as revealed by the use of veratridine. , 1947, The Journal of pharmacology and experimental therapeutics.

[33]  J. Nonidez.  Distribution of the aortic nerve fibers and the epithelioid bodies (Supracardial ‘Paraganglia’) in the dog , 1937 .

[34]  P. Schwartz,et al.  Effects of unilateral cardiac sympathetic denervation on the ventricular fibrillation threshold. , 1976, The American journal of cardiology.

[35]  B. Lown,et al.  Changes in ventricular fibrillation threshold during coronary artery occlusion and release induced by beta adrenergic blockade , 1976 .

[36]  W. C. Randall,et al.  Experimental cardiac arrhythmias in the unbalanced heart , 1976 .

[37]  R. Bruce,et al.  Significance of “exertional hypotension” in men with ischemic heart disease , 1976 .

[38]  G. T. Smith,et al.  A new therapeutic approach to acute coronary occlusion. I. Production of standardized coronary occlusion with microspheres. , 1962, The American journal of cardiology.

[39]  A. V. Bezold.  Uber die physiologischen Wirkungen des essigsauren Veratrins , 1867 .