Disruption of the surfactant protein A receptor SP-R210L (CD245α/MYO18Aα) alters respiratory function and iron sequestration in alveolar macrophages of aged mice
暂无分享,去创建一个
Amy C. Arnold | S. Diangelo | T. Cooper | Hannah M. Atkins | E. Yau | T. Umstead | Z. Chroneos | E. Halstead | Raz Abdulqadir | S. Bingaman | Sanmei Hu | Z. Guan | Kristin K. Fino | Kavitha Hassan | Raz F. Abdulqadir
[1] Shi-You Chen,et al. Surfactant Protein A, a Novel Regulator for Smooth Muscle Phenotypic Modulation and Vascular Remodeling. , 2020, Arteriosclerosis, thrombosis, and vascular biology.
[2] D. Hume,et al. Network analysis of transcriptomic diversity amongst resident tissue macrophages and dendritic cells in the mouse mononuclear phagocyte system , 2020, PLoS biology.
[3] Sijin Liu,et al. The cardinal roles of ferroportin and its partners in controlling cellular iron in and out. , 2020, Life sciences.
[4] Xiaopeng Zhou,et al. Osmolarity controls the differentiation of adipose-derived stem cells into nucleus pulposus cells via histone demethylase KDM4B , 2020, Molecular and Cellular Biochemistry.
[5] A. Mammoto,et al. Vascular Niche in Lung Alveolar Development, Homeostasis, and Regeneration , 2019, Front. Bioeng. Biotechnol..
[6] C. Casals,et al. Soluble defense collagens: Sweeping up immune threats. , 2019, Molecular immunology.
[7] R. Veldhuizen,et al. The effects of aging and exercise on lung mechanics, surfactant and alveolar macrophages , 2019, Experimental lung research.
[8] W. Linke,et al. A novel isoform of myosin 18A (Myo18Aγ) is an essential sarcomeric protein in mouse heart , 2019, The Journal of Biological Chemistry.
[9] A. Krieg,et al. KDM4B: A Nail for Every Hammer? , 2019, Genes.
[10] M. Rundgren,et al. Feto-maternal osmotic balance at term. A prospective observational study , 2018, Journal of perinatal medicine.
[11] S. Morikawa,et al. Morphological characterization of pulmonary microvascular disease in bronchopulmonary dysplasia caused by hyperoxia in newborn mice , 2018, Medical Molecular Morphology.
[12] C. Nunemaker,et al. STEAP4: its emerging role in metabolism and homeostasis of cellular iron and copper. , 2017, The Journal of endocrinology.
[13] M. Vijay-Kumar,et al. Lipocalin 2: An Emerging Player in Iron Homeostasis and Inflammation. , 2017, Annual review of nutrition.
[14] T. Sutherland,et al. Local amplifiers of IL-4Rα–mediated macrophage activation promote repair in lung and liver , 2017, Science.
[15] Johannes Kung,et al. Structure, genetics and function of the pulmonary associated surfactant proteins A and D: The extra-pulmonary role of these C type lectins. , 2017, Annals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft.
[16] A. Katz,et al. Non-Pulmonary Immune Functions of Surfactant Proteins A and D , 2016, Journal of Innate Immunity.
[17] D. Cornfield,et al. Unique Aspects of the Developing Lung Circulation: Structural Development and Regulation of Vasomotor Tone , 2016, Pulmonary circulation.
[18] D. Gossot,et al. Identification of CD245 as myosin 18A, a receptor for surfactant A: A novel pathway for activating human NK lymphocytes , 2016, Oncoimmunology.
[19] E. Karteris,et al. Surfactant Proteins SP-A and SP-D Modulate Uterine Contractile Events in ULTR Myometrial Cell Line , 2015, PloS one.
[20] Xiaoping Yang,et al. IL-37 Is a Novel Proangiogenic Factor of Developmental and Pathological Angiogenesis , 2015, Arteriosclerosis, thrombosis, and vascular biology.
[21] S. England,et al. Fetal-to-maternal signaling to initiate parturition. , 2015, The Journal of clinical investigation.
[22] N. Christensen,et al. SP-R210 (Myo18A) Isoforms as Intrinsic Modulators of Macrophage Priming and Activation , 2015, PloS one.
