potential of the common megakaryocytic-erythroid progenitor mutation alters the proliferation / differentiationlowThe hypomorphic
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S. Orkin | A. Migliaccio | A. Vannucchi | G. Migliaccio | B. Ghinassi | G. Amabile | F. Martelli | M. Sanchez
[1] J. Crispino,et al. GATA1s goes germline , 2006, Nature Genetics.
[2] A. F. Cunha,et al. An inherited mutation leading to production of only the short isoform of GATA-1 is associated with impaired erythropoiesis , 2006, Nature Genetics.
[3] Jie Zheng,et al. Multipotential differentiation ability of GATA-1-null erythroid-committed cells. , 2006, Genes & development.
[4] I. Weissman,et al. Differential Amplification of Murine Bipotent Megakaryocytic/Erythroid Progenitor and Precursor Cells During Recovery from Acute and Chronic Erythroid Stress , 2006, Stem cells.
[5] C. Bogani,et al. Variegation of the phenotype induced by the Gata1low mutation in mice of different genetic backgrounds. , 2005, Blood.
[6] M. Weiss,et al. Early block to erythromegakaryocytic development conferred by loss of transcription factor GATA-1. , 2005, Blood.
[7] I. Weissman,et al. Identification of mast cell progenitors in adult mice. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[8] A. Migliaccio,et al. Role of GATA‐1 in Normal and Neoplastic Hemopoiesis , 2005, Annals of the New York Academy of Sciences.
[9] S. Orkin,et al. Developmental stage–selective effect of somatically mutated leukemogenic transcription factor GATA1 , 2005, Nature Genetics.
[10] Lina A. Thoren,et al. Identification of Flt3+ Lympho-Myeloid Stem Cells Lacking Erythro-Megakaryocytic Potential A Revised Road Map for Adult Blood Lineage Commitment , 2005, Cell.
[11] M. Tsai,et al. Mast cells in the development of adaptive immune responses , 2005, Nature Immunology.
[12] S. Philipsen,et al. Leukemogenesis Caused by Incapacitated GATA-1 Function , 2004, Molecular and Cellular Biology.
[13] T. Toki,et al. The GATA1 mutation in an adult patient with acute megakaryoblastic leukemia not accompanying Down syndrome. , 2004, Blood.
[14] P. Vyas,et al. Natural history of GATA1 mutations in Down syndrome. , 2004, Blood.
[15] Y. Hayashi,et al. Frequent mutations in the GATA-1 gene in the transient myeloproliferative disorder of Down syndrome. , 2003, Blood.
[16] S. Orkin,et al. GATA-1 as a Regulator of Mast Cell Differentiation Revealed by the Phenotype of the GATA-1low Mouse Mutant , 2003, The Journal of experimental medicine.
[17] I. Weissman,et al. Characterization of mouse clonogenic megakaryocyte progenitors , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[18] S. Orkin,et al. X-linked thrombocytopenia with thalassemia from a mutation in the amino finger of GATA-1 affecting DNA binding rather than FOG-1 interaction. , 2002, Blood.
[19] A. Migliaccio,et al. Development of myelofibrosis in mice genetically impaired for GATA-1 expression (GATA-1(low) mice). , 2002, Blood.
[20] M. L. Beau,et al. Acquired mutations in GATA1 in the megakaryoblastic leukemia of Down syndrome , 2002, Nature Genetics.
[21] T. Graf,et al. Making Eosinophils Through Subtle Shifts in Transcription Factor Expression , 2002, The Journal of experimental medicine.
[22] S. Orkin,et al. Targeted Deletion of a High-Affinity GATA-binding Site in the GATA-1 Promoter Leads to Selective Loss of the Eosinophil Lineage In Vivo , 2002, The Journal of experimental medicine.
[23] S. Orkin,et al. Transcriptional regulation of erythropoiesis: an affair involving multiple partners , 2002, Oncogene.
[24] M. Crossley,et al. X-linked thrombocytopenia caused by a novel mutation of GATA-1. , 2001, Blood.
[25] K. Austen,et al. The Diverse Roles of Mast Cells , 2001, The Journal of experimental medicine.
[26] C Thys,et al. Platelet characteristics in patients with X-linked macrothrombocytopenia because of a novel GATA1 mutation. , 2001, Blood.
[27] Neil D. Theise,et al. Multi-Organ, Multi-Lineage Engraftment by a Single Bone Marrow-Derived Stem Cell , 2001, Cell.
[28] J. Sambrook,et al. Molecular Cloning: A Laboratory Manual , 2001 .
[29] A. Migliaccio,et al. Identification and characterization of a bipotent (erythroid and megakaryocytic) cell precursor from the spleen of phenylhydrazine-treated mice. , 2000, Blood.
[30] I. Weissman,et al. A clonogenic common myeloid progenitor that gives rise to all myeloid lineages , 2000, Nature.
[31] S. Orkin,et al. Familial dyserythropoietic anaemia and thrombocytopenia due to an inherited mutation in GATA1 , 2000, Nature Genetics.
