Thromboxane production in morbidly obese subjects.
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F. Crea | G. Mingrone | S. Giubilato | A. Iaconelli | G. Liuzzo | F. Graziani | P. Cialdella | F. Pulcinelli | R. Della Bona | L. Biasucci
[1] V. Basevi. Diagnosis and Classification of Diabetes Mellitus , 2011, Diabetes Care.
[2] F. Crea,et al. Paradoxical preservation of vascular function in severe obesity. , 2010, The American journal of medicine.
[3] B. Chong,et al. Serum thromboxane B2 compared to five other platelet function tests for the evaluation of aspirin effect in stable cardiovascular disease. , 2010, Heart, lung & circulation.
[4] E. Grove,et al. A comparison of platelet function tests and thromboxane metabolites to evaluate aspirin response in healthy individuals and patients with coronary artery disease , 2010, Thrombosis and Haemostasis.
[5] C. Lavie,et al. Obesity and cardiovascular disease: risk factor, paradox, and impact of weight loss. , 2009, Journal of the American College of Cardiology.
[6] F. Crea,et al. COX-1 sensitivity and thromboxane A2 production in type 1 and type 2 diabetic patients under chronic aspirin treatment. , 2009, European heart journal.
[7] A. Carter,et al. Elevated C-Reactive Protein and Long-Term Mortality After Ischaemic Stroke: Relationship With Markers of Endothelial Cell and Platelet Activation , 2009, Stroke.
[8] G. Hasenfuss,et al. Absence of leptin resistance in platelets from morbidly obese individuals may contribute to the increased thrombosis risk in obesity , 2008, Thrombosis and Haemostasis.
[9] A. Corsonello,et al. Leptin, obesity and platelet responsiveness: another piece in the puzzle. , 2008, Thrombosis and haemostasis.
[10] M. Reilly,et al. Leptin resistance: a possible interface of inflammation and metabolism in obesity-related cardiovascular disease. , 2008, Journal of the American College of Cardiology.
[11] L. Bortolotto,et al. Systemic Inflammation and Cardiovascular Risk Factors: Are Morbidly Obese Subjects Different? , 2008, Obesity surgery.
[12] G. Rossi,et al. Diagnosis and Classification of Diabetes Mellitus The information that follows is based largely on the reports of the Expert Committee on the Diagnosis and Classification of Diabetes (Diabetes Care 20:1183–1197, 1997, and Diabetes Care 26:3160–3167, 2003). , 2008, Diabetes Care.
[13] A. Saitta,et al. Relationship between metabolic syndrome and platelet responsiveness to leptin in overweight and obese patients , 2007, International Journal of Obesity.
[14] M. Lopes,et al. Postmortem findings in morbidly obese individuals dying after gastric bypass procedures. , 2007, Human pathology.
[15] F. Santilli,et al. Insulin resistance as a determinant of platelet activation in obese women. , 2006, Journal of the American College of Cardiology.
[16] M. Laposata,et al. Effects of physiological leptin administration on markers of inflammation, platelet activation, and platelet aggregation during caloric deprivation. , 2005, The Journal of clinical endocrinology and metabolism.
[17] R. Czekay,et al. The leptin receptor system of human platelets , 2005, Journal of thrombosis and haemostasis : JTH.
[18] R. Turner,et al. Homeostasis model assessment: insulin resistance and β-cell function from fasting plasma glucose and insulin concentrations in man , 1985, Diabetologia.
[19] M. Buemi,et al. Leptin-dependent platelet aggregation in healthy, overweight and obese subjects , 2003, International Journal of Obesity.
[20] G. Davı̀,et al. Platelet activation in obese women: role of inflammation and oxidant stress. , 2002, JAMA.
[21] Kortelainen Ml. Myocardial infarction and coronary pathology in severely obese people examined at autopsy. , 2002 .
[22] S. Koschnick,et al. Leptin-dependent platelet aggregation and arterial thrombosis suggests a mechanism for atherothrombotic disease in obesity. , 2001, The Journal of clinical investigation.
[23] A. Yalçın,et al. Leptin does not play a major role in platelet aggregation in obesity and leptin deficiency. , 2001, Obesity research.
[24] M. Nakata,et al. Leptin promotes aggregation of human platelets via the long form of its receptor. , 1999, Diabetes.
[25] R. Virmani,et al. Sudden death as a result of heart disease in morbid obesity. , 1995, American heart journal.
[26] M. Reilly,et al. Cellular activation by thromboxane A2 and other eicosanoids. , 1993, European heart journal.
[27] S. Chierchia,et al. Pathophysiology of coronary occlusion in acute infarction. , 1986, Circulation.