Carotid FDG Uptake Improves Prediction of Future Cardiovascular Events in Asymptomatic Individuals.
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Kyung-Han Lee | J. Choi | S. Moon | Y. Cho | Byung‐Tae Kim | T. Noh
[1] D. Goff,et al. 2013 ACC/AHA guideline on the assessment of cardiovascular risk: a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines. , 2014, Journal of the American College of Cardiology.
[2] T. Brady,et al. Measurement of arterial activity on routine FDG PET/CT images improves prediction of risk of future CV events. , 2013, JACC. Cardiovascular imaging.
[3] Kyung-Han Lee,et al. Relation of Carotid Artery 18F-FDG Uptake to C-Reactive Protein and Framingham Risk Score in a Large Cohort of Asymptomatic Adults , 2013, The Journal of Nuclear Medicine.
[4] Giel Nijpels,et al. Common carotid intima-media thickness measurements in cardiovascular risk prediction: a meta-analysis. , 2012, JAMA.
[5] J. Rudd,et al. Vascular imaging with positron emission tomography , 2011, Journal of internal medicine.
[6] Eric Boerwinkle,et al. Carotid intima-media thickness and presence or absence of plaque improves prediction of coronary heart disease risk: the ARIC (Atherosclerosis Risk In Communities) study. , 2010, Journal of the American College of Cardiology.
[7] Paul M Ridker,et al. Inflammation in atherosclerosis: from pathophysiology to practice. , 2009, Journal of the American College of Cardiology.
[8] S. Grundy,et al. Harmonizing the metabolic syndrome: a joint interim statement of the International Diabetes Federation Task Force on Epidemiology and Prevention; National Heart, Lung, and Blood Institute; American Heart Association; World Heart Federation; International Atherosclerosis Society; and International As , 2009, Circulation.
[9] M. Reiser,et al. 18F-FDG PET/CT Identifies Patients at Risk for Future Vascular Events in an Otherwise Asymptomatic Cohort with Neoplastic Disease , 2009, Journal of Nuclear Medicine.
[10] J. Wassélius,et al. Assessment of inactive, active and mixed atherosclerotic plaques by 18F-FDG-PET; an age group-based correlation with cardiovascular risk factors , 2009, The International Journal of Cardiovascular Imaging.
[12] Marc Faraggi,et al. Arterial wall uptake of fluorodeoxyglucose on PET imaging in stable cancer disease patients indicates higher risk for cardiovascular events , 2008, Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology.
[13] T. Imaizumi,et al. Vascular inflammation evaluated by [18F]-fluorodeoxyglucose positron emission tomography is associated with the metabolic syndrome. , 2007, Journal of the American College of Cardiology.
[14] Ahmed Tawakol,et al. In vivo 18F-fluorodeoxyglucose positron emission tomography imaging provides a noninvasive measure of carotid plaque inflammation in patients. , 2006, Journal of the American College of Cardiology.
[15] Guoqing Diao,et al. Estimation of time‐dependent area under the ROC curve for long‐term risk prediction , 2006, Statistics in medicine.
[16] F. Harrell,et al. Prognostic/Clinical Prediction Models: Multivariable Prognostic Models: Issues in Developing Models, Evaluating Assumptions and Adequacy, and Measuring and Reducing Errors , 2005 .
[17] Elena Bonanno,et al. Diffuse and active inflammation occurs in both vulnerable and stable plaques of the entire coronary tree: a histopathologic study of patients dying of acute myocardial infarction. , 2005, Journal of the American College of Cardiology.
[18] H. Watabe,et al. (18)F-FDG accumulation in atherosclerotic plaques: immunohistochemical and PET imaging study. , 2004, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.
[19] Renu Virmani,et al. Intraplaque hemorrhage and progression of coronary atheroma. , 2003, The New England journal of medicine.
[20] O. Wagner,et al. Joint Effects of C-Reactive Protein and Glycated Hemoglobin in Predicting Future Cardiovascular Events of Patients With Advanced Atherosclerosis , 2003, Circulation.
[21] Antonio Colombo,et al. From vulnerable plaque to vulnerable patient: a call for new definitions and risk assessment strategies: Part II. , 2003, Circulation.
[22] E. Boerwinkle,et al. From vulnerable plaque to vulnerable patient: a call for new definitions and risk assessment strategies: Part I. , 2003, Circulation.
[23] S. Devaraj,et al. C-Reactive Protein Increases Plasminogen Activator Inhibitor-1 Expression and Activity in Human Aortic Endothelial Cells: Implications for the Metabolic Syndrome and Atherothrombosis , 2003, Circulation.
[24] Ren-Ke Li,et al. A Self-Fulfilling Prophecy: C-Reactive Protein Attenuates Nitric Oxide Production and Inhibits Angiogenesis , 2002, Circulation.
[25] W. James,et al. Appropriate Asian body mass indices? , 2002, Obesity reviews : an official journal of the International Association for the Study of Obesity.
[26] J. Pickard,et al. Imaging Atherosclerotic Plaque Inflammation With [18F]-Fluorodeoxyglucose Positron Emission Tomography , 2002, Circulation.
[27] P. Ridker,et al. C-reactive protein and other markers of inflammation in the prediction of cardiovascular disease in women. , 2000, The New England journal of medicine.
[28] A H Davies,et al. The symptomatic carotid plaque. , 2000, Stroke.
[29] K. Williams,et al. Atherosclerosis--an inflammatory disease. , 1999, The New England journal of medicine.
[30] A. Rebuzzi,et al. Elevated levels of C-reactive protein at discharge in patients with unstable angina predict recurrent instability. , 1999, Circulation.
[31] A. Döring,et al. C-Reactive protein, a sensitive marker of inflammation, predicts future risk of coronary heart disease in initially healthy middle-aged men: results from the MONICA (Monitoring Trends and Determinants in Cardiovascular Disease) Augsburg Cohort Study, 1984 to 1992. , 1999, Circulation.
[32] H. Steinmetz,et al. Inflammation in high-grade carotid stenosis: a possible role for macrophages and T cells in plaque destabilization. , 1998, Stroke.
[33] P. Ridker,et al. Inflammation, aspirin, and the risk of cardiovascular disease in apparently healthy men. , 1997, The New England journal of medicine.
[34] L. Kuller,et al. Relation of C-reactive protein and coronary heart disease in the MRFIT nested case-control study. Multiple Risk Factor Intervention Trial. , 1996, American journal of epidemiology.
[35] Aldons J. Lusis,et al. Atherosclerosis : Vascular biology , 2000 .