[32] M. Koury,et al. Increased expression of the distal, but not of the proximal, Gata1 transcripts during differentiation of primary erythroid cells , 1999, Journal of cellular physiology.
[33] R. Shivdasani,et al. Cellular and molecular biology of megakaryocyte differentiation in the absence of lineage‐restricted transcription factors , 1998, Stem cells.
[34] S. Orkin,et al. A "knockdown" mutation created by cis-element gene targeting reveals the dependence of erythroid cell maturation on the level of transcription factor GATA-1. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[35] N. Hayashi,et al. Arrest in Primitive Erythroid Cell Development Caused by Promoter-specific Disruption of the GATA-1 Gene* , 1997, The Journal of Biological Chemistry.
[36] G. Stamatoyannopoulos,et al. Binary transgenic mouse model for studying the trans control of globin gene switching: evidence that GATA-1 is an in vivo repressor of human epsilon gene expression. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[37] M. Greaves,et al. Multilineage gene expression precedes commitment in the hemopoietic system. , 1997, Genes & development.
[38] S. Galli,et al. Identification of a Committed Precursor for the Mast Cell Lineage , 1996, Science.
[39] A. Nienhuis,et al. Dysregulated expression of GATA-1 following retrovirus-mediated gene transfer into murine hematopoietic stem cells increases erythropoiesis. , 1995, Blood.
[40] S. Orkin,et al. Transcription factor GATA-1 permits survival and maturation of erythroid precursors by preventing apoptosis. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[41] T. Graf,et al. GATA-1 reprograms avian myelomonocytic cell lines into eosinophils, thromboblasts, and erythroblasts. , 1995, Genes & development.
[42] J. Adamson,et al. Long-term generation of human mast cells in serum-free cultures of CD34+ cord blood cells stimulated with stem cell factor and interleukin-3. , 1994, Blood.
[43] J. Stamatoyannopoulos,et al. Position independence and proper developmental control of gamma-globin gene expression require both a 5' locus control region and a downstream sequence element , 1994, Molecular and cellular biology.
[44] D. Friend,et al. Strain-specific and tissue-specific expression of mouse mast cell secretory granule proteases. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[45] D. Gurley,et al. Isolation, characterization, and transcription of the gene encoding mouse mast cell protease 7. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[46] S. Orkin,et al. Erythroid differentiation in chimaeric mice blocked by a targeted mutation in the gene for transcription factor GATA-1 , 1991, Nature.
[47] K. Nocka,et al. Candidate ligand for the c‐kit transmembrane kinase receptor: KL, a fibroblast derived growth factor stimulates mast cells and erythroid progenitors. , 1990, The EMBO journal.
[48] K. Nocka,et al. Molecular bases of dominant negative and loss of function mutations at the murine c‐kit/white spotting locus: W37, Wv, W41 and W. , 1990, The EMBO journal.
[49] William Arbuthnot Sir Lane,et al. Different mouse mast cell populations express various combinations of at least six distinct mast cell serine proteases. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[50] Stuart H. Orkin,et al. Expression of an erythroid transcription factor in megakaryocytic and mast cell lineages , 1990, Nature.
[51] W. Vainchenker,et al. Megakaryocytic and erythrocytic lineages share specific transcription factors , 1990, Nature.
[52] William Arbuthnot Sir Lane,et al. Isolation and molecular cloning of mast cell carboxypeptidase A. A novel member of the carboxypeptidase gene family. , 1989, The Journal of biological chemistry.
[53] J. Adamson,et al. Selection of lineage-restricted cell lines immortalized at different stages of hematopoietic differentiation from the murine cell line 32D , 1989, The Journal of cell biology.
[54] Shih-Feng Tsai,et al. Cloning of cDNA for the major DNA-binding protein of the erythroid lineage through expression in mammalian cells , 1989, Nature.
[55] I. Peake. The Molecular Basis of Blood Diseases , 1988 .
[56] T. Suda,et al. Single-cell origin of mouse hemopoietic colonies expressing multiple lineages in variable combinations. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[57] J. Greenberger,et al. Demonstration of permanent factor-dependent multipotential (erythroid/neutrophil/basophil) hematopoietic progenitor cell lines. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[58] M. Nishiyama,et al. FOG-1 represses GATA-1-dependent FcepsilonRI beta-chain transcription: transcriptional mechanism of mast-cell-specific gene expression in mice. , 2006, Blood.
[59] 岩崎 浩己,et al. GATA-1 converts lymphoid and myelomonocytic progenitors into the megakaryocyte/erythrocyte lineages , 2006 .
[60] L. Zon,et al. Loss of gata1 but not gata2 converts erythropoiesis to myelopoiesis in zebrafish embryos. , 2005, Developmental cell.
[61] S. Orkin,et al. Development of hematopoietic cells lacking transcription factor GATA-1. , 1995, Development.
[62] 孝吉 山本,et al. 本態性血小板血症から7年後にmyelofibrosis with myeloid metaplasiaへ移行した1例 , 1991 .