Differentiation of IL-26+ TH17 intermediates into IL-17A producers via epithelial crosstalk in psoriasis
暂无分享,去创建一个
M. Haniffa | F. Kuonen | J. Di Domizio | M. Gilliet | J. Gudjonsson | C. Conrad | T. Werfel | R. Modlin | F. Saidoune | Feiyang Ma | L. Roesner | A. Fries | Yang Li | Nadine Fournier | I. Dupanloup | A. C. Guerra de Souza | Raphael Gottardo | R. Gottardo
[1] M. Menden,et al. Spatial transcriptomics landscape of lesions from non-communicable inflammatory skin diseases , 2022, Nature communications.
[2] V. Koeken,et al. Single‐cell profiles reveal distinctive immune response in atopic dermatitis in contrast to psoriasis , 2022, Allergy.
[3] Scott N. Mueller,et al. Discrete tissue microenvironments instruct diversity in resident memory T cell function and plasticity , 2021, Nature Immunology.
[4] Helena L. Crowell,et al. SpatialExperiment: infrastructure for spatially-resolved transcriptomics data in R using Bioconductor , 2021, bioRxiv.
[5] S. Teichmann,et al. Developmental cell programs are co-opted in inflammatory skin disease , 2021, Science.
[6] Raphael Gottardo,et al. Integrated analysis of multimodal single-cell data , 2020, Cell.
[7] R. Bowden,et al. Single-cell atlas of colonic CD8+ T cells in ulcerative colitis , 2020, Nature Medicine.
[8] M. Allez,et al. KLRG1 and CD103 Expressions Define Distinct Intestinal Tissue-Resident Memory CD8 T Cell Subsets Modulated in Crohn's Disease , 2020, Frontiers in Immunology.
[9] J. Kere,et al. Microbe-host interplay in atopic dermatitis and psoriasis , 2019, Nature Communications.
[10] Shinichi Sato,et al. Increased interleukin-26 expression promotes Th17 and Th2-associated cytokine production by keratinocytes in atopic dermatitis. , 2019, The Journal of investigative dermatology.
[11] Bryan D. Bryson,et al. IL-1β Induces the Rapid Secretion of the Antimicrobial Protein IL-26 from Th17 Cells , 2019, The Journal of Immunology.
[12] C. Singer,et al. Regulation of IL-17A and implications for TGF-β1 comodulation of airway smooth muscle remodeling in severe asthma. , 2019, American journal of physiology. Lung cellular and molecular physiology.
[13] Andrew J. Hill,et al. The single cell transcriptional landscape of mammalian organogenesis , 2019, Nature.
[14] W. Boehncke,et al. The IL-17 Family of Cytokines in Psoriasis: IL-17A and Beyond , 2018, Front. Immunol..
[15] Song Zhang. The role of transforming growth factor β in T helper 17 differentiation , 2018, Immunology.
[16] V. Procaccio,et al. IL-26 Confers Proinflammatory Properties to Extracellular DNA , 2017, The Journal of Immunology.
[17] S. Krebs,et al. Melanocyte antigen triggers autoimmunity in human psoriasis , 2015, The Journal of experimental medicine.
[18] S. Watowich,et al. TH17 cells promote microbial killing and innate immune sensing of DNA via interleukin 26 , 2015, Nature Immunology.
[19] A. Regev,et al. Spatial reconstruction of single-cell gene expression , 2015, Nature Biotechnology.
[20] B. Marinari,et al. The antimicrobial peptide LL37 is a T-cell autoantigen in psoriasis , 2014, Nature Communications.
[21] Xiao-ming Meng,et al. Transforming growth factor‐β1 mediates psoriasis‐like lesions via a Smad3‐dependent mechanism in mice , 2014, Clinical and experimental pharmacology & physiology.
[22] H. Al‐Shobaili,et al. Serum Vascular Endothelial Growth Factor, Transforming Growth Factor β1, and Nitric Oxide Levels in Patients With Psoriasis Vulgaris: Their Correlation to Disease Severity , 2014, Journal of clinical laboratory analysis.
[23] M. Audran,et al. IL-26 Is Overexpressed in Rheumatoid Arthritis and Induces Proinflammatory Cytokine Production and Th17 Cell Generation , 2012, PLoS biology.
[24] D. Valmori,et al. Ex Vivo IL-1 Receptor Type I Expression in Human CD4+ T Cells Identifies an Early Intermediate in the Differentiation of Th17 from FOXP3+ Naive Regulatory T Cells , 2011, The Journal of Immunology.
[25] D. Valmori,et al. Human RORγt+ TH17 cells preferentially differentiate from naive FOXP3+Treg in the presence of lineage-specific polarizing factors , 2010, Proceedings of the National Academy of Sciences.
[26] Casey T. Weaver,et al. TGF-β Promotes Th17 Cell Development through Inhibition of SOCS31 , 2009, The Journal of Immunology.
[27] Thomas Korn,et al. IL-17 and Th17 Cells. , 2009, Annual review of immunology.
[28] James T. Elder,et al. Induction of IL-17+ T Cell Trafficking and Development by IFN-γ: Mechanism and Pathological Relevance in Psoriasis1 , 2008, The Journal of Immunology.
[29] David E. Anderson,et al. IL-21 and TGF-β are required for differentiation of human TH17 cells , 2008, Nature.
[30] E. Barillot,et al. A critical function for transforming growth factor-β, interleukin 23 and proinflammatory cytokines in driving and modulating human TH-17 responses , 2008, Nature Immunology.
[31] S. Brand,et al. The role of the novel Th17 cytokine IL-26 in intestinal inflammation , 2008, Gut.
[32] D. Littman,et al. The differentiation of human TH-17 cells requires transforming growth factor-β and induction of the nuclear receptor RORγt , 2008, Nature Immunology.
[33] I. Flisiak,et al. Plasma TGF-β1, TIMP-1, MMP-1 and IL-18 as a combined biomarker of psoriasis activity , 2008, Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals.
[34] Kathleen M. Smith,et al. Development, cytokine profile and function of human interleukin 17–producing helper T cells , 2007, Nature Immunology.
[35] F. Sallusto,et al. Interleukins 1β and 6 but not transforming growth factor-β are essential for the differentiation of interleukin 17–producing human T helper cells , 2007, Nature Immunology.
[36] Ying Wang,et al. A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17 , 2005, Nature Immunology.
[37] R. D. Hatton,et al. Interleukin 17–producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages , 2005, Nature Immunology.
[38] Xin-Hua Feng,et al. Latent TGFβ1 overexpression in keratinocytes results in a severe psoriasis‐like skin disorder , 2004, The EMBO journal.
[39] R. Flisiak,et al. ASSOCIATION BETWEEN PSORIASIS SEVERITY AND TRANSFORMING GROWTH FACTOR β1 AND β2 IN PLASMA AND SCALES FROM PSORIATIC LESIONS , 2002 .