Effect of superoxide dismutase, catalase, chelating agents, and free radical scavengers on the toxicity of alloxan to isolated pancreatic islets in vitro.
暂无分享,去创建一个
[1] R. Munday,et al. Comparative toxicity of alloxan, N-alkylalloxans and ninhydrin to isolated pancreatic islets in vitro. , 1997, The Journal of endocrinology.
[2] S. Lenzen,et al. Relation Between Antioxidant Enzyme Gene Expression and Antioxidative Defense Status of Insulin-Producing Cells , 1997, Diabetes.
[3] M. Abdel‐Rahman,et al. Protection of B cells against the effect of alloxan. , 1992, Toxicology letters.
[4] R. Munday,et al. Thiol-group reactivity, hydrophilicity and stability of alloxan, its reduction products and its N-methyl derivatives and a comparison with ninhydrin. , 1991, Biochemical pharmacology.
[5] U. Brunk,et al. Effects of alloxan and reducing agents on macrophages in culture , 1991, APMIS : acta pathologica, microbiologica, et immunologica Scandinavica.
[6] K. Sakurai,et al. Inhibitory effect of glutathione on the generation of hydroxyl radicals in the reaction system of glutathione-alloxan. , 1991 .
[7] T. Miura,et al. Role of alloxan radical in generation of hydroxyl radical by reaction of alloxan with glutathione in the presence of Fe3+-ethylenediaminetetraacetic acid. , 1990 .
[8] R. Munday,et al. Glutathione-mediated redox cycling of alloxan. Mechanisms of superoxide dismutase inhibition and of metal-catalyzed OH. formation. , 1989, Biochemical pharmacology.
[9] R. Munday. Dialuric acid autoxidation. Effects of transition metals on the reaction rate and on the generation of "active oxygen" species. , 1988, Biochemical pharmacology.
[10] G. Buettner. 1018 - ACTIVATION OF OXYGEN BY METAL COMPLEXES AND ITS RELEVANCE TO AUTOXIDATIVE PROCESSES IN LIVING SYSTEMS. , 1987, Journal of electroanalytical chemistry and interfacial electrochemistry.
[11] V. Ferrans,et al. Mechanism of the protective activity of ICRF-187 against alloxan-induced diabetes in mice. , 1986, Research communications in chemical pathology and pharmacology.
[12] K. Asayama,et al. Chemiluminescence as an Index of Drug-induced Free Radical Production in Pancreatic Islets , 1984, Diabetes.
[13] S. Marklund,et al. Opposite effects of two metal-chelators on alloxan-induced diabetes in mice. , 1983, Life sciences.
[14] S. Marklund,et al. Superoxide dismutase is a prophylactic against alloxan diabetes , 1981, Nature.
[15] V. Ferrans,et al. Reduction in the diabetogenic effect of alloxan in mice by treatment with the antineoplastic agent ICRF-187. , 1981, Research communications in chemical pathology and pharmacology.
[16] L. J. Fischer,et al. Inhibition of Alloxan Action in Isolated Pancreatic Islets by Superoxide Dismutase, Catalase, and a Metal Chelator , 1980, Diabetes.
[17] R. Heikkila,et al. Protection against alloxan-induced diabetes by various urea derivatives: relationship between protective effects and reactivity with the hydroxyl radical. , 1979, The Journal of pharmacology and experimental therapeutics.
[18] S. Marklund,et al. Superoxide dismutase, catalase and scavengers of hydroxyl radical protect against the toxic action of alloxan on pancreatic islet cells in vitro. , 1979, The Biochemical journal.
[19] R. Heikkila,et al. Protection against alloxan-induced diabetes in mice by the hydroxyl radical scavenger dimethylurea. , 1978, European journal of pharmacology.
[20] J. Crapo,et al. The failure of aerosolized superoxide dismutase to modify pulmonary oxygen toxicity. , 1977, The American review of respiratory disease.
[21] C. Schauberger,et al. Mechanism of protection from alloxan diabetes provided by n-butanol. , 1977, The Journal of pharmacology and experimental therapeutics.
[22] G. Cohen,et al. Alloxan-induced diabetes-evidence for hydroxyl radical as a cytotoxic intermediate. , 1976, Biochemical pharmacology.
[23] G. Cohen,et al. Prevention of alloxan-induced diabetes by ethanol administration. , 1974, Journal of Pharmacology and Experimental Therapeutics.
[24] M. Anbar,et al. Selected specific rates of reactions of transients from water in aqueous solution. 1. Hydrated electron , 1973 .
[25] M. Anbar,et al. A compilation of specific bimolecular rate constants for the reactions of hydrated electrons, hydrogen atoms and hydroxyl radicals with inorganic and organic compounds in aqueous solution , 1967 .
[26] W. Carter,et al. Studies on Protection Against the Diabetogenic Effect of Alloxan by Glucose.∗ , 1962, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[27] B. Waisbren,et al. A Method for Consistent Induction of Chronic Hyperglycemia with Alloxan , 1945, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[28] T. Masukawa,et al. Involvement of blood glucose in the dimethylthiourea-induced protection against alloxan-induced diabetes. , 1994, Japanese journal of pharmacology.
[29] B. Halliwell,et al. Protection against tissue damage in vivo by desferrioxamine: what is its mechanism of action? , 1989, Free radical biology & medicine.
[30] L. Oberley. Free radicals and diabetes. , 1988, Free radical biology & medicine.
[31] A. Boschero,et al. 1,10 phenanthroline, a metal chelator, protects against alloxan- but not streptozotocin-induced diabetes. , 1986, Journal of free radicals in biology & medicine.
[32] S. J. Cooperstein,et al. 15 – Action of Toxic Drugs on Islet Cells , 1981 .
[33] Farhataziz,et al. Selected specific rates of reactions of transients from water in aqueous solution. III. Hydroxyl radical and perhydroxyl radical and their radical ions , 1977 .
[34] John Leyden. Webb,et al. Enzyme and metabolic inhibitors , 1963 .