The protective microRNA-199a-5p-mediated unfolded protein response in hypoxic cardiomyocytes is regulated by STAT3 pathway
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Peng Li | Yang Zhou | Bo Pang | Simin Zhou | Yingbin Xiao | Fan Zhang | Wei Liu | Huali Peng | Bin He
[1] H. Ouyang,et al. Protective role of downregulated MLK3 in myocardial adaptation to chronic hypoxia , 2016, Journal of Physiology and Biochemistry.
[2] David J. Arenillas,et al. JASPAR 2016: a major expansion and update of the open-access database of transcription factor binding profiles , 2015, Nucleic Acids Res..
[3] P. Doevendans,et al. HypoxamiRs: regulators of cardiac hypoxia and energy metabolism , 2015, Trends in Endocrinology & Metabolism.
[4] Z. Tian,et al. miR-146a is directly regulated by STAT3 in human hepatocellular carcinoma cells and involved in anti-tumor immune suppression , 2015, Cell cycle.
[5] Xi Dong,et al. Elatoside C protects against hypoxia/reoxygenation-induced apoptosis in H9c2 cardiomyocytes through the reduction of endoplasmic reticulum stress partially depending on STAT3 activation , 2014, Apoptosis.
[6] J. Mak,et al. Cellular mechanisms in intermittent hypoxia-induced cardiac damage in vivo , 2014, Journal of Physiology and Biochemistry.
[7] Chia-Yi Hsu,et al. miRNA‐199a‐5p regulates VEGFA in endometrial mesenchymal stem cells and contributes to the pathogenesis of endometriosis , 2014, The Journal of pathology.
[8] P. Buckley,et al. miR-199a-5p silencing regulates the unfolded protein response in chronic obstructive pulmonary disease and α1-antitrypsin deficiency. , 2014, American journal of respiratory and critical care medicine.
[9] Chun-Hsi Huang,et al. LASAGNA-Search: an integrated web tool for transcription factor binding site search and visualization. , 2013, BioTechniques.
[10] Anindita Das,et al. Rapamycin protects against myocardial ischemia-reperfusion injury through JAK2-STAT3 signaling pathway. , 2012, Journal of molecular and cellular cardiology.
[11] Y. Fujio,et al. Therapeutic administration of IL-11 exhibits the postconditioning effects against ischemia-reperfusion injury via STAT3 in the heart. , 2012, American journal of physiology. Heart and circulatory physiology.
[12] Yingchun Zhou,et al. Interleukin 6 protects H2O2‐induced cardiomyocytes injury through upregulation of prohibitin via STAT3 phosphorylation , 2012, Cell biochemistry and function.
[13] K. Clarke,et al. Metabolic adaptation to chronic hypoxia in cardiac mitochondria , 2012, Basic Research in Cardiology.
[14] Michael A McGuckin,et al. The interplay between endoplasmic reticulum stress and inflammation , 2012, Immunology and cell biology.
[15] K. Huber,et al. Inflammation and cardiac outcome , 2011, Current opinion in infectious diseases.
[16] Yingbin Xiao,et al. Hypoxia-induced SOCS3 is limiting STAT3 phosphorylation and NF-κB activation in congenital heart disease. , 2011, Biochimie.
[17] M. Muckenthaler,et al. Signal transducer and activator of transcription 3-mediated regulation of miR-199a-5p links cardiomyocyte and endothelial cell function in the heart: a key role for ubiquitin-conjugating enzymes. , 2011, European heart journal.
[18] J. Minna,et al. STAT3 mediates resistance to MEK inhibitor through microRNA miR-17. , 2011, Cancer research.
[19] I. Komuro,et al. Therapeutic Activation of Signal Transducer and Activator of Transcription 3 by Interleukin-11 Ameliorates Cardiac Fibrosis After Myocardial Infarction , 2010, Circulation.
[20] S. Vatner,et al. Downregulation of MiR-199a Derepresses Hypoxia-Inducible Factor-1α and Sirtuin 1 and Recapitulates Hypoxia Preconditioning in Cardiac Myocytes , 2009, Circulation research.
[21] V. Kim,et al. Regulation of microRNA biogenesis , 2014, Nature Reviews Molecular Cell Biology.
[22] J. Uney,et al. Twist-1 regulates the miR-199a/214 cluster during development , 2008, Nucleic acids research.
[23] Randal J. Kaufman,et al. From endoplasmic-reticulum stress to the inflammatory response , 2008, Nature.
[24] Mark A Sussman,et al. Hypoxic Cultured Cardiac Myocytes Activation of the Unfolded Protein Response in Infarcted Mouse Heart , 2006 .
[25] S. S. Zhang,et al. Cardiomyocyte-restricted knockout of STAT3 results in higher sensitivity to inflammation, cardiac fibrosis, and heart failure with advanced age , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[26] V. Fineschi. Measuring myocyte oxidative stress and targeting cytokines to evaluate inflammatory response and cardiac repair after myocardial infarction. , 2015, Current vascular pharmacology.
[27] M. Neri,et al. Cardiac oxidative stress and inflammatory cytokines response after myocardial infarction. , 2015, Current vascular pharmacology.