Combined effects of interleukin-1α and transforming growth factor-β1 on modulation of human cardiac fibroblast function.

[1]  N. Turner,et al.  The role of cardiac fibroblasts in the transition from inflammation to fibrosis following myocardial infarction. , 2013, Vascular pharmacology.

[2]  S. Ball,et al.  p38 MAPK alpha mediates cytokine-induced IL-6 and MMP-3 expression in human cardiac fibroblasts , 2013, Biochemical and biophysical research communications.

[3]  S. Ball,et al.  Human cardiac fibroblasts express ICAM-1, E-selectin and CXC chemokines in response to proinflammatory cytokine stimulation. , 2011, The international journal of biochemistry & cell biology.

[4]  J. Gardner,et al.  Induction of cardiac fibroblast lysyl oxidase by TGF-β1 requires PI3K/Akt, Smad3, and MAPK signaling. , 2011, Cytokine.

[5]  S. Ball,et al.  Modulatory effect of interleukin-1α on expression of structural matrix proteins, MMPs and TIMPs in human cardiac myofibroblasts: Role of p38 MAP kinase , 2010, Matrix biology : journal of the International Society for Matrix Biology.

[6]  A. Leask Potential Therapeutic Targets for Cardiac Fibrosis: TGF&bgr;, Angiotensin, Endothelin, CCN2, and PDGF, Partners in Fibroblast Activation , 2010, Circulation research.

[7]  Stephanie L. K. Bowers,et al.  Cardiac Fibroblast: The Renaissance Cell , 2009, Circulation research.

[8]  N. Turner,et al.  Interleukin-1alpha stimulates proinflammatory cytokine expression in human cardiac myofibroblasts. , 2009, American journal of physiology. Heart and circulatory physiology.

[9]  N. Turner,et al.  Cardiac fibroblasts: at the heart of myocardial remodeling. , 2009, Pharmacology & therapeutics.

[10]  A. Trafford,et al.  Extracellular matrix profiles in the progression to heart failure , 2008, Acta physiologica.

[11]  C. Long,et al.  Cytokines regulate matrix metalloproteinases and migration in cardiac fibroblasts. , 2007, Biochemical and biophysical research communications.

[12]  I. Dixon,et al.  Differential and combined effects of cardiotrophin-1 and TGF-beta1 on cardiac myofibroblast proliferation and contraction. , 2007, American journal of physiology. Heart and circulatory physiology.

[13]  D. Sorescu,et al.  NAD(P)H Oxidase 4 Mediates Transforming Growth Factor-β1–Induced Differentiation of Cardiac Fibroblasts Into Myofibroblasts , 2005, Circulation research.

[14]  P. Insel,et al.  Inhibition of cardiac myofibroblast formation and collagen synthesis by activation and overexpression of adenylyl cyclase. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[15]  T. Krieg,et al.  Myofibroblast differentiation is induced in keratinocyte-fibroblast co-cultures and is antagonistically regulated by endogenous transforming growth factor-beta and interleukin-1. , 2004, The American journal of pathology.

[16]  V. Petrov,et al.  Transforming growth factor-beta 1-mediated collagen gel contraction by cardiac fibroblasts. , 2004, Journal of the renin-angiotensin-aldosterone system : JRAAS.

[17]  S. Ball,et al.  Chronic beta2-adrenergic receptor stimulation increases proliferation of human cardiac fibroblasts via an autocrine mechanism. , 2003, Cardiovascular research.

[18]  S. Ball,et al.  The mechanism of angiotensin II-induced extracellular signal-regulated kinase-1/2 activation is independent of angiotensin AT(1A) receptor internalisation. , 2001, Cellular signalling.

[19]  V. Petrov,et al.  Induction of cardiac fibrosis by transforming growth factor-beta(1). , 2000, Molecular genetics and metabolism.

[20]  W. Colucci,et al.  Interleukin-1β and Tumor Necrosis Factor-α Decrease Collagen Synthesis and Increase Matrix Metalloproteinase Activity in Cardiac Fibroblasts In Vitro , 2000 .

[21]  J. Michel,et al.  Cardiac fibrosis and inflammation: interaction with hemodynamic and hormonal factors. , 1999, Cardiovascular research.

[22]  A. Newby,et al.  Synergistic upregulation of metalloproteinase‐9 by growth factors and inflammatory cytokines: an absolute requirement for transcription factor NF‐κB , 1998, FEBS letters.

[23]  G. Laurent,et al.  Collagen production and replication by cardiac fibroblasts is enhanced in response to diverse classes of growth factors. , 1995, European journal of cell biology.

[24]  M. Wilson,et al.  Integrin-mediated collagen gel contraction by cardiac fibroblasts. Effects of angiotensin II. , 1994, Circulation research.

[25]  M. Eghbali,et al.  Differential effects of transforming growth factor-beta 1 and phorbol myristate acetate on cardiac fibroblasts. Regulation of fibrillar collagen mRNAs and expression of early transcription factors. , 1991, Circulation research.

[26]  Johan Verjans,et al.  Myocardial remodeling after infarction: the role of myofibroblasts , 2010, Nature Reviews Cardiology.

[27]  C. Long,et al.  The cardiac fibroblast: therapeutic target in myocardial remodeling and failure. , 2005, Annual review of pharmacology and toxicology.

[28]  B. Chua,et al.  Effect of growth factors on collagen metabolism in cultured human heart fibroblasts. , 1991, Connective tissue research.

[29]  R. J. Kelm,et al.  Transforming Growth Factor (cid:1) 1-mediated Activation of the Smooth Muscle (cid:2) -Actin Gene in Human Pulmonary Myofibroblasts Is Inhibited by Tumor Necrosis Factor- (cid:2) via Mitogen-activated Protein Kinase Kinase 1-dependent Induction of the Egr-1 Transcriptional Repressor , 2022 .