[23] J. Whisstock,et al. IL-37 requires the receptors IL-18Rα and IL-1R8 (SIGIRR) to carry out its multifaceted anti-inflammatory program upon innate signal transduction , 2015, Nature Immunology.
[24] M. Mhanna,et al. Pathophysiology, Management, and Outcome of Persistent Pulmonary Hypertension of the Newborn: A Clinical Review , 2013, Front. Pediatr..
[25] Steve D. M. Brown,et al. Otitis media in the Tgif knockout mouse implicates TGFβ signalling in chronic middle ear inflammatory disease , 2013, Human molecular genetics.
[26] M. Fleming,et al. HRG1 is essential for heme transport from the phagolysosome of macrophages during erythrophagocytosis. , 2013, Cell metabolism.
[27] B. Geisbrecht,et al. Surfactant Protein A (SP-A)-mediated Clearance of Staphylococcus aureus Involves Binding of SP-A to the Staphylococcal Adhesin Eap and the Macrophage Receptors SP-A Receptor 210 and Scavenger Receptor Class A* , 2010, The Journal of Biological Chemistry.
[28] B. Barna,et al. Targeted PPAR³ Deficiency In Alveolar Macrophages Disrupts Surfactant Catabolism , 2010, ATS 2010.
[29] Z. Sever-Chroneos,et al. Pulmonary Surfactant: An Immunological Perspective , 2009, Cellular Physiology and Biochemistry.
[30] H. L. Persson,et al. Lysosomal iron in pulmonary alveolar proteinosis: a case report , 2009, European Respiratory Journal.
[31] Z. Sever-Chroneos,et al. An antibody against the surfactant protein A (SP‐A)‐binding domain of the SP‐A receptor inhibits T cell‐mediated immune responses to Mycobacterium tuberculosis , 2008, Journal of leukocyte biology.
[32] B. Reizis,et al. Notch–RBP-J signaling controls the homeostasis of CD8− dendritic cells in the spleen , 2007, The Journal of experimental medicine.
[33] J. Shabanowitz,et al. Identification of the Surfactant Protein A Receptor 210 as the Unconventional Myosin 18A* , 2005, Journal of Biological Chemistry.
[34] C. Mendelson,et al. Surfactant protein secreted by the maturing mouse fetal lung acts as a hormone that signals the initiation of parturition. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[35] M. Obinata,et al. Genome structure and differential expression of two isoforms of a novel PDZ-containing myosin (MysPDZ) (Myo18A). , 2003, Journal of biochemistry.
[36] O. Elidemir,et al. Time course of hemosiderin production by alveolar macrophages in a murine model. , 2001, Chest.
[37] J. Whitsett,et al. GM-CSF regulates alveolar macrophage differentiation and innate immunity in the lung through PU.1. , 2001, Immunity.
[38] J. Whitsett,et al. GM-CSF regulates protein and lipid catabolism by alveolar macrophages. , 2001, American journal of physiology. Lung cellular and molecular physiology.
[39] J. Whitsett,et al. Purification of a Cell-surface Receptor for Surfactant Protein A* , 1996, The Journal of Biological Chemistry.
[40] V. Shepherd,et al. Differential regulation of the mannose and SP-A receptors on macrophages. , 1995, The American journal of physiology.
[41] H. Blau,et al. Secretion of cytokines by rat alveolar epithelial cells: possible regulatory role for SP-A. , 1994, The American journal of physiology.
[42] H. Bluethmann,et al. Targeted disruption of the MHC class II Aa gene in C57BL/6 mice. , 1993, International immunology.
[43] R. Auten,et al. Surfactant apoprotein A (SP-A) is synthesized in airway cells. , 1990, American journal of respiratory cell and molecular biology.
[44] N. Andrews,et al. Iron homeostasis. , 2007, Annual review of physiology.
[45] W. Friedrichs,et al. Cell type-specific and inflammatory-induced expression of haptoglobin gene in lung. , 1995, Laboratory investigation; a journal of technical methods and pathology.
[46] C. Stewart,et al. Simple screening procedure to detect gene targeting events in embryonic stem cells. , 1993, Methods in enzymology.