Recombinant VEGF-C (Cys156Ser) improves mesenteric lymphatic drainage and gut immune surveillance in experimental cirrhosis
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S. Sarin | A. Rastogi | Pinky Juneja | S. Kaur | I. Kaur | V. Naidu | D. Tripathi | S. Baweja | P. Rawal | S. Rohilla | A. K. Mourya | Syed Nazrin Ruhina Rahman | Subham Banerjee | V. Tiwari | Deepika Jakhar | Abhishek Gupta | V.G.M. Naidu | S. Banerjee
[1] G. D’Amico,et al. Corrigendum to 'Towards a new definition of decompensated cirrhosis' [J Hepatol 76 (2022) 202-207]. , 2022, Journal of hepatology.
[2] D. Weissman,et al. Nucleoside-modified VEGFC mRNA induces organ-specific lymphatic growth and reverses experimental lymphedema , 2021, Nature Communications.
[3] H. Qian,et al. Implications of lymphatic alterations in the pathogenesis and treatment of inflammatory bowel disease. , 2021, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[4] H. Fukui. Leaky Gut and Gut-Liver Axis in Liver Cirrhosis: Clinical Studies Update , 2020, Gut and liver.
[5] M. McConnell,et al. Enhanced meningeal lymphatic drainage ameliorates neuroinflammation and hepatic encephalopathy in cirrhotic rats , 2020, bioRxiv.
[6] A. de Gottardi,et al. The gut-liver axis in liver disease: pathophysiological basis for therapy. , 2020, Journal of hepatology.
[7] T. H. Nguyen,et al. The global, regional, and national burden of cirrhosis by cause in 195 countries and territories, 1990–2017: a systematic analysis for the Global Burden of Disease Study 2017 , 2020, The lancet. Gastroenterology & hepatology.
[8] Ho Eun Park,et al. Secondary Lymphedema After Intestinal Tuberculosis: A Case Report , 2019, Annals of rehabilitation medicine.
[9] D. Mcclements,et al. Oil-in-water Pickering emulsions via microfluidization with cellulose nanocrystals: 1. Formation and stability , 2019, Food Hydrocolloids.
[10] K. Bhaumik,et al. Mechanisms of the effectiveness of lipid nanoparticle formulations loaded with anti-tubercular drugs combinations toward overcoming drug bioavailability in tuberculosis , 2019, Journal of drug targeting (Print).
[11] Jonathan Pillai,et al. Solid lipid matrix mediated nanoarchitectonics for improved oral bioavailability of drugs , 2019, Expert opinion on drug metabolism & toxicology.
[12] V. Engelhard,et al. The Antigen Processing and Presentation Machinery in Lymphatic Endothelial Cells , 2019, Front. Immunol..
[13] P. Kamath,et al. Burden of liver diseases in the world. , 2019, Journal of hepatology.
[14] A. Eichmann,et al. The Intestinal Lymphatic System: Functions and Metabolic Implications , 2018, Cellular and molecular gastroenterology and hepatology.
[15] M. Detmar,et al. Antibody-mediated delivery of VEGF-C potently reduces chronic skin inflammation. , 2018, JCI insight.
[16] J. Bosch,et al. Simvastatin Prevents Progression of Acute on Chronic Liver Failure in Rats With Cirrhosis and Portal Hypertension. , 2018, Gastroenterology.
[17] D. Greaves,et al. The cardiac lymphatic system stimulates resolution of inflammation following myocardial infarction , 2018, The Journal of clinical investigation.
[18] E. Schwarz,et al. Targeting lymphatic function as a novel therapeutic intervention for rheumatoid arthritis , 2018, Nature Reviews Rheumatology.
[19] G. Tovar,et al. Controlled Release of Vascular Endothelial Growth Factor from Heparin-Functionalized Gelatin Type A and Albumin Hydrogels , 2017, Gels.
[20] J. Hubbell,et al. Local induction of lymphangiogenesis with engineered fibrin-binding VEGF-C promotes wound healing by increasing immune cell trafficking and matrix remodeling. , 2017, Biomaterials.
[21] T. Petrova,et al. Intestinal lymphatic vasculature: structure, mechanisms and functions , 2017, Nature Reviews Gastroenterology &Hepatology.
[22] K. Alitalo,et al. VEGF-C is required for intestinal lymphatic vessel maintenance and lipid absorption , 2015, EMBO molecular medicine.
[23] K. Alitalo,et al. VEGF-C and VEGF-C156S in the pro-lymphangiogenic growth factor therapy of lymphedema: a large animal study , 2015, Angiogenesis.
[24] M. Simons,et al. Molecular Controls of Lymphatic VEGFR3 Signaling , 2015, Arteriosclerosis, thrombosis, and vascular biology.
[25] A. Gandelli,et al. VEGF-C-dependent stimulation of lymphatic function ameliorates experimental inflammatory bowel disease. , 2014, The Journal of clinical investigation.
[26] M. Detmar,et al. Lymphatic vessels: new targets for the treatment of inflammatory diseases , 2014, Angiogenesis.
[27] K. Maruyama,et al. The VEGF-C/VEGFR3 signaling pathway contributes to resolving chronic skin inflammation by activating lymphatic vessel function. , 2014, Journal of dermatological science.
[28] Y. Iwakiri,et al. The lymphatic vascular system in liver diseases: its role in ascites formation , 2013, Clinical and molecular hepatology.
[29] Kara F. Held,et al. Increased nitric oxide production in lymphatic endothelial cells causes impairment of lymphatic drainage in cirrhotic rats , 2012, Gut.
[30] Hua Song,et al. Sustained release of VEGF from PLGA nanoparticles embedded thermo-sensitive hydrogel in full-thickness porcine bladder acellular matrix , 2011, Nanoscale research letters.
[31] H. Freeman,et al. Intestinal lymphangiectasia in adults. , 2011, World journal of gastrointestinal oncology.
[32] S. Danese. Role of the vascular and lymphatic endothelium in the pathogenesis of inflammatory bowel disease: ‘brothers in arms’ , 2011, Gut.
[33] R. Francés,et al. Critical role of the liver in the induction of systemic inflammation in rats with preascitic cirrhosis , 2010, Hepatology.
[34] M. Detmar,et al. Activation of the VEGFR-3 pathway by VEGF-C attenuates UVB-induced edema formation and skin inflammation by promoting lymphangiogenesis. , 2009, The Journal of investigative dermatology.
[35] A. Macpherson,et al. Mesenteric lymph nodes at the center of immune anatomy , 2006, The Journal of experimental medicine.
[36] T. Veikkola,et al. Lymphangiogenic growth factors, receptors and therapies , 2003, Thrombosis and Haemostasis.
[37] M. Maynar,et al. Abdominal decompression plays a major role in early postparacentesis haemodynamic changes in cirrhotic patients with tense ascites , 2001, Gut.
[38] A. West,et al. Bacterial translocation to mesenteric lymph nodes is increased in cirrhotic rats with ascites. , 1995, Gastroenterology.
[39] G. Laine,et al. Intestinal lymphatic flow during portal venous hypertension. , 1989, The American journal of physiology.
[40] M. Geuking,et al. Pathological bacterial translocation in liver cirrhosis. , 2014, Journal of hepatology.